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Artigo de periodico
MDSCs use a complex molecular network to suppress T-cell immunity in a pulmonary model of fungal infection (2024)
Kaminski, Valéria de Lima ; Borges, Bruno Montanari ; Santos, Bianca Vieira dos; Preite, Nycolas Willian ; Loures, Flavio Vieira ; Calich, Vera Lúcia Garcia
Fonte: Frontiers in Cellular and Infection Microbiology. Unidade: ICB
Assuntos: IMUNOLOGIA, PARACOCCIDIOIDES, PARACOCCIDIOIDOMICOSE, DERMATOPATIA PARASITÁRIA ANIMAL, MODELOS ANIMAIS DE DOENÇAS, CAMUNDONGOS, ANTÍGENOS DE FUNGOS, FÁRMACOS IMUNOSSUPRESSORES, INTERLEUCINA 10, TRANSDUÇÃO DE SINAL CELULAR, LINFÓCITOS T, PULMÃO, LECTINAS, IMUNOLOGIA CELULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KAMINSKI, Valéria de Lima et al. MDSCs use a complex molecular network to suppress T-cell immunity in a pulmonary model of fungal infection. Frontiers in Cellular and Infection Microbiology, v. 14, p. 20 , 2024Tradução . . Disponível em: https://doi.org/10.3389/fcimb.2024.1392744. Acesso em: 05 out. 2024.
APA
Kaminski, V. de L., Borges, B. M., Santos, B. V. dos, Preite, N. W., Loures, F. V., & Calich, V. L. G. (2024). MDSCs use a complex molecular network to suppress T-cell immunity in a pulmonary model of fungal infection. Frontiers in Cellular and Infection Microbiology, 14, 20 . doi:10.3389/fcimb.2024.1392744
NLM
Kaminski V de L, Borges BM, Santos BV dos, Preite NW, Loures FV, Calich VLG. MDSCs use a complex molecular network to suppress T-cell immunity in a pulmonary model of fungal infection [Internet]. Frontiers in Cellular and Infection Microbiology. 2024 ; 14 20 .[citado 2024 out. 05 ] Available from: https://doi.org/10.3389/fcimb.2024.1392744
Vancouver
Kaminski V de L, Borges BM, Santos BV dos, Preite NW, Loures FV, Calich VLG. MDSCs use a complex molecular network to suppress T-cell immunity in a pulmonary model of fungal infection [Internet]. Frontiers in Cellular and Infection Microbiology. 2024 ; 14 20 .[citado 2024 out. 05 ] Available from: https://doi.org/10.3389/fcimb.2024.1392744
Artigo de periodico
Blocking the CTLA-4 and PD-1 pathways during pulmonary paracoccidioidomycosis improves immunity, reduces disease severity, and increases the survival of infected mice (2024)
Preite, Nycolas Willian ; Borges, Bruno Montanari ; Kaminski, Valéria de Lima ; Santos, Bianca Vieira dos; Loures, Flavio Vieira ; Ayupe, Marina Caçador ; Gonçalves, Leonardo Mandu ; Filgueiras, Igor Salerno ; Salgado, Caio Loureiro ; Muxel, Sandra Marcia ; Marques, Otavio Cabral ; Fonseca, Denise Morais da ; Calich, Vera Lúcia Garcia
Fonte: Frontiers in Immunology. Unidades: FM, ICB
Assuntos: IMUNOLOGIA, PARACOCCIDIOIDOMICOSE, PARACOCCIDIOIDES, FUNGOS MITOSPORICOS, IMUNIDADE, ANTÍGENOS, IMUNOGLOBULINAS, LINFÓCITOS T, CITOTOXICIDADE IMUNOLÓGICA, CITOCINAS, CAMUNDONGOS
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ABNT
PREITE, Nycolas Willian et al. Blocking the CTLA-4 and PD-1 pathways during pulmonary paracoccidioidomycosis improves immunity, reduces disease severity, and increases the survival of infected mice. Frontiers in Immunology, v. 15, p. 17 , 2024Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2024.1347318. Acesso em: 05 out. 2024.
