PMN leukocytes play a more prominent role in natural immunity of susceptible than resistant mice to pulmonary paracoccidiodomycosis (2001)
- Authors:
- Autor USP: CALICH, VERA LUCIA GARCIA - ICB
- Unidade: ICB
- Assunto: IMUNOLOGIA
- Language: Português
- Imprenta:
- Publisher place: Oxford, GB
- Date published: 2001
- Source:
- Título: Scandinavian Journal of Immunology
- Volume/Número/Paginação/Ano: v.54, n.1, p.75, 2001
-
ABNT
PINA, A. e CALICH, Vera Lúcia Garcia. PMN leukocytes play a more prominent role in natural immunity of susceptible than resistant mice to pulmonary paracoccidiodomycosis. Scandinavian Journal of Immunology, v. 54, n. 1, p. 75, 2001Tradução . . Acesso em: 10 fev. 2026. -
APA
Pina, A., & Calich, V. L. G. (2001). PMN leukocytes play a more prominent role in natural immunity of susceptible than resistant mice to pulmonary paracoccidiodomycosis. Scandinavian Journal of Immunology, 54( 1), 75. -
NLM
Pina A, Calich VLG. PMN leukocytes play a more prominent role in natural immunity of susceptible than resistant mice to pulmonary paracoccidiodomycosis. Scandinavian Journal of Immunology. 2001 ;54( 1): 75.[citado 2026 fev. 10 ] -
Vancouver
Pina A, Calich VLG. PMN leukocytes play a more prominent role in natural immunity of susceptible than resistant mice to pulmonary paracoccidiodomycosis. Scandinavian Journal of Immunology. 2001 ;54( 1): 75.[citado 2026 fev. 10 ] - cd28 costimulatory molecule deficiency in more severe paracoccidioides brasiliensis infection but protects mice from life-threatening immunopathology
- Paracoccidioides brasiliensis infection determines dentric cells to diferentiate to the plasmacytoid subpopulation which induces a more severe pulmonary infection when transfered to resistant mice
- The IDO-AhR axis controls Th17/Treg immunity in a pulmonary model of fungal infection
- TLR-2 is a negative regulator of TH17 cells and tissue pathology in a pulmonary model of fungal infection
- Absence of TLR2 results in less severe paracoccidioidomycosis but increased inflammatory response caused by PMN & TH17 cells
- Toll like receptors and the adaptor molecule MYD88 play an important role in pulmonary paracoccidioidomycosis
- TLR2 is a negative regulator of Th17 cells and tissue pathology in a pulmonary model of fungal infection
- Human alfa / beta and gama / delta t cells respond to proteins of the yeast form of paracoccidioides brasiliensis
- Specific depletion of myeloid-derived suppressor cells by the chemotherapy agent 5-fluorouracil enhances protective immune response in paracoccidioidomycosis
- The immune response of high (H) and low responders (L) mice (selection III) and F1 (HIII x LIII) hybrids to paracoccidioides brasiliensis infection
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