Effect of in vivo depletion of T CD4 cells in the pulmonary infection of susceptible (B10.A) and resistance (A/J) mice to P. brasiliensis infection (2001)
- Authors:
- Autor USP: CALICH, VERA LUCIA GARCIA - ICB
- Unidade: ICB
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Publisher place: Rio de Janeiro
- Date published: 2001
- Source:
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ABNT
CHIARELLA, A e CALICH, Vera Lúcia Garcia. Effect of in vivo depletion of T CD4 cells in the pulmonary infection of susceptible (B10.A) and resistance (A/J) mice to P. brasiliensis infection. Revista da Sociedade Brasileira de Medicina Tropical, 2001Tradução . . Acesso em: 17 mar. 2026. -
APA
Chiarella, A., & Calich, V. L. G. (2001). Effect of in vivo depletion of T CD4 cells in the pulmonary infection of susceptible (B10.A) and resistance (A/J) mice to P. brasiliensis infection. Revista da Sociedade Brasileira de Medicina Tropical. -
NLM
Chiarella A, Calich VLG. Effect of in vivo depletion of T CD4 cells in the pulmonary infection of susceptible (B10.A) and resistance (A/J) mice to P. brasiliensis infection. Revista da Sociedade Brasileira de Medicina Tropical. 2001 ;[citado 2026 mar. 17 ] -
Vancouver
Chiarella A, Calich VLG. Effect of in vivo depletion of T CD4 cells in the pulmonary infection of susceptible (B10.A) and resistance (A/J) mice to P. brasiliensis infection. Revista da Sociedade Brasileira de Medicina Tropical. 2001 ;[citado 2026 mar. 17 ] - cd28 costimulatory molecule deficiency in more severe paracoccidioides brasiliensis infection but protects mice from life-threatening immunopathology
- Paracoccidioides brasiliensis infection determines dentric cells to diferentiate to the plasmacytoid subpopulation which induces a more severe pulmonary infection when transfered to resistant mice
- The IDO-AhR axis controls Th17/Treg immunity in a pulmonary model of fungal infection
- TLR-2 is a negative regulator of TH17 cells and tissue pathology in a pulmonary model of fungal infection
- Absence of TLR2 results in less severe paracoccidioidomycosis but increased inflammatory response caused by PMN & TH17 cells
- Toll like receptors and the adaptor molecule MYD88 play an important role in pulmonary paracoccidioidomycosis
- TLR2 is a negative regulator of Th17 cells and tissue pathology in a pulmonary model of fungal infection
- Human alfa / beta and gama / delta t cells respond to proteins of the yeast form of paracoccidioides brasiliensis
- The immune response of high (H) and low responders (L) mice (selection III) and F1 (HIII x LIII) hybrids to paracoccidioides brasiliensis infection
- PMN leukocytes play a more prominent role in natural immunity of susceptible than resistant mice to pulmonary paracoccidiodomycosis
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