Restriction of Leishmania amazonensis infection by the inflammasomes (2010)
- Authors:
- USP affiliated authors: ZAMBONI, DARIO SIMÕES - FMRP ; SILVA, JOÃO SANTANA DA - FMRP
- Unidade: FMRP
- Subjects: LEISHMANIA; RECEPTORES
- Language: Inglês
- Imprenta:
- Source:
- Título: Proceedings
- Conference titles: Annual Meeting of the Brazilian Society of Protozoology
-
ABNT
LIMA JÚNIOR, Djalma de Souza et al. Restriction of Leishmania amazonensis infection by the inflammasomes. 2010, Anais.. São Paulo: SBPz, 2010. . Acesso em: 27 dez. 2025. -
APA
Lima Júnior, D. de S., Costa, D. L., Carregaro, V., Gutierrez, F. R. S., Mineo, T. W. P., Silva, J. S. da, & Zamboni, D. S. (2010). Restriction of Leishmania amazonensis infection by the inflammasomes. In Proceedings. São Paulo: SBPz. -
NLM
Lima Júnior D de S, Costa DL, Carregaro V, Gutierrez FRS, Mineo TWP, Silva JS da, Zamboni DS. Restriction of Leishmania amazonensis infection by the inflammasomes. Proceedings. 2010 ;[citado 2025 dez. 27 ] -
Vancouver
Lima Júnior D de S, Costa DL, Carregaro V, Gutierrez FRS, Mineo TWP, Silva JS da, Zamboni DS. Restriction of Leishmania amazonensis infection by the inflammasomes. Proceedings. 2010 ;[citado 2025 dez. 27 ] - Ros-dependent NLRP3-inflammasome activation is critical for restriction of Leishmania amazonensis infection
- Critical role of ASC inflammasomes in host innate resistance to Trypanosoma cruzi infection
- NOD/RIP2 signaling contribute to development of a TH1 response and resistance against L. major infection
- Apoptosis-associated speck–like protein containing a caspase recruitment domain inflammasomes mediate IL-1β response and host resistance to Trypanosoma cruzi infection
- A parent-of-origin effect determines the susceptibility of a non-informative F1 population to Trypanosoma cruzi infection in vivo
- IFN-γ plays a unique role in protection against low virulent Trypanosoma cruzi strain
- Cutting edge: nucleotide binding oligomerization domain 1-dependent responses account for murine resistance against trypanosoma cruzi infection
- Resistance to Leishmania spp. is mediated by the inflammasome induced duo: IL- 1β and nitric oxide
- NOD1/NOD2/RIP2 signaling in response to non-peptidoglycan containing pathogen contribute to development of a TH1-biased response and resistance against L. major infection
- Interplay between reactive oxygen species and the inflammasome are crucial for restriction of Neospora caninum replication
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