Specific depletion of myeloid-derived suppressor cells by the chemotherapy agent 5-fluorouracil enhances protective immune response in paracoccidioidomycosis (2024)
- Authors:
- Autor USP: CALICH, VERA LUCIA GARCIA - ICB
- Unidade: ICB
- DOI: 10.1093/infdis/jiae350
- Subjects: IMUNOLOGIA; CÉLULAS MIELOIDES; CAMUNDONGOS; LINFÓCITOS T; LEUCÓCITOS; PULMÃO; CITOCINAS; CÉLULAS DENDRÍTICAS; PARACOCCIDIOIDES; PARACOCCIDIOIDES BRASILIENSIS; FÁRMACOS IMUNOSSUPRESSORES
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: The Journal of Infectious Diseases
- ISSN: 1537-6613
- Volume/Número/Paginação/Ano: v. 30, n. 5, p. 1279-1290, 2024
- Status:
- Nenhuma versão em acesso aberto identificada
-
ABNT
PREITE, Nycolas Willian et al. Specific depletion of myeloid-derived suppressor cells by the chemotherapy agent 5-fluorouracil enhances protective immune response in paracoccidioidomycosis. The Journal of Infectious Diseases, v. 30, n. 5, p. 1279-1290, 2024Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiae350. Acesso em: 16 abr. 2026. -
APA
Preite, N. W., Kaminski, V. de L., Borges, B. M., Santos, B. V. dos, Loures, F. V., & Calich, V. L. G. (2024). Specific depletion of myeloid-derived suppressor cells by the chemotherapy agent 5-fluorouracil enhances protective immune response in paracoccidioidomycosis. The Journal of Infectious Diseases, 30( 5), 1279-1290. doi:10.1093/infdis/jiae350 -
NLM
Preite NW, Kaminski V de L, Borges BM, Santos BV dos, Loures FV, Calich VLG. Specific depletion of myeloid-derived suppressor cells by the chemotherapy agent 5-fluorouracil enhances protective immune response in paracoccidioidomycosis [Internet]. The Journal of Infectious Diseases. 2024 ; 30( 5): 1279-1290.[citado 2026 abr. 16 ] Available from: https://doi.org/10.1093/infdis/jiae350 -
Vancouver
Preite NW, Kaminski V de L, Borges BM, Santos BV dos, Loures FV, Calich VLG. Specific depletion of myeloid-derived suppressor cells by the chemotherapy agent 5-fluorouracil enhances protective immune response in paracoccidioidomycosis [Internet]. The Journal of Infectious Diseases. 2024 ; 30( 5): 1279-1290.[citado 2026 abr. 16 ] Available from: https://doi.org/10.1093/infdis/jiae350 - cd28 costimulatory molecule deficiency in more severe paracoccidioides brasiliensis infection but protects mice from life-threatening immunopathology
- Paracoccidioides brasiliensis infection determines dentric cells to diferentiate to the plasmacytoid subpopulation which induces a more severe pulmonary infection when transfered to resistant mice
- The IDO-AhR axis controls Th17/Treg immunity in a pulmonary model of fungal infection
- TLR-2 is a negative regulator of TH17 cells and tissue pathology in a pulmonary model of fungal infection
- Absence of TLR2 results in less severe paracoccidioidomycosis but increased inflammatory response caused by PMN & TH17 cells
- Toll like receptors and the adaptor molecule MYD88 play an important role in pulmonary paracoccidioidomycosis
- TLR2 is a negative regulator of Th17 cells and tissue pathology in a pulmonary model of fungal infection
- Human alfa / beta and gama / delta t cells respond to proteins of the yeast form of paracoccidioides brasiliensis
- The immune response of high (H) and low responders (L) mice (selection III) and F1 (HIII x LIII) hybrids to paracoccidioides brasiliensis infection
- PMN leukocytes play a more prominent role in natural immunity of susceptible than resistant mice to pulmonary paracoccidiodomycosis
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