IFN-γ plays a unique role in protection against low virulent Trypanosoma cruzi strain (2012)
- Authors:
- USP affiliated authors: ZAMBONI, DARIO SIMÕES - FMRP ; SILVA, JOÃO SANTANA DA - FMRP
- Unidade: FMRP
- DOI: 10.1371/journal.pntd.0001598
- Subjects: TRYPANOSOMA CRUZI (VIROLOGIA); VIRULÊNCIA; DOENÇA DE CHAGAS; AMASTIGOTES (ANÁLISE); INFECÇÕES POR PROTOZOÁRIOS (TÉCNICAS MICROSCÓPICAS)
- Language: Inglês
- Imprenta:
- Publisher place: San Francisco
- Date published: 2012
- Source:
- Título: PLoS Neglected Tropical Diseases
- ISSN: 1935-2727
- Volume/Número/Paginação/Ano: v. 6, n. 4, art. e1598, 2012
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
RODRIGUES, Adele A. et al. IFN-γ plays a unique role in protection against low virulent Trypanosoma cruzi strain. PLoS Neglected Tropical Diseases, v. 6, n. 4, 2012Tradução . . Disponível em: https://doi.org/10.1371/journal.pntd.0001598. Acesso em: 27 dez. 2025. -
APA
Rodrigues, A. A., Saosa, J. S. S., Silva, G. K. da, Martins, F. A., Silva, A. A. da, Souza Neto, C. P. da S., et al. (2012). IFN-γ plays a unique role in protection against low virulent Trypanosoma cruzi strain. PLoS Neglected Tropical Diseases, 6( 4). doi:10.1371/journal.pntd.0001598 -
NLM
Rodrigues AA, Saosa JSS, Silva GK da, Martins FA, Silva AA da, Souza Neto CP da S, Horta CV, Zamboni DS, Silva JS da, Ferro EAV, Silva CV da. IFN-γ plays a unique role in protection against low virulent Trypanosoma cruzi strain [Internet]. PLoS Neglected Tropical Diseases. 2012 ; 6( 4):[citado 2025 dez. 27 ] Available from: https://doi.org/10.1371/journal.pntd.0001598 -
Vancouver
Rodrigues AA, Saosa JSS, Silva GK da, Martins FA, Silva AA da, Souza Neto CP da S, Horta CV, Zamboni DS, Silva JS da, Ferro EAV, Silva CV da. IFN-γ plays a unique role in protection against low virulent Trypanosoma cruzi strain [Internet]. PLoS Neglected Tropical Diseases. 2012 ; 6( 4):[citado 2025 dez. 27 ] Available from: https://doi.org/10.1371/journal.pntd.0001598 - Ros-dependent NLRP3-inflammasome activation is critical for restriction of Leishmania amazonensis infection
- Critical role of ASC inflammasomes in host innate resistance to Trypanosoma cruzi infection
- NOD/RIP2 signaling contribute to development of a TH1 response and resistance against L. major infection
- Apoptosis-associated speck–like protein containing a caspase recruitment domain inflammasomes mediate IL-1β response and host resistance to Trypanosoma cruzi infection
- A parent-of-origin effect determines the susceptibility of a non-informative F1 population to Trypanosoma cruzi infection in vivo
- Cutting edge: nucleotide binding oligomerization domain 1-dependent responses account for murine resistance against trypanosoma cruzi infection
- Resistance to Leishmania spp. is mediated by the inflammasome induced duo: IL- 1β and nitric oxide
- NOD1/NOD2/RIP2 signaling in response to non-peptidoglycan containing pathogen contribute to development of a TH1-biased response and resistance against L. major infection
- Interplay between reactive oxygen species and the inflammasome are crucial for restriction of Neospora caninum replication
- The inflammatose contributes for restriction of leishmania (Leishmania) amazonensis infection in macrophages and in vivo
Informações sobre o DOI: 10.1371/journal.pntd.0001598 (Fonte: oaDOI API)
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