Cutting edge: nucleotide binding oligomerization domain 1-dependent responses account for murine resistance against trypanosoma cruzi infection (2010)
- Authors:
- USP affiliated authors: SILVA, JOÃO SANTANA DA - FMRP ; ZAMBONI, DARIO SIMÕES - FMRP
- Unidade: FMRP
- Subjects: TRYPANOSOMA CRUZI; CITOCINAS
- Language: Inglês
- Imprenta:
- Source:
- Título: Journal of Immunology
- ISSN: 0022-1767
- Volume/Número/Paginação/Ano: v. 184, n. 3, p. 1148-1152, 2010
-
ABNT
SILVA, Grace Kelly da et al. Cutting edge: nucleotide binding oligomerization domain 1-dependent responses account for murine resistance against trypanosoma cruzi infection. Journal of Immunology, v. 184, n. 3, p. 1148-1152, 2010Tradução . . Acesso em: 28 dez. 2025. -
APA
Silva, G. K. da, Gutierrez, F. R. S., Guedes, P. M. da M., Horta, C. V., Cunha, L. D. da, Mineo, T. W. P., et al. (2010). Cutting edge: nucleotide binding oligomerization domain 1-dependent responses account for murine resistance against trypanosoma cruzi infection. Journal of Immunology, 184( 3), 1148-1152. -
NLM
Silva GK da, Gutierrez FRS, Guedes PM da M, Horta CV, Cunha LD da, Mineo TWP, Silva JS da, Kobayashi KS, Flavell RA, Silva JS da, Zamboni DS. Cutting edge: nucleotide binding oligomerization domain 1-dependent responses account for murine resistance against trypanosoma cruzi infection. Journal of Immunology. 2010 ; 184( 3): 1148-1152.[citado 2025 dez. 28 ] -
Vancouver
Silva GK da, Gutierrez FRS, Guedes PM da M, Horta CV, Cunha LD da, Mineo TWP, Silva JS da, Kobayashi KS, Flavell RA, Silva JS da, Zamboni DS. Cutting edge: nucleotide binding oligomerization domain 1-dependent responses account for murine resistance against trypanosoma cruzi infection. Journal of Immunology. 2010 ; 184( 3): 1148-1152.[citado 2025 dez. 28 ] - Ros-dependent NLRP3-inflammasome activation is critical for restriction of Leishmania amazonensis infection
- Critical role of ASC inflammasomes in host innate resistance to Trypanosoma cruzi infection
- NOD/RIP2 signaling contribute to development of a TH1 response and resistance against L. major infection
- Apoptosis-associated speck–like protein containing a caspase recruitment domain inflammasomes mediate IL-1β response and host resistance to Trypanosoma cruzi infection
- IFN-γ plays a unique role in protection against low virulent Trypanosoma cruzi strain
- A parent-of-origin effect determines the susceptibility of a non-informative F1 population to Trypanosoma cruzi infection in vivo
- Resistance to Leishmania spp. is mediated by the inflammasome induced duo: IL- 1β and nitric oxide
- NOD1/NOD2/RIP2 signaling in response to non-peptidoglycan containing pathogen contribute to development of a TH1-biased response and resistance against L. major infection
- Interplay between reactive oxygen species and the inflammasome are crucial for restriction of Neospora caninum replication
- The inflammatose contributes for restriction of leishmania (Leishmania) amazonensis infection in macrophages and in vivo
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