In vivo and in vitro characteristics of six Paracoccidioides brasiliensis strains (1985)
- Authors:
- Autor USP: CALICH, VERA LUCIA GARCIA - ICB
- Unidade: ICB
- DOI: 10.1007/bf00437630
- Subjects: IMUNOLOGIA; PARACOCCIDIOIDES BRASILIENSIS
- Language: Português
- Imprenta:
- Este artigo NÃO possui versão em acesso aberto
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Status: Nenhuma versão em acesso aberto identificada -
ABNT
KASHINO, Suely Sanae et al. In vivo and in vitro characteristics of six Paracoccidioides brasiliensis strains. 1985Tradução . . Disponível em: https://doi.org/10.1007/bf00437630. Acesso em: 13 mar. 2026. -
APA
Kashino, S. S., Calich, V. L. G., Burger, E., & Singer Vermes, L. M. (1985). In vivo and in vitro characteristics of six Paracoccidioides brasiliensis strains. doi:10.1007/bf00437630 -
NLM
Kashino SS, Calich VLG, Burger E, Singer Vermes LM. In vivo and in vitro characteristics of six Paracoccidioides brasiliensis strains [Internet]. 1985 ;[citado 2026 mar. 13 ] Available from: https://doi.org/10.1007/bf00437630 -
Vancouver
Kashino SS, Calich VLG, Burger E, Singer Vermes LM. In vivo and in vitro characteristics of six Paracoccidioides brasiliensis strains [Internet]. 1985 ;[citado 2026 mar. 13 ] Available from: https://doi.org/10.1007/bf00437630 - cd28 costimulatory molecule deficiency in more severe paracoccidioides brasiliensis infection but protects mice from life-threatening immunopathology
- Paracoccidioides brasiliensis infection determines dentric cells to diferentiate to the plasmacytoid subpopulation which induces a more severe pulmonary infection when transfered to resistant mice
- The IDO-AhR axis controls Th17/Treg immunity in a pulmonary model of fungal infection
- TLR-2 is a negative regulator of TH17 cells and tissue pathology in a pulmonary model of fungal infection
- Absence of TLR2 results in less severe paracoccidioidomycosis but increased inflammatory response caused by PMN & TH17 cells
- Toll like receptors and the adaptor molecule MYD88 play an important role in pulmonary paracoccidioidomycosis
- TLR2 is a negative regulator of Th17 cells and tissue pathology in a pulmonary model of fungal infection
- Human alfa / beta and gama / delta t cells respond to proteins of the yeast form of paracoccidioides brasiliensis
- The immune response of high (H) and low responders (L) mice (selection III) and F1 (HIII x LIII) hybrids to paracoccidioides brasiliensis infection
- PMN leukocytes play a more prominent role in natural immunity of susceptible than resistant mice to pulmonary paracoccidiodomycosis
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