Ros-dependent NLRP3-inflammasome activation is critical for restriction of Leishmania amazonensis infection (2011)
- Authors:
- USP affiliated authors: SILVA, JOÃO SANTANA DA - FMRP ; ZAMBONI, DARIO SIMÕES - FMRP
- Unidade: FMRP
- Subjects: LEISHMANIA; BIOQUÍMICA
- Language: Inglês
- Imprenta:
- Publisher: SBI
- Publisher place: Foz do Iguaçu
- Date published: 2011
- Source:
- Título: Abstracts
- Conference titles: Congress of the Brazilian Society of Immunology (IMMUNO)
-
ABNT
LIMA JÚNIOR, Djalma de Souza et al. Ros-dependent NLRP3-inflammasome activation is critical for restriction of Leishmania amazonensis infection. 2011, Anais.. Foz do Iguaçu: SBI, 2011. . Acesso em: 21 jan. 2026. -
APA
Lima Júnior, D. de S., Costa, D. L., Carregaro, V., Cunha, L. D. da, Mineo, T. W. P., Gutierrez, F. R. S., et al. (2011). Ros-dependent NLRP3-inflammasome activation is critical for restriction of Leishmania amazonensis infection. In Abstracts. Foz do Iguaçu: SBI. -
NLM
Lima Júnior D de S, Costa DL, Carregaro V, Cunha LD da, Mineo TWP, Gutierrez FRS, Silva JS da, Zamboni DS. Ros-dependent NLRP3-inflammasome activation is critical for restriction of Leishmania amazonensis infection. Abstracts. 2011 ;[citado 2026 jan. 21 ] -
Vancouver
Lima Júnior D de S, Costa DL, Carregaro V, Cunha LD da, Mineo TWP, Gutierrez FRS, Silva JS da, Zamboni DS. Ros-dependent NLRP3-inflammasome activation is critical for restriction of Leishmania amazonensis infection. Abstracts. 2011 ;[citado 2026 jan. 21 ] - Relevance of the myeloid differentiation factor 88 (MyD88) on RANKL, OPG, and nod expressions induced by TLR and IL-1R signaling in bone marrow stromal cells
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- Role of NLRC4 inflammasome in Paracoccidioides brasiliensis pulmonary inflammation
- The inflammatose contributes for restriction of leishmania (Leishmania) amazonensis infection in macrophages and in vivo
- Restriction of Leishmania amazonensis infection by the inflammasomes
- Intrinsic expression of Nod2 in CD4+ T lymphocytes is not necessary for the development of cell-mediated immunity and host resistance to Toxoplasma gondii
- Cutting edge: nucleotide binding oligomerization domain 1-dependent responses account for murine resistance against trypanosoma cruzi infection
- The role of Rip2 in the signaling pathwy downstream of NOD1 in response to Trypanosoma cruz/infection
- Interplay between reactive oxygen species and the inflammasome are crucial for restriction of Neospora caninum replication
- A parent-of-origin effect determines the susceptibility of a non-informative F1 population to Trypanosoma cruzi infection in vivo
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