Cutting edge: nucleotide binding oligomerization domain 1-dependent responses account for murine resistance against trypanosoma cruzi infection (2010)
- Authors:
- USP affiliated authors: SILVA, JOÃO SANTANA DA - FMRP ; ZAMBONI, DARIO SIMÕES - FMRP
- Unidade: FMRP
- Subjects: TRYPANOSOMA CRUZI; CITOCINAS
- Language: Inglês
- Imprenta:
- Source:
- Título: Journal of Immunology
- ISSN: 0022-1767
- Volume/Número/Paginação/Ano: v. 184, n. 3, p. 1148-1152, 2010
-
ABNT
SILVA, Grace Kelly da et al. Cutting edge: nucleotide binding oligomerization domain 1-dependent responses account for murine resistance against trypanosoma cruzi infection. Journal of Immunology, v. 184, n. 3, p. 1148-1152, 2010Tradução . . Acesso em: 12 fev. 2026. -
APA
Silva, G. K. da, Gutierrez, F. R. S., Guedes, P. M. da M., Horta, C. V., Cunha, L. D. da, Mineo, T. W. P., et al. (2010). Cutting edge: nucleotide binding oligomerization domain 1-dependent responses account for murine resistance against trypanosoma cruzi infection. Journal of Immunology, 184( 3), 1148-1152. -
NLM
Silva GK da, Gutierrez FRS, Guedes PM da M, Horta CV, Cunha LD da, Mineo TWP, Silva JS da, Kobayashi KS, Flavell RA, Silva JS da, Zamboni DS. Cutting edge: nucleotide binding oligomerization domain 1-dependent responses account for murine resistance against trypanosoma cruzi infection. Journal of Immunology. 2010 ; 184( 3): 1148-1152.[citado 2026 fev. 12 ] -
Vancouver
Silva GK da, Gutierrez FRS, Guedes PM da M, Horta CV, Cunha LD da, Mineo TWP, Silva JS da, Kobayashi KS, Flavell RA, Silva JS da, Zamboni DS. Cutting edge: nucleotide binding oligomerization domain 1-dependent responses account for murine resistance against trypanosoma cruzi infection. Journal of Immunology. 2010 ; 184( 3): 1148-1152.[citado 2026 fev. 12 ] - Critical role of ASC inflammasomes in host innate resistance to Trypanosoma cruzi infection
- Nucleotide-binding oligomerization domain-containing protein 2 prompts potent inflammatory stimuli during Neospora caninum infection
- Ros-dependent NLRP3-inflammasome activation is critical for restriction of Leishmania amazonensis infection
- Relevance of the myeloid differentiation factor 88 (MyD88) on RANKL, OPG, and nod expressions induced by TLR and IL-1R signaling in bone marrow stromal cells
- Role of NLRC4 inflammasome in Paracoccidioides brasiliensis pulmonary inflammation
- The inflammatose contributes for restriction of leishmania (Leishmania) amazonensis infection in macrophages and in vivo
- Resistance to Leishmania spp. is mediated by the inflammasome induced duo: IL- 1β and nitric oxide
- Restriction of Leishmania amazonensis infection by the inflammasomes
- NOD1 and NOD2 receptors account to innate and adaptive immune response against Leishmania infection
- NOD1/NOD2/RIP2 signaling in response to non-peptidoglycan containing pathogen contribute to development of a TH1-biased response and resistance against L. major infection
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas