Early dystrophin disruption in the pathogenesis of experimental chronic Chagas cardiomyopathy (2012)
- Authors:
- USP affiliated authors: SILVA, JOÃO SANTANA DA - FMRP ; ROSSI, MARCOS ANTONIO - FMRP
- Unidade: FMRP
- DOI: 10.1016/j.micinf.2011.08.010
- Subjects: DOENÇA DE CHAGAS; IMAGEM POR RESSONÂNCIA MAGNÉTICA; MIOCARDITE (PARASITOLOGIA); MIOCÁRDIO (PATOLOGIA); TRYPANOSOMA CRUZI
- Language: Inglês
- Imprenta:
- Publisher place: Issy les Moulineaux
- Date published: 2012
- Source:
- Título: Microbes and Infection
- ISSN: 1286-4579
- Volume/Número/Paginação/Ano: v. 14, n. 1 p. 59-68, 2012
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
PRADO, Cibele Maria et al. Early dystrophin disruption in the pathogenesis of experimental chronic Chagas cardiomyopathy. Microbes and Infection, v. 14, n. 1 p. 59-68, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2011.08.010. Acesso em: 13 fev. 2026. -
APA
Prado, C. M., Celes, M. R. N., Malvestio, L. M., Campos, E. C., Silva, J. S. da, Jelicks, L., et al. (2012). Early dystrophin disruption in the pathogenesis of experimental chronic Chagas cardiomyopathy. Microbes and Infection, 14( 1 p. 59-68). doi:10.1016/j.micinf.2011.08.010 -
NLM
Prado CM, Celes MRN, Malvestio LM, Campos EC, Silva JS da, Jelicks L, Tanowitz HB, Rossi MA. Early dystrophin disruption in the pathogenesis of experimental chronic Chagas cardiomyopathy [Internet]. Microbes and Infection. 2012 ; 14( 1 p. 59-68):[citado 2026 fev. 13 ] Available from: https://doi.org/10.1016/j.micinf.2011.08.010 -
Vancouver
Prado CM, Celes MRN, Malvestio LM, Campos EC, Silva JS da, Jelicks L, Tanowitz HB, Rossi MA. Early dystrophin disruption in the pathogenesis of experimental chronic Chagas cardiomyopathy [Internet]. Microbes and Infection. 2012 ; 14( 1 p. 59-68):[citado 2026 fev. 13 ] Available from: https://doi.org/10.1016/j.micinf.2011.08.010 - nitric oxide synthase-2 modulates chemokine production by Trypanosoma Cruzi-infected cardiac myocytes
- B cell-deficient mice display enhanced susceptibility to Paracoccidioides brasiliensis infection
- Early dystrophin disruption in the pathogenesis of experimental chronic chagas cardiomyopathy
- Intercellular adhesion molecule 1 deficieney leads to impaired recruitment of T lymphocytes and enhanced host susceptibility to infection with Trypanosoma cruzi
- Role of interleukin (IL)-18 in experimental paracoccidioidomycosis
- Anti- 'CD IND.4' abrogates rejection and restabliches long-term tolerance to sybgenetic newborn hearts grafted in mice chronically infected with trypanosoma cruzi
- Intensification of acute t. Cruzi myocarditis in balb / c nice pretreated
- Permeability alteration of the sarcolemmal membrane particulary at the site of macrophage contact, in experimental chronic trypanosoma cruzi miocarditis in mice
- The left lung is preferentially targeted during experimental paracoccidioidomycosis in C57BL/6 mice
- Haeme oxygenase activity protects the host against excessive cardiac inflammation during experimental Trypanosoma cruzi infection
Informações sobre o DOI: 10.1016/j.micinf.2011.08.010 (Fonte: oaDOI API)
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