Experimental paracoccidioidomycosis: mechanisms of resistance to fungi (1986)
- Autor:
- Autor USP: CALICH, VERA LUCIA GARCIA - ICB
- Unidade: ICB
- Subjects: PARACOCCIDIOIDOMICOSE; FUNGOS
- Language: Português
- Imprenta:
- Source:
- Título do periódico: Programa Y Resumenes
- Conference titles: Coloquio Internacional sobre la Paracoccidioidomycosis
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ABNT
CALICH, Vera Lúcia Garcia. Experimental paracoccidioidomycosis: mechanisms of resistance to fungi. Anais.. Medellin: [s.n.], 1986. -
APA
Calich, V. L. G. (1986). Experimental paracoccidioidomycosis: mechanisms of resistance to fungi. In Programa Y Resumenes. Medellin. -
NLM
Calich VLG. Experimental paracoccidioidomycosis: mechanisms of resistance to fungi. Programa Y Resumenes. 1986 ; -
Vancouver
Calich VLG. Experimental paracoccidioidomycosis: mechanisms of resistance to fungi. Programa Y Resumenes. 1986 ; - Exarcebation of murine paracoccidioidomycosis induces type 2 cytokines, whereas the protective model of the disease is associated to both, type 1 and type 2 T cell responses
- Role of CD4, CD8 T cells, IFN-gama and IL-12 in the protective immunity against murine pulmonary paracoccidioidomycosis (PCM)
- The immune response of high (H) and low responders (L) mice (selection III) and F1 (HIII x LIII) hybrids to paracoccidioides brasiliensis infection
- Leucócitos polimorfonucleares (PMN) não exercem papel protetor na fase adquirida da imunidade contra a infecção com Paracoccidioides brasiliensis
- Pulmonary paracoccidioidomycosis (PCM) in IL-10 knock-out (KO) mice
- Anti-CD25 treatment depletes treg cells and decreases disease severity in susceptible and resistant mice infected with Paracoccidioides brasiliensis
- CD28 exerts protective and detrimental effects in a pulmonary model of paracoccidioidomycosis
- TLR4 is used by a fungal pathogen to infect macrophages but also to induce protective immunity
- Suceptible and resistant mice to p. brasiliensis infection use an indoleamine 2,3-dioxygenase mechanism to control fungal growth
- CD28 costimulatory molecule deficiency results in more severe Paracoccidioides brasiliensis infection but protects mice from life-threatening immunopathology
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