The immune response of high (H) and low responders (L) mice (selection III) and F1 (HIII x LIII) hybrids to paracoccidioides brasiliensis infection (2000)
- Authors:
- Autor USP: CALICH, VERA LUCIA GARCIA - ICB
- Unidade: ICB
- Assunto: IMMUNOLOGIA
- Language: Inglês
- Imprenta:
- Publisher place: Florianópolis
- Date published: 2000
- Source:
- Título: Abstract
- Conference titles: Meeting of the Brazilian Society of Immunology
-
ABNT
DA HORA, Daisy Cristina Borges et al. The immune response of high (H) and low responders (L) mice (selection III) and F1 (HIII x LIII) hybrids to paracoccidioides brasiliensis infection. 2000, Anais.. Florianópolis: Instituto de Ciências Biomédicas, Universidade de São Paulo, 2000. . Acesso em: 11 fev. 2026. -
APA
Da Hora, D. C. B., Sant'Anna, O. A. B. E., Calich, V. L. G., & Baueb, S. (2000). The immune response of high (H) and low responders (L) mice (selection III) and F1 (HIII x LIII) hybrids to paracoccidioides brasiliensis infection. In Abstract. Florianópolis: Instituto de Ciências Biomédicas, Universidade de São Paulo. -
NLM
Da Hora DCB, Sant'Anna OABE, Calich VLG, Baueb S. The immune response of high (H) and low responders (L) mice (selection III) and F1 (HIII x LIII) hybrids to paracoccidioides brasiliensis infection. Abstract. 2000 ;[citado 2026 fev. 11 ] -
Vancouver
Da Hora DCB, Sant'Anna OABE, Calich VLG, Baueb S. The immune response of high (H) and low responders (L) mice (selection III) and F1 (HIII x LIII) hybrids to paracoccidioides brasiliensis infection. Abstract. 2000 ;[citado 2026 fev. 11 ] - cd28 costimulatory molecule deficiency in more severe paracoccidioides brasiliensis infection but protects mice from life-threatening immunopathology
- Paracoccidioides brasiliensis infection determines dentric cells to diferentiate to the plasmacytoid subpopulation which induces a more severe pulmonary infection when transfered to resistant mice
- The IDO-AhR axis controls Th17/Treg immunity in a pulmonary model of fungal infection
- TLR-2 is a negative regulator of TH17 cells and tissue pathology in a pulmonary model of fungal infection
- Absence of TLR2 results in less severe paracoccidioidomycosis but increased inflammatory response caused by PMN & TH17 cells
- Toll like receptors and the adaptor molecule MYD88 play an important role in pulmonary paracoccidioidomycosis
- TLR2 is a negative regulator of Th17 cells and tissue pathology in a pulmonary model of fungal infection
- Human alfa / beta and gama / delta t cells respond to proteins of the yeast form of paracoccidioides brasiliensis
- Specific depletion of myeloid-derived suppressor cells by the chemotherapy agent 5-fluorouracil enhances protective immune response in paracoccidioidomycosis
- PMN leukocytes play a more prominent role in natural immunity of susceptible than resistant mice to pulmonary paracoccidiodomycosis
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