Toll like receptors and the adaptor molecule MYD88 play an important role in pulmonary paracoccidioidomycosis (2008)
- Authors:
- Autor USP: CALICH, VERA LUCIA GARCIA - ICB
- Unidade: ICB
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Biomédica
- Volume/Número/Paginação/Ano: v. 28, suppl. 1, 2008
- Conference titles: Proceedings of the International Congress on Paracoccidioidomycosis
-
ABNT
CALICH, Vera Lúcia Garcia e LOURES, Flávio V. Toll like receptors and the adaptor molecule MYD88 play an important role in pulmonary paracoccidioidomycosis. Biomédica. Bogotá: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 06 out. 2024. , 2008 -
APA
Calich, V. L. G., & Loures, F. V. (2008). Toll like receptors and the adaptor molecule MYD88 play an important role in pulmonary paracoccidioidomycosis. Biomédica. Bogotá: Instituto de Ciências Biomédicas, Universidade de São Paulo. -
NLM
Calich VLG, Loures FV. Toll like receptors and the adaptor molecule MYD88 play an important role in pulmonary paracoccidioidomycosis. Biomédica. 2008 ; 28[citado 2024 out. 06 ] -
Vancouver
Calich VLG, Loures FV. Toll like receptors and the adaptor molecule MYD88 play an important role in pulmonary paracoccidioidomycosis. Biomédica. 2008 ; 28[citado 2024 out. 06 ] - Role of IL-12 in the innate immunity of athymic and euthymic balb/c mice with paracoccidioidomycosis: effect of local and systemic il-12 neutralization
- Experimental paracoccidioidomycosis: mechanisms of resistance to fungi
- Exarcebation of murine paracoccidioidomycosis induces type 2 cytokines, whereas the protective model of the disease is associated to both, type 1 and type 2 T cell responses
- Effect of in vivo depletion of T CD4 cells in the pulmonary infection of susceptible (B10.A) and resistance (A/J) mice to P. brasiliensis infection
- Leucócitos polimorfonucleares (PMN) não exercem papel protetor na fase adquirida da imunidade contra a infecção com Paracoccidioides brasiliensis
- Role of CD4, CD8 T cells, IFN-gama and IL-12 in the protective immunity against murine pulmonary paracoccidioidomycosis (PCM)
- The immune response of high (H) and low responders (L) mice (selection III) and F1 (HIII x LIII) hybrids to paracoccidioides brasiliensis infection
- Pulmonary paracoccidioidomycosis (PCM) in IL-10 knock-out (KO) mice
- CD28 costimulatory molecule deficiency results in more severe Paracoccidioides brasiliensis infection but protects mice from life-threatening immunopathology
- Susceptible and resistant mice to P. Brasiliensis infection use an indoleamine 2,3-dioxygenase mechanism to control fungal growth
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