Absence of TLR2 results in less severe paracoccidioidomycosis but increased inflammatory response caused by PMN & TH17 cells (2008)
- Authors:
- Autor USP: CALICH, VERA LUCIA GARCIA - ICB
- Unidade: ICB
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Biomédica
- Volume/Número/Paginação/Ano: v. 28, suppl. 1, 2008
- Conference titles: Proceedings of the International Congress on Paracoccidioidomycosis
-
ABNT
LOURES, Flávio V. e CALICH, Vera Lúcia Garcia. Absence of TLR2 results in less severe paracoccidioidomycosis but increased inflammatory response caused by PMN & TH17 cells. Biomédica. Bogotá: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 10 fev. 2026. , 2008 -
APA
Loures, F. V., & Calich, V. L. G. (2008). Absence of TLR2 results in less severe paracoccidioidomycosis but increased inflammatory response caused by PMN & TH17 cells. Biomédica. Bogotá: Instituto de Ciências Biomédicas, Universidade de São Paulo. -
NLM
Loures FV, Calich VLG. Absence of TLR2 results in less severe paracoccidioidomycosis but increased inflammatory response caused by PMN & TH17 cells. Biomédica. 2008 ; 28[citado 2026 fev. 10 ] -
Vancouver
Loures FV, Calich VLG. Absence of TLR2 results in less severe paracoccidioidomycosis but increased inflammatory response caused by PMN & TH17 cells. Biomédica. 2008 ; 28[citado 2026 fev. 10 ] - cd28 costimulatory molecule deficiency in more severe paracoccidioides brasiliensis infection but protects mice from life-threatening immunopathology
- Paracoccidioides brasiliensis infection determines dentric cells to diferentiate to the plasmacytoid subpopulation which induces a more severe pulmonary infection when transfered to resistant mice
- The IDO-AhR axis controls Th17/Treg immunity in a pulmonary model of fungal infection
- TLR-2 is a negative regulator of TH17 cells and tissue pathology in a pulmonary model of fungal infection
- Absence of TLR2 results in less severe paracoccidioidomycosis but increased inflammatory response caused by PMN & TH17 cells
- Toll like receptors and the adaptor molecule MYD88 play an important role in pulmonary paracoccidioidomycosis
- TLR2 is a negative regulator of Th17 cells and tissue pathology in a pulmonary model of fungal infection
- Human alfa / beta and gama / delta t cells respond to proteins of the yeast form of paracoccidioides brasiliensis
- Specific depletion of myeloid-derived suppressor cells by the chemotherapy agent 5-fluorouracil enhances protective immune response in paracoccidioidomycosis
- The immune response of high (H) and low responders (L) mice (selection III) and F1 (HIII x LIII) hybrids to paracoccidioides brasiliensis infection
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