APA
Preite, N. W., Borges, B. M., Kaminski, V. de L., Santos, B. V. dos, Loures, F. V., Ayupe, M. C., et al. (2024). Blocking the CTLA-4 and PD-1 pathways during pulmonary paracoccidioidomycosis improves immunity, reduces disease severity, and increases the survival of infected mice. Frontiers in Immunology, 15, 17 . doi:10.3389/fimmu.2024.1347318
NLM
Preite NW, Borges BM, Kaminski V de L, Santos BV dos, Loures FV, Ayupe MC, Gonçalves LM, Filgueiras IS, Salgado CL, Muxel SM, Marques OC, Fonseca DM da, Calich VLG. Blocking the CTLA-4 and PD-1 pathways during pulmonary paracoccidioidomycosis improves immunity, reduces disease severity, and increases the survival of infected mice [Internet]. Frontiers in Immunology. 2024 ; 15 17 .[citado 2024 out. 05 ] Available from: https://doi.org/10.3389/fimmu.2024.1347318
Vancouver
Preite NW, Borges BM, Kaminski V de L, Santos BV dos, Loures FV, Ayupe MC, Gonçalves LM, Filgueiras IS, Salgado CL, Muxel SM, Marques OC, Fonseca DM da, Calich VLG. Blocking the CTLA-4 and PD-1 pathways during pulmonary paracoccidioidomycosis improves immunity, reduces disease severity, and increases the survival of infected mice [Internet]. Frontiers in Immunology. 2024 ; 15 17 .[citado 2024 out. 05 ] Available from: https://doi.org/10.3389/fimmu.2024.1347318
Artigo de periodico
The immunosuppressive activity of myeloid-derived suppressor cells in murine Paracoccidioidomycosis relies on Indoleamine 2,3-dioxygenase activity and Dectin-1 and TLRs signaling (2023)
Kaminsk, Valéria de Lima ; Preite, Nycolas Willian ; Borges, Bruno Montanari ; Santos, Bianca Vieira dos; Loures, Flavio Vieira ; Calich, Vera Lúcia Garcia
Fonte: Scientific Reports. Unidade: ICB
Assuntos: CAMUNDONGOS, FÁRMACOS IMUNOSSUPRESSORES, PARACOCCIDIOIDOMICOSE, SEROTONINA, RECEPTORES DE SEROTONINA, CÉLULAS SANGUÍNEAS, PROLIFERAÇÃO CELULAR, DOENÇAS AUTOIMUNES, ATIVAÇÃO ENZIMÁTICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KAMINSK, Valéria de Lima et al. The immunosuppressive activity of myeloid-derived suppressor cells in murine Paracoccidioidomycosis relies on Indoleamine 2,3-dioxygenase activity and Dectin-1 and TLRs signaling. Scientific Reports, v. 13, n. 1, p. 1-15, 2023Tradução . . Disponível em: https://doi.org/10.1038/s41598-023-39262-8. Acesso em: 05 out. 2024.
APA
Kaminsk, V. de L., Preite, N. W., Borges, B. M., Santos, B. V. dos, Loures, F. V., & Calich, V. L. G. (2023). The immunosuppressive activity of myeloid-derived suppressor cells in murine Paracoccidioidomycosis relies on Indoleamine 2,3-dioxygenase activity and Dectin-1 and TLRs signaling. Scientific Reports, 13( 1), 1-15. doi:10.1038/s41598-023-39262-8
NLM
Kaminsk V de L, Preite NW, Borges BM, Santos BV dos, Loures FV, Calich VLG. The immunosuppressive activity of myeloid-derived suppressor cells in murine Paracoccidioidomycosis relies on Indoleamine 2,3-dioxygenase activity and Dectin-1 and TLRs signaling [Internet]. Scientific Reports. 2023 ; 13( 1): 1-15.[citado 2024 out. 05 ] Available from: https://doi.org/10.1038/s41598-023-39262-8
Vancouver
Kaminsk V de L, Preite NW, Borges BM, Santos BV dos, Loures FV, Calich VLG. The immunosuppressive activity of myeloid-derived suppressor cells in murine Paracoccidioidomycosis relies on Indoleamine 2,3-dioxygenase activity and Dectin-1 and TLRs signaling [Internet]. Scientific Reports. 2023 ; 13( 1): 1-15.[citado 2024 out. 05 ] Available from: https://doi.org/10.1038/s41598-023-39262-8
Artigo de periodico
Transcriptional profiling of a fungal granuloma reveals a low metabolic activity of Paracoccidioides brasiliensis yeasts and an actively regulated host immune response (2023)
