Permeability alteration of the sarcolemmal membrane particulary at the site of macrophage contact, in experimental chronic trypanosoma cruzi miocarditis in mice (1990)
- Authors:
- USP affiliated authors: ROSSI, MARCOS ANTONIO - FMRP ; SILVA, JOAO SANTANA DA - FMRP
- Unidade: FMRP
- Assunto: PATOLOGIA
- Language: Inglês
- Source:
- Título: International Journal of Experimental Pathology
- Volume/Número/Paginação/Ano: v.71, p.545-5, 1990
-
ABNT
ROSSI, Marcos Antonio e SILVA, João Santana da. Permeability alteration of the sarcolemmal membrane particulary at the site of macrophage contact, in experimental chronic trypanosoma cruzi miocarditis in mice. International Journal of Experimental Pathology, v. 71, p. 545-5, 1990Tradução . . Acesso em: 12 fev. 2026. -
APA
Rossi, M. A., & Silva, J. S. da. (1990). Permeability alteration of the sarcolemmal membrane particulary at the site of macrophage contact, in experimental chronic trypanosoma cruzi miocarditis in mice. International Journal of Experimental Pathology, 71, 545-5. -
NLM
Rossi MA, Silva JS da. Permeability alteration of the sarcolemmal membrane particulary at the site of macrophage contact, in experimental chronic trypanosoma cruzi miocarditis in mice. International Journal of Experimental Pathology. 1990 ;71 545-5.[citado 2026 fev. 12 ] -
Vancouver
Rossi MA, Silva JS da. Permeability alteration of the sarcolemmal membrane particulary at the site of macrophage contact, in experimental chronic trypanosoma cruzi miocarditis in mice. International Journal of Experimental Pathology. 1990 ;71 545-5.[citado 2026 fev. 12 ] - nitric oxide synthase-2 modulates chemokine production by Trypanosoma Cruzi-infected cardiac myocytes
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- Early dystrophin disruption in the pathogenesis of experimental chronic chagas cardiomyopathy
- Intercellular adhesion molecule 1 deficieney leads to impaired recruitment of T lymphocytes and enhanced host susceptibility to infection with Trypanosoma cruzi
- Early dystrophin disruption in the pathogenesis of experimental chronic Chagas cardiomyopathy
- Role of interleukin (IL)-18 in experimental paracoccidioidomycosis
- Anti- 'CD IND.4' abrogates rejection and restabliches long-term tolerance to sybgenetic newborn hearts grafted in mice chronically infected with trypanosoma cruzi
- Intensification of acute t. Cruzi myocarditis in balb / c nice pretreated
- The left lung is preferentially targeted during experimental paracoccidioidomycosis in C57BL/6 mice
- Haeme oxygenase activity protects the host against excessive cardiac inflammation during experimental Trypanosoma cruzi infection
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