Interplay between reactive oxygen species and the inflammasome are crucial for restriction of Neospora caninum replication (2020)
- Authors:
- USP affiliated authors: SILVA, JOÃO SANTANA DA - FMRP ; ZAMBONI, DARIO SIMÕES - FMRP
- Unidade: FMRP
- DOI: 10.3389/fcimb.2020.00243
- Subjects: INTERLEUCINA 1; NEOSPORA CANINUM; ESPÉCIES REATIVAS DE OXIGÊNIO
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Frontiers in Cellular and Infection Microbiology
- ISSN: 2235-2988
- Volume/Número/Paginação/Ano: v. 10, art. 243, 2020
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
MOTA, Caroline M. et al. Interplay between reactive oxygen species and the inflammasome are crucial for restriction of Neospora caninum replication. Frontiers in Cellular and Infection Microbiology, v. 10, 2020Tradução . . Disponível em: https://doi.org/10.3389/fcimb.2020.00243. Acesso em: 12 fev. 2026. -
APA
Mota, C. M., Lima Júnior, D. de S., França, F. B. F., Torres, J. D. A., Barros, P. da S. C., Santiago, F. M., et al. (2020). Interplay between reactive oxygen species and the inflammasome are crucial for restriction of Neospora caninum replication. Frontiers in Cellular and Infection Microbiology, 10. doi:10.3389/fcimb.2020.00243 -
NLM
Mota CM, Lima Júnior D de S, França FBF, Torres JDA, Barros P da SC, Santiago FM, Silva JS da, Mineo JR, Zamboni DS, Mineo TWP. Interplay between reactive oxygen species and the inflammasome are crucial for restriction of Neospora caninum replication [Internet]. Frontiers in Cellular and Infection Microbiology. 2020 ; 10[citado 2026 fev. 12 ] Available from: https://doi.org/10.3389/fcimb.2020.00243 -
Vancouver
Mota CM, Lima Júnior D de S, França FBF, Torres JDA, Barros P da SC, Santiago FM, Silva JS da, Mineo JR, Zamboni DS, Mineo TWP. Interplay between reactive oxygen species and the inflammasome are crucial for restriction of Neospora caninum replication [Internet]. Frontiers in Cellular and Infection Microbiology. 2020 ; 10[citado 2026 fev. 12 ] Available from: https://doi.org/10.3389/fcimb.2020.00243 - Critical role of ASC inflammasomes in host innate resistance to Trypanosoma cruzi infection
- Nucleotide-binding oligomerization domain-containing protein 2 prompts potent inflammatory stimuli during Neospora caninum infection
- Cutting edge: nucleotide binding oligomerization domain 1-dependent responses account for murine resistance against trypanosoma cruzi infection
- Ros-dependent NLRP3-inflammasome activation is critical for restriction of Leishmania amazonensis infection
- Relevance of the myeloid differentiation factor 88 (MyD88) on RANKL, OPG, and nod expressions induced by TLR and IL-1R signaling in bone marrow stromal cells
- Role of NLRC4 inflammasome in Paracoccidioides brasiliensis pulmonary inflammation
- The inflammatose contributes for restriction of leishmania (Leishmania) amazonensis infection in macrophages and in vivo
- Resistance to Leishmania spp. is mediated by the inflammasome induced duo: IL- 1β and nitric oxide
- Restriction of Leishmania amazonensis infection by the inflammasomes
- NOD1 and NOD2 receptors account to innate and adaptive immune response against Leishmania infection
Informações sobre o DOI: 10.3389/fcimb.2020.00243 (Fonte: oaDOI API)
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