Human alfa / beta and gama / delta t cells respond to proteins of the yeast form of paracoccidioides brasiliensis (1993)
- Authors:
- Autor USP: CALICH, VERA LUCIA GARCIA - ICB
- Unidade: ICB
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Publisher: Sociedade Brasileira de Imunologia
- Publisher place: São Paulo
- Date published: 1993
- Source:
- Título: Programa e Resumos
- Conference titles: Congresso Brasileiro de Imunologia
-
ABNT
MUNK, M E et al. Human alfa / beta and gama / delta t cells respond to proteins of the yeast form of paracoccidioides brasiliensis. 1993, Anais.. São Paulo: Sociedade Brasileira de Imunologia, 1993. . Acesso em: 10 fev. 2026. -
APA
Munk, M. E., Teixeira, H. C., Kuner, A., Fazioli, R. A., Calich, V. L. G., & Kaufmann, S. H. E. (1993). Human alfa / beta and gama / delta t cells respond to proteins of the yeast form of paracoccidioides brasiliensis. In Programa e Resumos. São Paulo: Sociedade Brasileira de Imunologia. -
NLM
Munk ME, Teixeira HC, Kuner A, Fazioli RA, Calich VLG, Kaufmann SHE. Human alfa / beta and gama / delta t cells respond to proteins of the yeast form of paracoccidioides brasiliensis. Programa e Resumos. 1993 ;[citado 2026 fev. 10 ] -
Vancouver
Munk ME, Teixeira HC, Kuner A, Fazioli RA, Calich VLG, Kaufmann SHE. Human alfa / beta and gama / delta t cells respond to proteins of the yeast form of paracoccidioides brasiliensis. Programa e Resumos. 1993 ;[citado 2026 fev. 10 ] - cd28 costimulatory molecule deficiency in more severe paracoccidioides brasiliensis infection but protects mice from life-threatening immunopathology
- Paracoccidioides brasiliensis infection determines dentric cells to diferentiate to the plasmacytoid subpopulation which induces a more severe pulmonary infection when transfered to resistant mice
- The IDO-AhR axis controls Th17/Treg immunity in a pulmonary model of fungal infection
- TLR-2 is a negative regulator of TH17 cells and tissue pathology in a pulmonary model of fungal infection
- Absence of TLR2 results in less severe paracoccidioidomycosis but increased inflammatory response caused by PMN & TH17 cells
- Toll like receptors and the adaptor molecule MYD88 play an important role in pulmonary paracoccidioidomycosis
- TLR2 is a negative regulator of Th17 cells and tissue pathology in a pulmonary model of fungal infection
- Specific depletion of myeloid-derived suppressor cells by the chemotherapy agent 5-fluorouracil enhances protective immune response in paracoccidioidomycosis
- The immune response of high (H) and low responders (L) mice (selection III) and F1 (HIII x LIII) hybrids to paracoccidioides brasiliensis infection
- PMN leukocytes play a more prominent role in natural immunity of susceptible than resistant mice to pulmonary paracoccidiodomycosis
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