Virilizing Leydig-cell ovarian tumor responsive to depot GnRH analoque (2004)
- Authors:
- Autor USP: MENDONÇA, BERENICE BILHARINHO DE - FM
- Unidade: FM
- Subjects: GONADOTROFINAS; HORMÔNIOS SEXUAIS FEMININOS (ANORMALIDADES); ANDRÓGENOS (ANORMALIDADES); NEOPLASIAS OVARIANAS (GENÉTICA;CIRURGIA;FISIOPATOLOGIA)
- Language: Inglês
- Imprenta:
- Publisher: International Society of Endocrinology
- Publisher place: Lisboa
- Date published: 2004
- Source:
- Título do periódico: Program and Abstracts
- Conference titles: International Congress of Endocrinology
-
ABNT
FERRAZ DE SOUZA, B. e MARCONDES, J. A. M. e MENDONCA, B. B. Virilizing Leydig-cell ovarian tumor responsive to depot GnRH analoque. 2004, Anais.. Lisboa: International Society of Endocrinology, 2004. . Acesso em: 10 ago. 2024. -
APA
Ferraz de Souza, B., Marcondes, J. A. M., & Mendonca, B. B. (2004). Virilizing Leydig-cell ovarian tumor responsive to depot GnRH analoque. In Program and Abstracts. Lisboa: International Society of Endocrinology. -
NLM
Ferraz de Souza B, Marcondes JAM, Mendonca BB. Virilizing Leydig-cell ovarian tumor responsive to depot GnRH analoque. Program and Abstracts. 2004 ;[citado 2024 ago. 10 ] -
Vancouver
Ferraz de Souza B, Marcondes JAM, Mendonca BB. Virilizing Leydig-cell ovarian tumor responsive to depot GnRH analoque. Program and Abstracts. 2004 ;[citado 2024 ago. 10 ] - Molecular analysis of SRY gene in Brazilian 46,XX sex reversed patients: absense of SRY sequence in gonadal tissue
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- Maternal isodisomy causing homozygosity for a dominant activating mutation of the luteinizing hormone receptor gene in a boy with familial male-limited precocius puberty
- Missense mutation a300v in the DAX1 gene in a Brazilian male with X-linked adrenal hypoplasia congenita and hypogonadotropic hypogonadism
- An unusual phenotype of fraiser syndrome due to IVS9 +4C>T mutation in the WT1 gene: predominantly male ambiguous genitalia and absence of gonadal dysgenesis
- Mutations in the SRY, DAX1, SF1 and WNT4 genes in brazilian sex-reversed patients
- Microconversão no promotor do gene CYP21A2 envolvendo os sítios de ligação dos fatores de transcrição Sp1 e ASP: nova etiologia molecular da deficiência de 21-hidroxilase (21OHD)
- Variable ACTH-stimulated 17-Hydroxyprogesterone values in 21-hydroxylase deficiency carriers are not related to the different CYP21 gene mutations
- Absense of duplication on DAX1 gene in patients with 46,XY sex reversal
- Crescimento linear e estatura final em crianças com tumores adrenais predominantemente virilizantes
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