Mannose receptors play a distinct role in the interaction of p. Brasiliensis cells with murine macrophages of resistant and susceptible. (2008)
- Authors:
- Autor USP: CALICH, VERA LUCIA GARCIA - ICB
- Unidade: ICB
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Publisher: Comissão de Cultura e Extensão Universitária do ICB/USP
- Publisher place: São Paulo
- Date published: 2008
- Source:
- Título: Resumos
- Conference titles: Congresso do Instituto de Ciências Biomédicas da Universidade de São Paulo
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ABNT
TUNES, Feriotti Claudia e CALICH, Vera Lúcia Garcia. Mannose receptors play a distinct role in the interaction of p. Brasiliensis cells with murine macrophages of resistant and susceptible. 2008, Anais.. São Paulo: Comissão de Cultura e Extensão Universitária do ICB/USP, 2008. . Acesso em: 22 jan. 2026. -
APA
Tunes, F. C., & Calich, V. L. G. (2008). Mannose receptors play a distinct role in the interaction of p. Brasiliensis cells with murine macrophages of resistant and susceptible. In Resumos. São Paulo: Comissão de Cultura e Extensão Universitária do ICB/USP. -
NLM
Tunes FC, Calich VLG. Mannose receptors play a distinct role in the interaction of p. Brasiliensis cells with murine macrophages of resistant and susceptible. Resumos. 2008 ;[citado 2026 jan. 22 ] -
Vancouver
Tunes FC, Calich VLG. Mannose receptors play a distinct role in the interaction of p. Brasiliensis cells with murine macrophages of resistant and susceptible. Resumos. 2008 ;[citado 2026 jan. 22 ] - Depletion of natural killer cells induces a more severe pulmonary paracoccidioidomycosis in athymic and euthymic BALB/C mice
- Mannose receptors play a different role in the activation of macrophages from resistant and susceptible mice to Paracoccidioides brasiliensis infection
- iNOS knock-out mice are not more susceptible to pulmonary paracoccidioidomycosis
- Role of CD4, CD8 T cells, IFN-gama and IL-12 in the protective immunity against murine pulmonary paracoccidioidomycosis (PCM)
- Paracoccidioides brasiliensis-TLR-4 interaction induces macrophage activation but results in more severe paracoccidioimycosis
- Genetic deficiency of CD28 costimulatory molecule results in more severe Paracoccicioidomycosis (PCM) associated with decreased antibodies and cytokines production
- Suceptible and resistant mice to p. brasiliensis infection use an indoleamine 2,3-dioxygenase mechanism to control fungal growth
- IL-10 deficiency determines a better fungicidal ability associated with overproduction of IFN-? and nitric oxide by Paracoccidioides brasiliensis infected macrophages
- Paracoccidioides brasiliensis infection determines dendritic cells to differentiate to the plasmocytoid subpopulation which induces a more severe pulmonary infection when transferred to resistant mice
- Nitric oxide but not treg cells plays a major immunoregulatory role in a pulmonary model of fungal infection
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