A parent-of-origin effect determines the susceptibility of a non-informative F1 population to Trypanosoma cruzi infection in vivo (2013)
- Authors:
- USP affiliated authors: SILVA, JOÃO SANTANA DA - FMRP ; ZAMBONI, DARIO SIMÕES - FMRP
- Unidade: FMRP
- DOI: 10.1371/journal.pone.0056347
- Subjects: DOENÇA DE CHAGAS (GENÉTICA); HIBRIDIZAÇÃO; TRYPANOSOMA CRUZI; CROMOSSOMO X
- Language: Inglês
- Imprenta:
- Publisher place: San Francisco
- Date published: 2013
- Source:
- Este artigo possui versão em acesso aberto
- URL de acesso aberto
- PDF de acesso aberto
- Versão do Documento: Versão publicada (Published version)
-
Status: Artigo publicado em periódico de acesso aberto (Gold Open Access) -
ABNT
SILVA, Grace Kelly da et al. A parent-of-origin effect determines the susceptibility of a non-informative F1 population to Trypanosoma cruzi infection in vivo. PLoS One, v. 8, n. 2, p. e56347-1 - e56347-10, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0056347. Acesso em: 13 mar. 2026. -
APA
Silva, G. K. da, Cunha, L. D. da, Horta, C. V., Silva, A. L. N., Gutierrez, F. R. S., Silva, J. S. da, & Zamboni, D. S. (2013). A parent-of-origin effect determines the susceptibility of a non-informative F1 population to Trypanosoma cruzi infection in vivo. PLoS One, 8( 2), e56347-1 - e56347-10. doi:10.1371/journal.pone.0056347 -
NLM
Silva GK da, Cunha LD da, Horta CV, Silva ALN, Gutierrez FRS, Silva JS da, Zamboni DS. A parent-of-origin effect determines the susceptibility of a non-informative F1 population to Trypanosoma cruzi infection in vivo [Internet]. PLoS One. 2013 ; 8( 2): e56347-1 - e56347-10.[citado 2026 mar. 13 ] Available from: https://doi.org/10.1371/journal.pone.0056347 -
Vancouver
Silva GK da, Cunha LD da, Horta CV, Silva ALN, Gutierrez FRS, Silva JS da, Zamboni DS. A parent-of-origin effect determines the susceptibility of a non-informative F1 population to Trypanosoma cruzi infection in vivo [Internet]. PLoS One. 2013 ; 8( 2): e56347-1 - e56347-10.[citado 2026 mar. 13 ] Available from: https://doi.org/10.1371/journal.pone.0056347 - Critical role of ASC inflammasomes in host innate resistance to Trypanosoma cruzi infection
- Nucleotide-binding oligomerization domain-containing protein 2 prompts potent inflammatory stimuli during Neospora caninum infection
- Cutting edge: nucleotide binding oligomerization domain 1-dependent responses account for murine resistance against trypanosoma cruzi infection
- Ros-dependent NLRP3-inflammasome activation is critical for restriction of Leishmania amazonensis infection
- Relevance of the myeloid differentiation factor 88 (MyD88) on RANKL, OPG, and nod expressions induced by TLR and IL-1R signaling in bone marrow stromal cells
- Role of NLRC4 inflammasome in Paracoccidioides brasiliensis pulmonary inflammation
- The inflammatose contributes for restriction of leishmania (Leishmania) amazonensis infection in macrophages and in vivo
- Resistance to Leishmania spp. is mediated by the inflammasome induced duo: IL- 1β and nitric oxide
- Restriction of Leishmania amazonensis infection by the inflammasomes
- NOD1 and NOD2 receptors account to innate and adaptive immune response against Leishmania infection
Informações sobre a disponibilidade de versões do artigo em acesso aberto coletadas automaticamente via oaDOI API (Unpaywall).
Download do texto completo
| Tipo | Nome | Link | |
|---|---|---|---|
| 002430055.pdf | Direct link |
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas