NOD/RIP2 signaling contribute to development of a TH1 response and resistance against L. major infection (2014)
- Authors:
- USP affiliated authors: SILVA, JOÃO SANTANA DA - FMRP ; ZAMBONI, DARIO SIMÕES - FMRP
- Unidade: FMRP
- Subjects: RECEPTORES; IMUNIDADE; LEISHMANIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Anais
- Conference titles: Reunião Anual da Sociedade Brasileira de Protozoologia
-
ABNT
LIMA JÚNIOR, Djalma de Souza e SILVA, João Santana da e ZAMBONI, Dario Simões. NOD/RIP2 signaling contribute to development of a TH1 response and resistance against L. major infection. 2014, Anais.. Caxambu: SBPz, 2014. Disponível em: http://itpack31.itarget.com.br/uploads/spz/arquivos/08-hp%2039%20ate%20131-2014.pdf. Acesso em: 01 out. 2024. -
APA
Lima Júnior, D. de S., Silva, J. S. da, & Zamboni, D. S. (2014). NOD/RIP2 signaling contribute to development of a TH1 response and resistance against L. major infection. In Anais. Caxambu: SBPz. Recuperado de http://itpack31.itarget.com.br/uploads/spz/arquivos/08-hp%2039%20ate%20131-2014.pdf -
NLM
Lima Júnior D de S, Silva JS da, Zamboni DS. NOD/RIP2 signaling contribute to development of a TH1 response and resistance against L. major infection [Internet]. Anais. 2014 ;[citado 2024 out. 01 ] Available from: http://itpack31.itarget.com.br/uploads/spz/arquivos/08-hp%2039%20ate%20131-2014.pdf -
Vancouver
Lima Júnior D de S, Silva JS da, Zamboni DS. NOD/RIP2 signaling contribute to development of a TH1 response and resistance against L. major infection [Internet]. Anais. 2014 ;[citado 2024 out. 01 ] Available from: http://itpack31.itarget.com.br/uploads/spz/arquivos/08-hp%2039%20ate%20131-2014.pdf - Ros-dependent NLRP3-inflammasome activation is critical for restriction of Leishmania amazonensis infection
- Critical role of ASC inflammasomes in host innate resistance to Trypanosoma cruzi infection
- Cutting edge: nucleotide binding oligomerization domain 1-dependent responses account for murine resistance against trypanosoma cruzi infection
- Resistance to Leishmania spp. is mediated by the inflammasome induced duo: IL- 1β and nitric oxide
- NOD1/NOD2/RIP2 signaling in response to non-peptidoglycan containing pathogen contribute to development of a TH1-biased response and resistance against L. major infection
- Interplay between reactive oxygen species and the inflammasome are crucial for restriction of Neospora caninum replication
- Apoptosis-associated speck–like protein containing a caspase recruitment domain inflammasomes mediate IL-1β response and host resistance to Trypanosoma cruzi infection
- IFN-γ plays a unique role in protection against low virulent Trypanosoma cruzi strain
- A parent-of-origin effect determines the susceptibility of a non-informative F1 population to Trypanosoma cruzi infection in vivo
- Restriction of Leishmania amazonensis infection by the inflammasomes
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