nitric oxide synthase-2 modulates chemokine production by Trypanosoma Cruzi-infected cardiac myocytes (2008)
- Authors:
- USP affiliated authors: ROSSI, MARCOS ANTONIO - FMRP ; SILVA, JOÃO SANTANA DA - FMRP
- Unidade: FMRP
- DOI: 10.1016/j.micinf.2008.09.009
- Subjects: TRYPANOSOMA CRUZI; MIOCARDITE; ÓXIDO NÍTRICO (EFEITOS)
- Language: Inglês
- Imprenta:
- Source:
- Título: Microbes and Infection
- ISSN: 1286-4579
- Volume/Número/Paginação/Ano: v. 10, n. 14-15, p. 1558-1566, 2008
- Status:
- Artigo possui versão em acesso aberto em repositório (Green Open Access)
- Versão do Documento:
- Versão submetida (Pré-print)
- Acessar versão aberta:
-
ABNT
MACHADO, Fabiana Simão et al. nitric oxide synthase-2 modulates chemokine production by Trypanosoma Cruzi-infected cardiac myocytes. Microbes and Infection, v. 10, n. 14-15, p. 1558-1566, 2008Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2008.09.009. Acesso em: 12 abr. 2026. -
APA
Machado, F. S., Souto, J. T., Rossi, M. A., Esper, L., Tanowitz, H. B., Aliberti, J., & Silva, J. S. da. (2008). nitric oxide synthase-2 modulates chemokine production by Trypanosoma Cruzi-infected cardiac myocytes. Microbes and Infection, 10( 14-15), 1558-1566. doi:10.1016/j.micinf.2008.09.009 -
NLM
Machado FS, Souto JT, Rossi MA, Esper L, Tanowitz HB, Aliberti J, Silva JS da. nitric oxide synthase-2 modulates chemokine production by Trypanosoma Cruzi-infected cardiac myocytes [Internet]. Microbes and Infection. 2008 ; 10( 14-15): 1558-1566.[citado 2026 abr. 12 ] Available from: https://doi.org/10.1016/j.micinf.2008.09.009 -
Vancouver
Machado FS, Souto JT, Rossi MA, Esper L, Tanowitz HB, Aliberti J, Silva JS da. nitric oxide synthase-2 modulates chemokine production by Trypanosoma Cruzi-infected cardiac myocytes [Internet]. Microbes and Infection. 2008 ; 10( 14-15): 1558-1566.[citado 2026 abr. 12 ] Available from: https://doi.org/10.1016/j.micinf.2008.09.009 - B cell-deficient mice display enhanced susceptibility to Paracoccidioides brasiliensis infection
- Early dystrophin disruption in the pathogenesis of experimental chronic chagas cardiomyopathy
- Intercellular adhesion molecule 1 deficieney leads to impaired recruitment of T lymphocytes and enhanced host susceptibility to infection with Trypanosoma cruzi
- Early dystrophin disruption in the pathogenesis of experimental chronic Chagas cardiomyopathy
- Role of interleukin (IL)-18 in experimental paracoccidioidomycosis
- Anti- 'CD IND.4' abrogates rejection and restabliches long-term tolerance to sybgenetic newborn hearts grafted in mice chronically infected with trypanosoma cruzi
- Intensification of acute t. Cruzi myocarditis in balb / c nice pretreated
- Permeability alteration of the sarcolemmal membrane particulary at the site of macrophage contact, in experimental chronic trypanosoma cruzi miocarditis in mice
- The left lung is preferentially targeted during experimental paracoccidioidomycosis in C57BL/6 mice
- Haeme oxygenase activity protects the host against excessive cardiac inflammation during experimental Trypanosoma cruzi infection
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