NOD1/NOD2/RIP2 signaling in response to non-peptidoglycan containing pathogen contribute to development of a TH1-biased response and resistance against L. major infection (2015)
- Authors:
- USP affiliated authors: ZAMBONI, DARIO SIMÕES - FMRP ; SILVA, JOÃO SANTANA DA - FMRP
- Unidade: FMRP
- Subjects: LEISHMANIA; CITOCINAS; RECEPTORES CELULARES; PROTEÍNAS
- Language: Inglês
- Imprenta:
- Source:
- Título: Abstracts
- Conference titles: INNATE IMMUNITY 2015
-
ABNT
LIMA JÚNIOR, Djalma de Souza e SILVA, João Santana da e ZAMBONI, Dario Simões. NOD1/NOD2/RIP2 signaling in response to non-peptidoglycan containing pathogen contribute to development of a TH1-biased response and resistance against L. major infection. 2015, Anais.. Guarujá: SBI, 2015. Disponível em: https://icongresso.itarget.com.br/useradm/anais/?clt=imu.3&lng=I. Acesso em: 27 dez. 2025. -
APA
Lima Júnior, D. de S., Silva, J. S. da, & Zamboni, D. S. (2015). NOD1/NOD2/RIP2 signaling in response to non-peptidoglycan containing pathogen contribute to development of a TH1-biased response and resistance against L. major infection. In Abstracts. Guarujá: SBI. Recuperado de https://icongresso.itarget.com.br/useradm/anais/?clt=imu.3&lng=I -
NLM
Lima Júnior D de S, Silva JS da, Zamboni DS. NOD1/NOD2/RIP2 signaling in response to non-peptidoglycan containing pathogen contribute to development of a TH1-biased response and resistance against L. major infection [Internet]. Abstracts. 2015 ;[citado 2025 dez. 27 ] Available from: https://icongresso.itarget.com.br/useradm/anais/?clt=imu.3&lng=I -
Vancouver
Lima Júnior D de S, Silva JS da, Zamboni DS. NOD1/NOD2/RIP2 signaling in response to non-peptidoglycan containing pathogen contribute to development of a TH1-biased response and resistance against L. major infection [Internet]. Abstracts. 2015 ;[citado 2025 dez. 27 ] Available from: https://icongresso.itarget.com.br/useradm/anais/?clt=imu.3&lng=I - Ros-dependent NLRP3-inflammasome activation is critical for restriction of Leishmania amazonensis infection
- Critical role of ASC inflammasomes in host innate resistance to Trypanosoma cruzi infection
- NOD/RIP2 signaling contribute to development of a TH1 response and resistance against L. major infection
- Apoptosis-associated speck–like protein containing a caspase recruitment domain inflammasomes mediate IL-1β response and host resistance to Trypanosoma cruzi infection
- IFN-γ plays a unique role in protection against low virulent Trypanosoma cruzi strain
- A parent-of-origin effect determines the susceptibility of a non-informative F1 population to Trypanosoma cruzi infection in vivo
- Cutting edge: nucleotide binding oligomerization domain 1-dependent responses account for murine resistance against trypanosoma cruzi infection
- Resistance to Leishmania spp. is mediated by the inflammasome induced duo: IL- 1β and nitric oxide
- Interplay between reactive oxygen species and the inflammasome are crucial for restriction of Neospora caninum replication
- The inflammatose contributes for restriction of leishmania (Leishmania) amazonensis infection in macrophages and in vivo
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
