Early dystrophin disruption in the pathogenesis of experimental chronic chagas cardiomyopathy (2011)
- Authors:
- USP affiliated authors: ROSSI, MARCOS ANTONIO - FMRP ; SILVA, JOÃO SANTANA DA - FMRP
- Unidade: FMRP
- Subjects: MIOCARDIOPATIA CHAGÁSICA; FÁRMACOS
- Language: Inglês
- Imprenta:
- Publisher place: Beverly Hills
- Date published: 2011
- Source:
- Título: The Journal of Heart Disease
- Volume/Número/Paginação/Ano: v. 8, n. 1, p.108, res.429, 2011
- Conference titles: World Congress on Heart Disease
-
ABNT
PRADO, C. M. et al. Early dystrophin disruption in the pathogenesis of experimental chronic chagas cardiomyopathy. The Journal of Heart Disease. Beverly Hills: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 13 fev. 2026. , 2011 -
APA
Prado, C. M., Celes, M. R. N., Malvestio, L. M., Silva, J. S., Jelicks, L. A., Tanowitz, H. B., & Rossi, M. A. (2011). Early dystrophin disruption in the pathogenesis of experimental chronic chagas cardiomyopathy. The Journal of Heart Disease. Beverly Hills: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. -
NLM
Prado CM, Celes MRN, Malvestio LM, Silva JS, Jelicks LA, Tanowitz HB, Rossi MA. Early dystrophin disruption in the pathogenesis of experimental chronic chagas cardiomyopathy. The Journal of Heart Disease. 2011 ; 8( 1): 108.[citado 2026 fev. 13 ] -
Vancouver
Prado CM, Celes MRN, Malvestio LM, Silva JS, Jelicks LA, Tanowitz HB, Rossi MA. Early dystrophin disruption in the pathogenesis of experimental chronic chagas cardiomyopathy. The Journal of Heart Disease. 2011 ; 8( 1): 108.[citado 2026 fev. 13 ] - nitric oxide synthase-2 modulates chemokine production by Trypanosoma Cruzi-infected cardiac myocytes
- B cell-deficient mice display enhanced susceptibility to Paracoccidioides brasiliensis infection
- Intercellular adhesion molecule 1 deficieney leads to impaired recruitment of T lymphocytes and enhanced host susceptibility to infection with Trypanosoma cruzi
- Early dystrophin disruption in the pathogenesis of experimental chronic Chagas cardiomyopathy
- Role of interleukin (IL)-18 in experimental paracoccidioidomycosis
- Anti- 'CD IND.4' abrogates rejection and restabliches long-term tolerance to sybgenetic newborn hearts grafted in mice chronically infected with trypanosoma cruzi
- Intensification of acute t. Cruzi myocarditis in balb / c nice pretreated
- Permeability alteration of the sarcolemmal membrane particulary at the site of macrophage contact, in experimental chronic trypanosoma cruzi miocarditis in mice
- The left lung is preferentially targeted during experimental paracoccidioidomycosis in C57BL/6 mice
- Haeme oxygenase activity protects the host against excessive cardiac inflammation during experimental Trypanosoma cruzi infection
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