Intercellular adhesion molecule 1 deficieney leads to impaired recruitment of T lymphocytes and enhanced host susceptibility to infection with Trypanosoma cruzi (2004)
- Authors:
- USP affiliated authors: ROSSI, MARCOS ANTONIO - FMRP ; SILVA, JOAO SANTANA DA - FMRP
- Unidade: FMRP
- Assunto: TRYPANOSOMA CRUZI (IMUNOLOGIA)
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: The Journal of Immunology
- ISSN: 0022-1767
- Volume/Número/Paginação/Ano: v. 173, p. 463-470, 2004
-
ABNT
MICHAILOWSKY, Vladimir; CELES, Mara Rúbia Nunes; MARINO, Ana P.; et al. Intercellular adhesion molecule 1 deficieney leads to impaired recruitment of T lymphocytes and enhanced host susceptibility to infection with Trypanosoma cruzi. The Journal of Immunology, Bethesda, v. 173, p. 463-470, 2004. -
APA
Michailowsky, V., Celes, M. R. N., Marino, A. P., Silva, A. A., Vieira, L. Q., Rossi, M. A., et al. (2004). Intercellular adhesion molecule 1 deficieney leads to impaired recruitment of T lymphocytes and enhanced host susceptibility to infection with Trypanosoma cruzi. The Journal of Immunology, 173, 463-470. -
NLM
Michailowsky V, Celes MRN, Marino AP, Silva AA, Vieira LQ, Rossi MA, Gazzinelli RT, Lannes-Vieira J, Silva JS da. Intercellular adhesion molecule 1 deficieney leads to impaired recruitment of T lymphocytes and enhanced host susceptibility to infection with Trypanosoma cruzi. The Journal of Immunology. 2004 ; 173 463-470. -
Vancouver
Michailowsky V, Celes MRN, Marino AP, Silva AA, Vieira LQ, Rossi MA, Gazzinelli RT, Lannes-Vieira J, Silva JS da. Intercellular adhesion molecule 1 deficieney leads to impaired recruitment of T lymphocytes and enhanced host susceptibility to infection with Trypanosoma cruzi. The Journal of Immunology. 2004 ; 173 463-470. - Early dystrophin disruption in the pathogenesis of experimental chronic chagas cardiomyopathy
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- B cell-deficient mice display enhanced susceptibility to Paracoccidioides brasiliensis infection
- Role of interleukin (IL)-18 in experimental paracoccidioidomycosis
- Anti- 'CD IND.4' abrogates rejection and restabliches long-term tolerance to sybgenetic newborn hearts grafted in mice chronically infected with trypanosoma cruzi
- The left lung is preferentially targeted during experimental paracoccidioidomycosis in C57BL/6 mice
- Intensification of acute t. Cruzi myocarditis in balb / c nice pretreated
- Permeability alteration of the sarcolemmal membrane particulary at the site of macrophage contact, in experimental chronic trypanosoma cruzi miocarditis in mice
- 5-lipoxygenase activity increases susceptibility to experimental paracoccidioides brasiliensis infection
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