Role of NLRC4 inflammasome in Paracoccidioides brasiliensis pulmonary inflammation (2015)
- Authors:
- USP affiliated authors: ZAMBONI, DARIO SIMÕES - FMRP ; SILVA, JOÃO SANTANA DA - FMRP
- Unidade: FMRP
- Subjects: PARACOCCIDIOIDES BRASILIENSIS; PROTEÍNAS; RECEPTORES
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Abstracts
- Conference titles: INNATE IMMUNITY 2015
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ABNT
SOUZA, Camila de Oliveira Silva e et al. Role of NLRC4 inflammasome in Paracoccidioides brasiliensis pulmonary inflammation. 2015, Anais.. Guarujá: SBI, 2015. Disponível em: https://icongresso.itarget.com.br/useradm/anais/?clt=imu.3&lng=I. Acesso em: 24 abr. 2024. -
APA
Souza, C. de O. S. e, Carneiro, N. K., Oliveira, G. G., Silva, G. K. da, Zamboni, D. S., & Silva, J. S. da. (2015). Role of NLRC4 inflammasome in Paracoccidioides brasiliensis pulmonary inflammation. In Abstracts. Guarujá: SBI. Recuperado de https://icongresso.itarget.com.br/useradm/anais/?clt=imu.3&lng=I -
NLM
Souza C de OS e, Carneiro NK, Oliveira GG, Silva GK da, Zamboni DS, Silva JS da. Role of NLRC4 inflammasome in Paracoccidioides brasiliensis pulmonary inflammation [Internet]. Abstracts. 2015 ;[citado 2024 abr. 24 ] Available from: https://icongresso.itarget.com.br/useradm/anais/?clt=imu.3&lng=I -
Vancouver
Souza C de OS e, Carneiro NK, Oliveira GG, Silva GK da, Zamboni DS, Silva JS da. Role of NLRC4 inflammasome in Paracoccidioides brasiliensis pulmonary inflammation [Internet]. Abstracts. 2015 ;[citado 2024 abr. 24 ] Available from: https://icongresso.itarget.com.br/useradm/anais/?clt=imu.3&lng=I - Ros-dependent NLRP3-inflammasome activation is critical for restriction of Leishmania amazonensis infection
- Critical role of ASC inflammasomes in host innate resistance to Trypanosoma cruzi infection
- NOD/RIP2 signaling contribute to development of a TH1 response and resistance against L. major infection
- Apoptosis-associated speck–like protein containing a caspase recruitment domain inflammasomes mediate IL-1β response and host resistance to Trypanosoma cruzi infection
- A parent-of-origin effect determines the susceptibility of a non-informative F1 population to Trypanosoma cruzi infection in vivo
- IFN-γ plays a unique role in protection against low virulent Trypanosoma cruzi strain
- Cutting edge: nucleotide binding oligomerization domain 1-dependent responses account for murine resistance against trypanosoma cruzi infection
- Resistance to Leishmania spp. is mediated by the inflammasome induced duo: IL- 1β and nitric oxide
- NOD1/NOD2/RIP2 signaling in response to non-peptidoglycan containing pathogen contribute to development of a TH1-biased response and resistance against L. major infection
- Interplay between reactive oxygen species and the inflammasome are crucial for restriction of Neospora caninum replication
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