Myeloid-derived suppressor cells are associated with impaired Th1 and Th17 responses and severe pulmonary paracoccidioidomycosis which is reversed by anti-Gr1 therapy (2023)
- Authors:
- Autor USP: CALICH, VERA LUCIA GARCIA - ICB
- Unidade: ICB
- DOI: 10.3389/fimmu.2023.1039244
- Subjects: IMUNOLOGIA; CÉLULAS SANGUÍNEAS; MEDULA ÓSSEA DE ANIMAL; CÉLULAS MIELOIDES; PARACOCCIDIOIDOMICOSE; DERMATOPATIAS INFECCIOSAS; FÁRMACOS IMUNOSSUPRESSORES; PULMÃO; INFECÇÕES RESPIRATÓRIAS
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Frontiers in Immunology
- ISSN: 1664-3224
- Volume/Número/Paginação/Ano: v. 14, art. 1039244, p. 1-19, 2023
- Status:
- Artigo publicado em periódico de acesso aberto (Gold Open Access)
- Versão do Documento:
- Versão publicada (Published version)
- Acessar versão aberta:
-
ABNT
PREITE, Nycolas Willian et al. Myeloid-derived suppressor cells are associated with impaired Th1 and Th17 responses and severe pulmonary paracoccidioidomycosis which is reversed by anti-Gr1 therapy. Frontiers in Immunology, v. 14, p. 1-19, 2023Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2023.1039244. Acesso em: 15 abr. 2026. -
APA
Preite, N. W., Kaminski, V. de L., Borges, B. M., Calich, V. L. G., & Loures, F. V. (2023). Myeloid-derived suppressor cells are associated with impaired Th1 and Th17 responses and severe pulmonary paracoccidioidomycosis which is reversed by anti-Gr1 therapy. Frontiers in Immunology, 14, 1-19. doi:10.3389/fimmu.2023.1039244 -
NLM
Preite NW, Kaminski V de L, Borges BM, Calich VLG, Loures FV. Myeloid-derived suppressor cells are associated with impaired Th1 and Th17 responses and severe pulmonary paracoccidioidomycosis which is reversed by anti-Gr1 therapy [Internet]. Frontiers in Immunology. 2023 ; 14 1-19.[citado 2026 abr. 15 ] Available from: https://doi.org/10.3389/fimmu.2023.1039244 -
Vancouver
Preite NW, Kaminski V de L, Borges BM, Calich VLG, Loures FV. Myeloid-derived suppressor cells are associated with impaired Th1 and Th17 responses and severe pulmonary paracoccidioidomycosis which is reversed by anti-Gr1 therapy [Internet]. Frontiers in Immunology. 2023 ; 14 1-19.[citado 2026 abr. 15 ] Available from: https://doi.org/10.3389/fimmu.2023.1039244 - cd28 costimulatory molecule deficiency in more severe paracoccidioides brasiliensis infection but protects mice from life-threatening immunopathology
- Paracoccidioides brasiliensis infection determines dentric cells to diferentiate to the plasmacytoid subpopulation which induces a more severe pulmonary infection when transfered to resistant mice
- The IDO-AhR axis controls Th17/Treg immunity in a pulmonary model of fungal infection
- TLR-2 is a negative regulator of TH17 cells and tissue pathology in a pulmonary model of fungal infection
- Absence of TLR2 results in less severe paracoccidioidomycosis but increased inflammatory response caused by PMN & TH17 cells
- Toll like receptors and the adaptor molecule MYD88 play an important role in pulmonary paracoccidioidomycosis
- TLR2 is a negative regulator of Th17 cells and tissue pathology in a pulmonary model of fungal infection
- Human alfa / beta and gama / delta t cells respond to proteins of the yeast form of paracoccidioides brasiliensis
- The immune response of high (H) and low responders (L) mice (selection III) and F1 (HIII x LIII) hybrids to paracoccidioides brasiliensis infection
- PMN leukocytes play a more prominent role in natural immunity of susceptible than resistant mice to pulmonary paracoccidiodomycosis
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