Specific depletion of myeloid-derived suppressor cells by the chemotherapy agent 5-fluorouracil enhances protective immune response in paracoccidioidomycosis (2024)
- Authors:
- Autor USP: CALICH, VERA LUCIA GARCIA - ICB
- Unidade: ICB
- DOI: 10.1093/infdis/jiae350
- Subjects: IMUNOLOGIA; CÉLULAS MIELOIDES; CAMUNDONGOS; LINFÓCITOS T; LEUCÓCITOS; PULMÃO; CITOCINAS; CÉLULAS DENDRÍTICAS; PARACOCCIDIOIDES; PARACOCCIDIOIDES BRASILIENSIS; FÁRMACOS IMUNOSSUPRESSORES
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: The Journal of Infectious Diseases
- ISSN: 1537-6613
- Volume/Número/Paginação/Ano: v. 30, n. 5, p. 1279-1290, 2024
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
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ABNT
PREITE, Nycolas Willian et al. Specific depletion of myeloid-derived suppressor cells by the chemotherapy agent 5-fluorouracil enhances protective immune response in paracoccidioidomycosis. The Journal of Infectious Diseases, v. 30, n. 5, p. 1279-1290, 2024Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiae350. Acesso em: 22 jan. 2026. -
APA
Preite, N. W., Kaminski, V. de L., Borges, B. M., Santos, B. V. dos, Loures, F. V., & Calich, V. L. G. (2024). Specific depletion of myeloid-derived suppressor cells by the chemotherapy agent 5-fluorouracil enhances protective immune response in paracoccidioidomycosis. The Journal of Infectious Diseases, 30( 5), 1279-1290. doi:10.1093/infdis/jiae350 -
NLM
Preite NW, Kaminski V de L, Borges BM, Santos BV dos, Loures FV, Calich VLG. Specific depletion of myeloid-derived suppressor cells by the chemotherapy agent 5-fluorouracil enhances protective immune response in paracoccidioidomycosis [Internet]. The Journal of Infectious Diseases. 2024 ; 30( 5): 1279-1290.[citado 2026 jan. 22 ] Available from: https://doi.org/10.1093/infdis/jiae350 -
Vancouver
Preite NW, Kaminski V de L, Borges BM, Santos BV dos, Loures FV, Calich VLG. Specific depletion of myeloid-derived suppressor cells by the chemotherapy agent 5-fluorouracil enhances protective immune response in paracoccidioidomycosis [Internet]. The Journal of Infectious Diseases. 2024 ; 30( 5): 1279-1290.[citado 2026 jan. 22 ] Available from: https://doi.org/10.1093/infdis/jiae350 - Depletion of natural killer cells induces a more severe pulmonary paracoccidioidomycosis in athymic and euthymic BALB/C mice
- Mannose receptors play a different role in the activation of macrophages from resistant and susceptible mice to Paracoccidioides brasiliensis infection
- iNOS knock-out mice are not more susceptible to pulmonary paracoccidioidomycosis
- Role of CD4, CD8 T cells, IFN-gama and IL-12 in the protective immunity against murine pulmonary paracoccidioidomycosis (PCM)
- Paracoccidioides brasiliensis-TLR-4 interaction induces macrophage activation but results in more severe paracoccidioimycosis
- Genetic deficiency of CD28 costimulatory molecule results in more severe Paracoccicioidomycosis (PCM) associated with decreased antibodies and cytokines production
- Suceptible and resistant mice to p. brasiliensis infection use an indoleamine 2,3-dioxygenase mechanism to control fungal growth
- Mannose receptors play a distinct role in the interaction of p. Brasiliensis cells with murine macrophages of resistant and susceptible.
- IL-10 deficiency determines a better fungicidal ability associated with overproduction of IFN-? and nitric oxide by Paracoccidioides brasiliensis infected macrophages
- Paracoccidioides brasiliensis infection determines dendritic cells to differentiate to the plasmocytoid subpopulation which induces a more severe pulmonary infection when transferred to resistant mice
Informações sobre o DOI: 10.1093/infdis/jiae350 (Fonte: oaDOI API)
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