Permeability alteration of the sarcolemmal membrane particulary at the site of macrophage contact, in experimental chronic trypanosoma cruzi miocarditis in mice (1990)
- Authors:
- USP affiliated authors: ROSSI, MARCOS ANTONIO - FMRP ; SILVA, JOAO SANTANA DA - FMRP
- Unidade: FMRP
- Assunto: PATOLOGIA
- Language: Inglês
- Source:
- Título: International Journal of Experimental Pathology
- Volume/Número/Paginação/Ano: v.71, p.545-5, 1990
-
ABNT
ROSSI, Marcos Antonio e SILVA, João Santana da. Permeability alteration of the sarcolemmal membrane particulary at the site of macrophage contact, in experimental chronic trypanosoma cruzi miocarditis in mice. International Journal of Experimental Pathology, v. 71, p. 545-5, 1990Tradução . . Acesso em: 12 out. 2024. -
APA
Rossi, M. A., & Silva, J. S. da. (1990). Permeability alteration of the sarcolemmal membrane particulary at the site of macrophage contact, in experimental chronic trypanosoma cruzi miocarditis in mice. International Journal of Experimental Pathology, 71, 545-5. -
NLM
Rossi MA, Silva JS da. Permeability alteration of the sarcolemmal membrane particulary at the site of macrophage contact, in experimental chronic trypanosoma cruzi miocarditis in mice. International Journal of Experimental Pathology. 1990 ;71 545-5.[citado 2024 out. 12 ] -
Vancouver
Rossi MA, Silva JS da. Permeability alteration of the sarcolemmal membrane particulary at the site of macrophage contact, in experimental chronic trypanosoma cruzi miocarditis in mice. International Journal of Experimental Pathology. 1990 ;71 545-5.[citado 2024 out. 12 ] - Anti- 'CD IND.4' abrogates rejection and restabliches long-term tolerance to sybgenetic newborn hearts grafted in mice chronically infected with trypanosoma cruzi
- Intercellular adhesion molecule 1 deficieney leads to impaired recruitment of T lymphocytes and enhanced host susceptibility to infection with Trypanosoma cruzi
- Role of interleukin (IL)-18 in experimental paracoccidioidomycosis
- B cell-deficient mice display enhanced susceptibility to Paracoccidioides brasiliensis infection
- nitric oxide synthase-2 modulates chemokine production by Trypanosoma Cruzi-infected cardiac myocytes
- Early dystrophin disruption in the pathogenesis of experimental chronic chagas cardiomyopathy
- Early dystrophin disruption in the pathogenesis of experimental chronic Chagas cardiomyopathy
- Intensification of acute t. Cruzi myocarditis in balb / c nice pretreated
- The left lung is preferentially targeted during experimental paracoccidioidomycosis in C57BL/6 mice
- Studies on the role of citokines and chemokines in the pathogenesis of coxsackievirus B5 (COX B5) in suckling mice infected by per-oral (PO)route
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