Borges, Bruno Montanari ; Ramos, Rafael Berton Correia ; Preite, Nycolas Willian ; Kaminski, Valéria de Lima ; Camacho, Maurício ; Campos, Claudia Barbosa Ladeira de; Zelanis, André ; Loures, Flavio Vieira ; Castro, Patrícia Alves de; Maximo, Marina Ferreira ; Fill, Taicia Pacheco ; Traynor, Aimee M. ; Özlem Sarikaya-Bayram ; Sean Doyle ; Bayram, Özgür ; Goldman, Gustavo Henrique ; Calich, Vera Lúcia Garcia
Fonte: Frontiers in Cellular and Infection Microbiology. Unidades: FCFRP, ICB
Assuntos: IMUNOLOGIA, GRANULOMA, PARACOCCIDIOIDOMICOSE, PARACOCCIDIOIDES, PROTEÔMICA, FERRO, CAMUNDONGOS ENDOGÂMICOS C57BL, TRANSCRIÇÃO GÊNICA
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ABNT
BORGES, Bruno Montanari et al. Transcriptional profiling of a fungal granuloma reveals a low metabolic activity of Paracoccidioides brasiliensis yeasts and an actively regulated host immune response. Frontiers in Cellular and Infection Microbiology, v. 13, p. 27 , 2023Tradução . . Disponível em: https://doi/org/10.3389/fcimb.2023.1268959. Acesso em: 05 out. 2024.
APA
Borges, B. M., Ramos, R. B. C., Preite, N. W., Kaminski, V. de L., Camacho, M., Campos, C. B. L. de, et al. (2023). Transcriptional profiling of a fungal granuloma reveals a low metabolic activity of Paracoccidioides brasiliensis yeasts and an actively regulated host immune response. Frontiers in Cellular and Infection Microbiology, 13, 27 . doi:10.3389/fcimb.2023.1268959
NLM
Borges BM, Ramos RBC, Preite NW, Kaminski V de L, Camacho M, Campos CBL de, Zelanis A, Loures FV, Castro PA de, Maximo MF, Fill TP, Traynor AM, Özlem Sarikaya-Bayram, Sean Doyle, Bayram Ö, Goldman GH, Calich VLG. Transcriptional profiling of a fungal granuloma reveals a low metabolic activity of Paracoccidioides brasiliensis yeasts and an actively regulated host immune response [Internet]. Frontiers in Cellular and Infection Microbiology. 2023 ; 13 27 .[citado 2024 out. 05 ] Available from: https://doi/org/10.3389/fcimb.2023.1268959
Vancouver
Borges BM, Ramos RBC, Preite NW, Kaminski V de L, Camacho M, Campos CBL de, Zelanis A, Loures FV, Castro PA de, Maximo MF, Fill TP, Traynor AM, Özlem Sarikaya-Bayram, Sean Doyle, Bayram Ö, Goldman GH, Calich VLG. Transcriptional profiling of a fungal granuloma reveals a low metabolic activity of Paracoccidioides brasiliensis yeasts and an actively regulated host immune response [Internet]. Frontiers in Cellular and Infection Microbiology. 2023 ; 13 27 .[citado 2024 out. 05 ] Available from: https://doi/org/10.3389/fcimb.2023.1268959
Artigo de periodico
Myeloid-derived suppressor cells are associated with impaired Th1 and Th17 responses and severe pulmonary paracoccidioidomycosis which is reversed by anti-Gr1 therapy (2023)
Preite, Nycolas Willian ; Kaminski, Valéria de Lima ; Borges, Bruno Montanari ; Calich, Vera Lúcia Garcia ; Loures, Flávio Vieira
Fonte: Frontiers in Immunology. Unidade: ICB
Assuntos: IMUNOLOGIA, CÉLULAS SANGUÍNEAS, MEDULA ÓSSEA DE ANIMAL, CÉLULAS MIELOIDES, PARACOCCIDIOIDOMICOSE, DERMATOPATIAS INFECCIOSAS, FÁRMACOS IMUNOSSUPRESSORES, PULMÃO, INFECÇÕES RESPIRATÓRIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PREITE, Nycolas Willian et al. Myeloid-derived suppressor cells are associated with impaired Th1 and Th17 responses and severe pulmonary paracoccidioidomycosis which is reversed by anti-Gr1 therapy. Frontiers in Immunology, v. 14, p. 1-19, 2023Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2023.1039244. Acesso em: 05 out. 2024.
