Paracoccidoides brasiliensis 30 kDa adhesin: identification as a 14-3-3 protein, cloning and subcellular localization in infection models (2013)
- Authors:
- USP affiliated authors: COSTA, TÂNIA ALVES DA - ICB ; CALICH, VERA LUCIA GARCIA - ICB
- Unidade: ICB
- DOI: 10.1371/journal.pone.0062533
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Publisher place: San Francisco
- Date published: 2013
- Source:
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
SILVA, Julhiany de Fatima da et al. Paracoccidoides brasiliensis 30 kDa adhesin: identification as a 14-3-3 protein, cloning and subcellular localization in infection models. PLOS ONE, v. 8, n. 4, p. 1-10, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0062533. Acesso em: 24 abr. 2024. -
APA
Silva, J. de F. da, Oliveira, H. C. de, Marcos, C. M., Silva, R. A. M. da, Costa, T. A. da, Calich, V. L. G., et al. (2013). Paracoccidoides brasiliensis 30 kDa adhesin: identification as a 14-3-3 protein, cloning and subcellular localization in infection models. PLOS ONE, 8( 4), 1-10. doi:10.1371/journal.pone.0062533 -
NLM
Silva J de F da, Oliveira HC de, Marcos CM, Silva RAM da, Costa TA da, Calich VLG, Almeida AMF, Mendes-Giannini MJS. Paracoccidoides brasiliensis 30 kDa adhesin: identification as a 14-3-3 protein, cloning and subcellular localization in infection models [Internet]. PLOS ONE. 2013 ;8( 4): 1-10.[citado 2024 abr. 24 ] Available from: https://doi.org/10.1371/journal.pone.0062533 -
Vancouver
Silva J de F da, Oliveira HC de, Marcos CM, Silva RAM da, Costa TA da, Calich VLG, Almeida AMF, Mendes-Giannini MJS. Paracoccidoides brasiliensis 30 kDa adhesin: identification as a 14-3-3 protein, cloning and subcellular localization in infection models [Internet]. PLOS ONE. 2013 ;8( 4): 1-10.[citado 2024 abr. 24 ] Available from: https://doi.org/10.1371/journal.pone.0062533 - In pulmonary Paracoccidioidomycosis IL-10 deficiency leads to increased immunity and regressive infection without enhancing tissue pathology
- Dectin-1 induces M1 macrophages and prominent expansion of CD8+IL-17+ cells in pulmonary Paracoccidioidomycosis
- Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis
- Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection
- A função da IL-10 na paracoccidioidomicose pulmonar murina
- Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis
- Loss- and gain-of-function approaches indicate a dual role exerted by regulatory T cells in pulmonary paracoccidioidomycosis
- AhR ligands modulate the differentiation of innate lymphoid cells and T helper cell subsets that control the severity of a pulmonary fungal infection
- In vivo treatment with ril-12 protects susceptible mice from Paracoccidioides brasiliensis infection whereas in vivo neutralization of il-4 does not alter disease outcome
- Invariant natural killer T cells as key players in host resistance against Paracoccidioides brasiliensis
Informações sobre o DOI: 10.1371/journal.pone.0062533 (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
