Intensification of acute t. Cruzi myocarditis in balb / c nice pretreated (1990)
- Authors:
- USP affiliated authors: ROSSI, MARCOS ANTONIO - FMRP ; SILVA, JOAO SANTANA DA - FMRP
- Unidade: FMRP
- Assunto: PARASITOLOGIA
- Language: Inglês
- Source:
- Título: Journal of Experimental Pathology
- Volume/Número/Paginação/Ano: v.71, n.1 , p.33-9, 1990
-
ABNT
SILVA, João Santana da e ROSSI, Marcos Antonio. Intensification of acute t. Cruzi myocarditis in balb / c nice pretreated. Journal of Experimental Pathology, v. 71, n. 1 , p. 33-9, 1990Tradução . . Acesso em: 13 fev. 2026. -
APA
Silva, J. S. da, & Rossi, M. A. (1990). Intensification of acute t. Cruzi myocarditis in balb / c nice pretreated. Journal of Experimental Pathology, 71( 1 ), 33-9. -
NLM
Silva JS da, Rossi MA. Intensification of acute t. Cruzi myocarditis in balb / c nice pretreated. Journal of Experimental Pathology. 1990 ;71( 1 ): 33-9.[citado 2026 fev. 13 ] -
Vancouver
Silva JS da, Rossi MA. Intensification of acute t. Cruzi myocarditis in balb / c nice pretreated. Journal of Experimental Pathology. 1990 ;71( 1 ): 33-9.[citado 2026 fev. 13 ] - nitric oxide synthase-2 modulates chemokine production by Trypanosoma Cruzi-infected cardiac myocytes
- B cell-deficient mice display enhanced susceptibility to Paracoccidioides brasiliensis infection
- Early dystrophin disruption in the pathogenesis of experimental chronic chagas cardiomyopathy
- Intercellular adhesion molecule 1 deficieney leads to impaired recruitment of T lymphocytes and enhanced host susceptibility to infection with Trypanosoma cruzi
- Early dystrophin disruption in the pathogenesis of experimental chronic Chagas cardiomyopathy
- Role of interleukin (IL)-18 in experimental paracoccidioidomycosis
- Anti- 'CD IND.4' abrogates rejection and restabliches long-term tolerance to sybgenetic newborn hearts grafted in mice chronically infected with trypanosoma cruzi
- Permeability alteration of the sarcolemmal membrane particulary at the site of macrophage contact, in experimental chronic trypanosoma cruzi miocarditis in mice
- The left lung is preferentially targeted during experimental paracoccidioidomycosis in C57BL/6 mice
- Haeme oxygenase activity protects the host against excessive cardiac inflammation during experimental Trypanosoma cruzi infection
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