APA
Preite, N. W., Kaminski, V. de L., Borges, B. M., Calich, V. L. G., & Loures, F. V. (2023). Myeloid-derived suppressor cells are associated with impaired Th1 and Th17 responses and severe pulmonary paracoccidioidomycosis which is reversed by anti-Gr1 therapy. Frontiers in Immunology, 14, 1-19. doi:10.3389/fimmu.2023.1039244
NLM
Preite NW, Kaminski V de L, Borges BM, Calich VLG, Loures FV. Myeloid-derived suppressor cells are associated with impaired Th1 and Th17 responses and severe pulmonary paracoccidioidomycosis which is reversed by anti-Gr1 therapy [Internet]. Frontiers in Immunology. 2023 ; 14 1-19.[citado 2024 out. 05 ] Available from: https://doi.org/10.3389/fimmu.2023.1039244
Vancouver
Preite NW, Kaminski V de L, Borges BM, Calich VLG, Loures FV. Myeloid-derived suppressor cells are associated with impaired Th1 and Th17 responses and severe pulmonary paracoccidioidomycosis which is reversed by anti-Gr1 therapy [Internet]. Frontiers in Immunology. 2023 ; 14 1-19.[citado 2024 out. 05 ] Available from: https://doi.org/10.3389/fimmu.2023.1039244
Artigo de periodico
AhR ligands modulate the differentiation of innate lymphoid cells and T helper cell subsets that control the severity of a pulmonary fungal infection (2021)
Araújo, Eliseu Frank de; Loures, Flávio Vieira ; Preite, Nycolas Willian ; Feriotti, Cláudia ; Galdino, Nayane Alves de Lima ; Costa, Tania Alves da; Calich, Vera Lúcia Garcia
Fonte: Frontiers in Immunology. Unidade: ICB
Assuntos: IMUNOLOGIA, LINFÓCITOS T, PARACOCCIDIOIDES, PARACOCCIDIOIDOMICOSE, INFECÇÕES RESPIRATÓRIAS, DOENÇAS RESPIRATÓRIAS, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS, LEUCÓCITOS, CÉLULAS DENDRÍTICAS
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ABNT
ARAÚJO, Eliseu Frank de et al. AhR ligands modulate the differentiation of innate lymphoid cells and T helper cell subsets that control the severity of a pulmonary fungal infection. Frontiers in Immunology, v. 12, p. 1-17, 2021Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2021.630938. Acesso em: 05 out. 2024.
APA
Araújo, E. F. de, Loures, F. V., Preite, N. W., Feriotti, C., Galdino, N. A. de L., Costa, T. A. da, & Calich, V. L. G. (2021). AhR ligands modulate the differentiation of innate lymphoid cells and T helper cell subsets that control the severity of a pulmonary fungal infection. Frontiers in Immunology, 12, 1-17. doi:10.3389/fimmu.2021.630938
NLM
Araújo EF de, Loures FV, Preite NW, Feriotti C, Galdino NA de L, Costa TA da, Calich VLG. AhR ligands modulate the differentiation of innate lymphoid cells and T helper cell subsets that control the severity of a pulmonary fungal infection [Internet]. Frontiers in Immunology. 2021 ; 12 1-17.[citado 2024 out. 05 ] Available from: https://doi.org/10.3389/fimmu.2021.630938
Vancouver
Araújo EF de, Loures FV, Preite NW, Feriotti C, Galdino NA de L, Costa TA da, Calich VLG. AhR ligands modulate the differentiation of innate lymphoid cells and T helper cell subsets that control the severity of a pulmonary fungal infection [Internet]. Frontiers in Immunology. 2021 ; 12 1-17.[citado 2024 out. 05 ] Available from: https://doi.org/10.3389/fimmu.2021.630938
Artigo de periodico
Pulmonary paracoccidioidomycosis in AhR deficient hosts is severe and associated with defective Treg and Th22 responses (2020)
Araújo, Eliseu Frank de; Preite, Nycolas Willian; Veldhoen, Marc ; Loures, Flávio Vieira ; Calich, Vera Lúcia Garcia
Fonte: Scientific Reports. Unidade: ICB
Assuntos: IMUNOLOGIA, CAMUNDONGOS, RATOS, PARACOCCIDIOIDES, CITOCINAS, LIGANTES, PARACOCCIDIOIDOMICOSE, ANTIFÚNGICOS
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ABNT
ARAÚJO, Eliseu Frank de et al. Pulmonary paracoccidioidomycosis in AhR deficient hosts is severe and associated with defective Treg and Th22 responses. Scientific Reports, v. 10, p. 16 , 2020Tradução . . Disponível em: https://doi.org/10.1038/s41598-020-68322-6. Acesso em: 05 out. 2024.
