AhR ligands modulate the differentiation of innate lymphoid cells and T helper cell subsets that control the severity of a pulmonary fungal infection (2021)
- Authors:
- USP affiliated authors: LOURES, FLÁVIO VIEIRA - ICB ; COSTA, TÂNIA ALVES DA - ICB ; CALICH, VERA LUCIA GARCIA - ICB
- Unidade: ICB
- DOI: 10.3389/fimmu.2021.630938
- Subjects: IMUNOLOGIA; LINFÓCITOS T; PARACOCCIDIOIDES; PARACOCCIDIOIDOMICOSE; INFECÇÕES RESPIRATÓRIAS; DOENÇAS RESPIRATÓRIAS; CAMUNDONGOS; MODELOS ANIMAIS DE DOENÇAS; LEUCÓCITOS; CÉLULAS DENDRÍTICAS
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Frontiers in Immunology
- ISSN: 1664-3224
- Volume/Número/Paginação/Ano: v. 12, art. 630938, p. 1-17, 2021
- Status:
- Artigo publicado em periódico de acesso aberto (Gold Open Access)
- Versão do Documento:
- Versão publicada (Published version)
- Acessar versão aberta:
-
ABNT
ARAÚJO, Eliseu Frank de et al. AhR ligands modulate the differentiation of innate lymphoid cells and T helper cell subsets that control the severity of a pulmonary fungal infection. Frontiers in Immunology, v. 12, p. 1-17, 2021Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2021.630938. Acesso em: 01 abr. 2026. -
APA
Araújo, E. F. de, Loures, F. V., Preite, N. W., Feriotti, C., Galdino, N. A. de L., Costa, T. A. da, & Calich, V. L. G. (2021). AhR ligands modulate the differentiation of innate lymphoid cells and T helper cell subsets that control the severity of a pulmonary fungal infection. Frontiers in Immunology, 12, 1-17. doi:10.3389/fimmu.2021.630938 -
NLM
Araújo EF de, Loures FV, Preite NW, Feriotti C, Galdino NA de L, Costa TA da, Calich VLG. AhR ligands modulate the differentiation of innate lymphoid cells and T helper cell subsets that control the severity of a pulmonary fungal infection [Internet]. Frontiers in Immunology. 2021 ; 12 1-17.[citado 2026 abr. 01 ] Available from: https://doi.org/10.3389/fimmu.2021.630938 -
Vancouver
Araújo EF de, Loures FV, Preite NW, Feriotti C, Galdino NA de L, Costa TA da, Calich VLG. AhR ligands modulate the differentiation of innate lymphoid cells and T helper cell subsets that control the severity of a pulmonary fungal infection [Internet]. Frontiers in Immunology. 2021 ; 12 1-17.[citado 2026 abr. 01 ] Available from: https://doi.org/10.3389/fimmu.2021.630938 - Invariant natural killer T cells as key players in host resistance against Paracoccidioides brasiliensis
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- Paracoccidoides brasiliensis 30 kDa adhesin: identification as a 14-3-3 protein, cloning and subcellular localization in infection models
- Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis
- Dectin-1 induces M1 macrophages and prominent expansion of CD8+IL-17+ cells in pulmonary Paracoccidioidomycosis
- In pulmonary Paracoccidioidomycosis IL-10 deficiency leads to increased immunity and regressive infection without enhancing tissue pathology
- Disease tolerance mediated by phosphorylated indoleamine-2,3 dioxygenase confers resistance to a primary fungal pathogen
- Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis
- Loss- and gain-of-function approaches indicate a dual role exerted by regulatory T cells in pulmonary paracoccidioidomycosis
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