Paracoccidioides brasiliensis infection determines dendritic cells to differentiate to the plasmocytoid subpopulation which induces a more severe pulmonary infection when transferred to resistant mice (2008)
- Authors:
- Autor USP: bittencourt, jackson - ICB
- Unidade: ICB
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Biomédica
- Volume/Número/Paginação/Ano: v. 28, suppl. 1, 2008
- Conference titles: Proceedings of the International Congress on Paracoccidioidomycosis
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ABNT
PINA, A. e CALICH, Vera Lúcia Garcia. Paracoccidioides brasiliensis infection determines dendritic cells to differentiate to the plasmocytoid subpopulation which induces a more severe pulmonary infection when transferred to resistant mice. Biomédica. Bogotá: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 12 jan. 2026. , 2008 -
APA
Pina, A., & Calich, V. L. G. (2008). Paracoccidioides brasiliensis infection determines dendritic cells to differentiate to the plasmocytoid subpopulation which induces a more severe pulmonary infection when transferred to resistant mice. Biomédica. Bogotá: Instituto de Ciências Biomédicas, Universidade de São Paulo. -
NLM
Pina A, Calich VLG. Paracoccidioides brasiliensis infection determines dendritic cells to differentiate to the plasmocytoid subpopulation which induces a more severe pulmonary infection when transferred to resistant mice. Biomédica. 2008 ; 28[citado 2026 jan. 12 ] -
Vancouver
Pina A, Calich VLG. Paracoccidioides brasiliensis infection determines dendritic cells to differentiate to the plasmocytoid subpopulation which induces a more severe pulmonary infection when transferred to resistant mice. Biomédica. 2008 ; 28[citado 2026 jan. 12 ] - Nitric oxide but not treg cells plays a major immunoregulatory role in a pulmonary model of fungal infection
- Mannose receptors play a different role in the activation of macrophages from resistant and susceptible mice to Paracoccidioides brasiliensis infection
- Leucócitos polimorfonucleares (PMN) não exercem papel protetor na fase adquirida da imunidade contra a infecção com Paracoccidioides brasiliensis
- Effect of pre-treatment with laminin in pulmonary paracoccidioidomycosis of resistant and suceptible mice to "Paracoccidioides brasiliensis"
- In vivo and in vitro characteristics of six Paracoccidioides brasiliensis strains
- Indoleamine 2,3-dioxygenase (IDO) controls the fungal loads and cellular immunity in pulmonary paracoccidiodomycosis (PCM) developed by susceptible and resistant mice to the fungus
- MyD88 signaling is required for efficient response to Paracoccidioides brasiliensis infection
- CD28 costimulatory molecule deficiency results in more severe Paracoccidioides brasiliensis infection but protects mice from life-threatening immunopathology
- Mannose receptors play a distinct role in the interaction of p. Brasiliensis cells with murine macrophages of resistant and susceptible.
- IL-10 deficiency determines a better fungicidal ability associated with overproduction of IFN-? and nitric oxide by Paracoccidioides brasiliensis infected macrophages
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