APA
Araújo, E. F. de, Preite, N. W., Veldhoen, M., Loures, F. V., & Calich, V. L. G. (2020). Pulmonary paracoccidioidomycosis in AhR deficient hosts is severe and associated with defective Treg and Th22 responses. Scientific Reports, 10, 16 . doi:10.1038/s41598-020-68322-6
NLM
Araújo EF de, Preite NW, Veldhoen M, Loures FV, Calich VLG. Pulmonary paracoccidioidomycosis in AhR deficient hosts is severe and associated with defective Treg and Th22 responses [Internet]. Scientific Reports. 2020 ; 10 16 .[citado 2024 out. 05 ] Available from: https://doi.org/10.1038/s41598-020-68322-6
Vancouver
Araújo EF de, Preite NW, Veldhoen M, Loures FV, Calich VLG. Pulmonary paracoccidioidomycosis in AhR deficient hosts is severe and associated with defective Treg and Th22 responses [Internet]. Scientific Reports. 2020 ; 10 16 .[citado 2024 out. 05 ] Available from: https://doi.org/10.1038/s41598-020-68322-6
Artigo de periodico
The IDO-AhR axis controls Th17/Treg immunity in a pulmonary model of fungal infection (2017)
Araújo, Eliseu Frank de; Feriotti, Claudia; Galdino, Nayane Alves de Lima; Preite, Nycolas Willian; Calich, Vera Lúcia Garcia; Loures, Flávio Vieira
Fonte: Frontiers in Immunology. Unidade: ICB
Assuntos: IMUNOLOGIA, PARACOCCIDIOIDOMICOSE, LINFÓCITOS T, PARACOCCIDIOIDES BRASILIENSIS, ENZIMAS OXIRREDUTORAS, CÉLULAS DENDRÍTICAS
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ABNT
ARAÚJO, Eliseu Frank de et al. The IDO-AhR axis controls Th17/Treg immunity in a pulmonary model of fungal infection. Frontiers in Immunology, v. 8, n. 880, p. 1-15, 2017Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2017.00880. Acesso em: 05 out. 2024.
APA
Araújo, E. F. de, Feriotti, C., Galdino, N. A. de L., Preite, N. W., Calich, V. L. G., & Loures, F. V. (2017). The IDO-AhR axis controls Th17/Treg immunity in a pulmonary model of fungal infection. Frontiers in Immunology, 8( 880), 1-15. doi:10.3389/fimmu.2017.00880
NLM
Araújo EF de, Feriotti C, Galdino NA de L, Preite NW, Calich VLG, Loures FV. The IDO-AhR axis controls Th17/Treg immunity in a pulmonary model of fungal infection [Internet]. Frontiers in Immunology. 2017 ; 8( 880): 1-15.[citado 2024 out. 05 ] Available from: https://doi.org/10.3389/fimmu.2017.00880
Vancouver
Araújo EF de, Feriotti C, Galdino NA de L, Preite NW, Calich VLG, Loures FV. The IDO-AhR axis controls Th17/Treg immunity in a pulmonary model of fungal infection [Internet]. Frontiers in Immunology. 2017 ; 8( 880): 1-15.[citado 2024 out. 05 ] Available from: https://doi.org/10.3389/fimmu.2017.00880

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