TLR2 is a negative regulator of Th17 cells and tissue pathology in a pulmonary model of fungal infection (2009)
- Authors:
- Autor USP: CALICH, VERA LUCIA GARCIA - ICB
- Unidade: ICB
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Journal of Immunology
- ISSN: 0022-1767
- Volume/Número/Paginação/Ano: v. 183, n. 2, p. 1279-90, 2009
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ABNT
LOURES, Flávio Vieira et al. TLR2 is a negative regulator of Th17 cells and tissue pathology in a pulmonary model of fungal infection. Journal of Immunology, v. 183, n. 2, p. 1279-90, 2009Tradução . . Acesso em: 10 fev. 2026. -
APA
Loures, F. V., Pina, A., Felonato, M., & Calich, V. L. G. (2009). TLR2 is a negative regulator of Th17 cells and tissue pathology in a pulmonary model of fungal infection. Journal of Immunology, 183( 2), 1279-90. -
NLM
Loures FV, Pina A, Felonato M, Calich VLG. TLR2 is a negative regulator of Th17 cells and tissue pathology in a pulmonary model of fungal infection. Journal of Immunology. 2009 ; 183( 2): 1279-90.[citado 2026 fev. 10 ] -
Vancouver
Loures FV, Pina A, Felonato M, Calich VLG. TLR2 is a negative regulator of Th17 cells and tissue pathology in a pulmonary model of fungal infection. Journal of Immunology. 2009 ; 183( 2): 1279-90.[citado 2026 fev. 10 ] - cd28 costimulatory molecule deficiency in more severe paracoccidioides brasiliensis infection but protects mice from life-threatening immunopathology
- Paracoccidioides brasiliensis infection determines dentric cells to diferentiate to the plasmacytoid subpopulation which induces a more severe pulmonary infection when transfered to resistant mice
- The IDO-AhR axis controls Th17/Treg immunity in a pulmonary model of fungal infection
- TLR-2 is a negative regulator of TH17 cells and tissue pathology in a pulmonary model of fungal infection
- Absence of TLR2 results in less severe paracoccidioidomycosis but increased inflammatory response caused by PMN & TH17 cells
- Toll like receptors and the adaptor molecule MYD88 play an important role in pulmonary paracoccidioidomycosis
- Human alfa / beta and gama / delta t cells respond to proteins of the yeast form of paracoccidioides brasiliensis
- Specific depletion of myeloid-derived suppressor cells by the chemotherapy agent 5-fluorouracil enhances protective immune response in paracoccidioidomycosis
- The immune response of high (H) and low responders (L) mice (selection III) and F1 (HIII x LIII) hybrids to paracoccidioides brasiliensis infection
- PMN leukocytes play a more prominent role in natural immunity of susceptible than resistant mice to pulmonary paracoccidiodomycosis
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