The left lung is preferentially targeted during experimental paracoccidioidomycosis in C57BL/6 mice (2013)
- Authors:
- USP affiliated authors: ROSSI, MARCOS ANTONIO - FMRP ; SILVA, JOÃO SANTANA DA - FMRP
- Unidade: FMRP
- DOI: 10.1590/1414-431X20133140
- Subjects: PULMÃO; PARACOCCIDIOIDES BRASILIENSIS; MODELOS ANIMAIS
- Language: Inglês
- Imprenta:
- Publisher place: Ribeirão Preto
- Date published: 2013
- Source:
- Título: Brazilian Journal of Medical and Biological Research
- ISSN: 1414-431X
- Volume/Número/Paginação/Ano: v. 46, n. 10, p. 839-843, 2013
- Status:
- Artigo publicado em periódico de acesso aberto (Gold Open Access)
- Versão do Documento:
- Versão publicada (Published version)
- Acessar versão aberta:
-
ABNT
TRISTÃO, Fabrine Sales Massafera et al. The left lung is preferentially targeted during experimental paracoccidioidomycosis in C57BL/6 mice. Brazilian Journal of Medical and Biological Research, v. 46, n. 10, p. 839-843, 2013Tradução . . Disponível em: https://doi.org/10.1590/1414-431X20133140. Acesso em: 01 abr. 2026. -
APA
Tristão, F. S. M., Rocha, F. A., Dias, F. C., Rossi, M. A., & Silva, J. S. da. (2013). The left lung is preferentially targeted during experimental paracoccidioidomycosis in C57BL/6 mice. Brazilian Journal of Medical and Biological Research, 46( 10), 839-843. doi:10.1590/1414-431X20133140 -
NLM
Tristão FSM, Rocha FA, Dias FC, Rossi MA, Silva JS da. The left lung is preferentially targeted during experimental paracoccidioidomycosis in C57BL/6 mice [Internet]. Brazilian Journal of Medical and Biological Research. 2013 ; 46( 10): 839-843.[citado 2026 abr. 01 ] Available from: https://doi.org/10.1590/1414-431X20133140 -
Vancouver
Tristão FSM, Rocha FA, Dias FC, Rossi MA, Silva JS da. The left lung is preferentially targeted during experimental paracoccidioidomycosis in C57BL/6 mice [Internet]. Brazilian Journal of Medical and Biological Research. 2013 ; 46( 10): 839-843.[citado 2026 abr. 01 ] Available from: https://doi.org/10.1590/1414-431X20133140 - nitric oxide synthase-2 modulates chemokine production by Trypanosoma Cruzi-infected cardiac myocytes
- B cell-deficient mice display enhanced susceptibility to Paracoccidioides brasiliensis infection
- Early dystrophin disruption in the pathogenesis of experimental chronic chagas cardiomyopathy
- Intercellular adhesion molecule 1 deficieney leads to impaired recruitment of T lymphocytes and enhanced host susceptibility to infection with Trypanosoma cruzi
- Early dystrophin disruption in the pathogenesis of experimental chronic Chagas cardiomyopathy
- Role of interleukin (IL)-18 in experimental paracoccidioidomycosis
- Anti- 'CD IND.4' abrogates rejection and restabliches long-term tolerance to sybgenetic newborn hearts grafted in mice chronically infected with trypanosoma cruzi
- Intensification of acute t. Cruzi myocarditis in balb / c nice pretreated
- Permeability alteration of the sarcolemmal membrane particulary at the site of macrophage contact, in experimental chronic trypanosoma cruzi miocarditis in mice
- Haeme oxygenase activity protects the host against excessive cardiac inflammation during experimental Trypanosoma cruzi infection
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