The innate and acquired immune responses in paracoccidioidomycosis: defining the targets for immunomodulation and immunotherapy (2005)
- Autor:
- Autor USP: CALICH, VERA LUCIA GARCIA - ICB
- Unidade: ICB
- Assunto: MICROBIOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Revista do Instituto de Medicina Tropical de São Paulo
- ISSN: 0036-4665
- Volume/Número/Paginação/Ano: v. 47, suppl. 14, p. 15, 2005
- Conference titles: International Meeting on Paracoccidioidomycosis
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ABNT
CALICH, Vera Lúcia Garcia. The innate and acquired immune responses in paracoccidioidomycosis: defining the targets for immunomodulation and immunotherapy. Revista do Instituto de Medicina Tropical de São Paulo. São Paulo: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 17 mar. 2026. , 2005 -
APA
Calich, V. L. G. (2005). The innate and acquired immune responses in paracoccidioidomycosis: defining the targets for immunomodulation and immunotherapy. Revista do Instituto de Medicina Tropical de São Paulo. São Paulo: Instituto de Ciências Biomédicas, Universidade de São Paulo. -
NLM
Calich VLG. The innate and acquired immune responses in paracoccidioidomycosis: defining the targets for immunomodulation and immunotherapy. Revista do Instituto de Medicina Tropical de São Paulo. 2005 ; 47 15.[citado 2026 mar. 17 ] -
Vancouver
Calich VLG. The innate and acquired immune responses in paracoccidioidomycosis: defining the targets for immunomodulation and immunotherapy. Revista do Instituto de Medicina Tropical de São Paulo. 2005 ; 47 15.[citado 2026 mar. 17 ] - cd28 costimulatory molecule deficiency in more severe paracoccidioides brasiliensis infection but protects mice from life-threatening immunopathology
- Paracoccidioides brasiliensis infection determines dentric cells to diferentiate to the plasmacytoid subpopulation which induces a more severe pulmonary infection when transfered to resistant mice
- The IDO-AhR axis controls Th17/Treg immunity in a pulmonary model of fungal infection
- TLR-2 is a negative regulator of TH17 cells and tissue pathology in a pulmonary model of fungal infection
- Absence of TLR2 results in less severe paracoccidioidomycosis but increased inflammatory response caused by PMN & TH17 cells
- Toll like receptors and the adaptor molecule MYD88 play an important role in pulmonary paracoccidioidomycosis
- TLR2 is a negative regulator of Th17 cells and tissue pathology in a pulmonary model of fungal infection
- Human alfa / beta and gama / delta t cells respond to proteins of the yeast form of paracoccidioides brasiliensis
- The immune response of high (H) and low responders (L) mice (selection III) and F1 (HIII x LIII) hybrids to paracoccidioides brasiliensis infection
- PMN leukocytes play a more prominent role in natural immunity of susceptible than resistant mice to pulmonary paracoccidiodomycosis
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