NOD1 and NOD2 receptors account to innate and adaptive immune response against Leishmania infection (2014)
- Authors:
- USP affiliated authors: SILVA, JOÃO SANTANA DA - FMRP ; ZAMBONI, DARIO SIMÕES - FMRP
- Unidade: FMRP
- Subjects: SISTEMA IMUNE; LEISHMANIA; RECEPTORES
- Language: Inglês
- Imprenta:
- Source:
- Título: Proceedings
- Conference titles: Congress of the Brazilian Society of Immunology - IMMUNO
-
ABNT
LIMA JUNIOR, Djalma de Souza e SILVA, João Santana da e ZAMBONI, Dario Simões. NOD1 and NOD2 receptors account to innate and adaptive immune response against Leishmania infection. 2014, Anais.. Buzios: SBI, 2014. . Acesso em: 29 mar. 2026. -
APA
Lima Junior, D. de S., Silva, J. S. da, & Zamboni, D. S. (2014). NOD1 and NOD2 receptors account to innate and adaptive immune response against Leishmania infection. In Proceedings. Buzios: SBI. -
NLM
Lima Junior D de S, Silva JS da, Zamboni DS. NOD1 and NOD2 receptors account to innate and adaptive immune response against Leishmania infection. Proceedings. 2014 ;[citado 2026 mar. 29 ] -
Vancouver
Lima Junior D de S, Silva JS da, Zamboni DS. NOD1 and NOD2 receptors account to innate and adaptive immune response against Leishmania infection. Proceedings. 2014 ;[citado 2026 mar. 29 ] - Critical role of ASC inflammasomes in host innate resistance to Trypanosoma cruzi infection
- Nucleotide-binding oligomerization domain-containing protein 2 prompts potent inflammatory stimuli during Neospora caninum infection
- Cutting edge: nucleotide binding oligomerization domain 1-dependent responses account for murine resistance against trypanosoma cruzi infection
- Ros-dependent NLRP3-inflammasome activation is critical for restriction of Leishmania amazonensis infection
- Relevance of the myeloid differentiation factor 88 (MyD88) on RANKL, OPG, and nod expressions induced by TLR and IL-1R signaling in bone marrow stromal cells
- Role of NLRC4 inflammasome in Paracoccidioides brasiliensis pulmonary inflammation
- The inflammatose contributes for restriction of leishmania (Leishmania) amazonensis infection in macrophages and in vivo
- Resistance to Leishmania spp. is mediated by the inflammasome induced duo: IL- 1β and nitric oxide
- Restriction of Leishmania amazonensis infection by the inflammasomes
- NOD1/NOD2/RIP2 signaling in response to non-peptidoglycan containing pathogen contribute to development of a TH1-biased response and resistance against L. major infection
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas