Neutrophil role in pulmonary paracoccidioidomycosis depends on the resistance pattern of hosts (2006)
- Authors:
- USP affiliated authors: CALICH, VERA LUCIA GARCIA - ICB ; SALDIVA, PAULO HILARIO NASCIMENTO - FM
- Unidades: ICB; FM
- DOI: 10.1189/jlb.0106052
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Journal of Leukocyte Biology
- ISSN: 0741-5400
- Volume/Número/Paginação/Ano: v. 79, n. 4, p. 1202-1213, 2006
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
PINA, Adriana et al. Neutrophil role in pulmonary paracoccidioidomycosis depends on the resistance pattern of hosts. Journal of Leukocyte Biology, v. 79, n. 4, p. 1202-1213, 2006Tradução . . Disponível em: https://doi.org/10.1189/jlb.0106052. Acesso em: 25 fev. 2026. -
APA
Pina, A., Saldiva, P. H. N., Restrepo, L. E. C., & Calich, V. L. G. (2006). Neutrophil role in pulmonary paracoccidioidomycosis depends on the resistance pattern of hosts. Journal of Leukocyte Biology, 79( 4), 1202-1213. doi:10.1189/jlb.0106052 -
NLM
Pina A, Saldiva PHN, Restrepo LEC, Calich VLG. Neutrophil role in pulmonary paracoccidioidomycosis depends on the resistance pattern of hosts [Internet]. Journal of Leukocyte Biology. 2006 ; 79( 4): 1202-1213.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1189/jlb.0106052 -
Vancouver
Pina A, Saldiva PHN, Restrepo LEC, Calich VLG. Neutrophil role in pulmonary paracoccidioidomycosis depends on the resistance pattern of hosts [Internet]. Journal of Leukocyte Biology. 2006 ; 79( 4): 1202-1213.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1189/jlb.0106052 - cd28 costimulatory molecule deficiency in more severe paracoccidioides brasiliensis infection but protects mice from life-threatening immunopathology
- Paracoccidioides brasiliensis infection determines dentric cells to diferentiate to the plasmacytoid subpopulation which induces a more severe pulmonary infection when transfered to resistant mice
- The IDO-AhR axis controls Th17/Treg immunity in a pulmonary model of fungal infection
- TLR-2 is a negative regulator of TH17 cells and tissue pathology in a pulmonary model of fungal infection
- Absence of TLR2 results in less severe paracoccidioidomycosis but increased inflammatory response caused by PMN & TH17 cells
- Toll like receptors and the adaptor molecule MYD88 play an important role in pulmonary paracoccidioidomycosis
- TLR2 is a negative regulator of Th17 cells and tissue pathology in a pulmonary model of fungal infection
- Human alfa / beta and gama / delta t cells respond to proteins of the yeast form of paracoccidioides brasiliensis
- The immune response of high (H) and low responders (L) mice (selection III) and F1 (HIII x LIII) hybrids to paracoccidioides brasiliensis infection
- PMN leukocytes play a more prominent role in natural immunity of susceptible than resistant mice to pulmonary paracoccidiodomycosis
Informações sobre o DOI: 10.1189/jlb.0106052 (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
