Dectin-1 induces M1 macrophages and prominent expansion of CD8+IL-17+ cells in pulmonary Paracoccidioidomycosis (2014)
- Authors:
- USP affiliated authors: COSTA, TÂNIA ALVES DA - ICB ; CALICH, VERA LUCIA GARCIA - ICB
- Unidade: ICB
- DOI: 10.1093/infdis/jiu136
- Subjects: IMUNOLOGIA; MACRÓFAGOS; PARACOCCIDIOIDOMICOSE
- Language: Inglês
- Imprenta:
- Source:
- Título: Journal of Infectious Diseases
- ISSN: 1537-6613
- Volume/Número/Paginação/Ano: v. 210, n. 5, p. 762-773, 2014
- Status:
- Artigo possui acesso gratuito no site do editor (Bronze Open Access)
- Versão do Documento:
- Versão publicada (Published version)
- Acessar versão aberta:
-
ABNT
LOURES, Flávio V. et al. Dectin-1 induces M1 macrophages and prominent expansion of CD8+IL-17+ cells in pulmonary Paracoccidioidomycosis. Journal of Infectious Diseases, v. 210, n. 5, p. 762-773, 2014Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiu136. Acesso em: 01 abr. 2026. -
APA
Loures, F. V., Araújo, E. F. de, Feriotti, C., Bazan, S. B., Costa, T. A. da, Brown, G. D., & Calich, V. L. G. (2014). Dectin-1 induces M1 macrophages and prominent expansion of CD8+IL-17+ cells in pulmonary Paracoccidioidomycosis. Journal of Infectious Diseases, 210( 5), 762-773. doi:10.1093/infdis/jiu136 -
NLM
Loures FV, Araújo EF de, Feriotti C, Bazan SB, Costa TA da, Brown GD, Calich VLG. Dectin-1 induces M1 macrophages and prominent expansion of CD8+IL-17+ cells in pulmonary Paracoccidioidomycosis [Internet]. Journal of Infectious Diseases. 2014 ; 210( 5): 762-773.[citado 2026 abr. 01 ] Available from: https://doi.org/10.1093/infdis/jiu136 -
Vancouver
Loures FV, Araújo EF de, Feriotti C, Bazan SB, Costa TA da, Brown GD, Calich VLG. Dectin-1 induces M1 macrophages and prominent expansion of CD8+IL-17+ cells in pulmonary Paracoccidioidomycosis [Internet]. Journal of Infectious Diseases. 2014 ; 210( 5): 762-773.[citado 2026 abr. 01 ] Available from: https://doi.org/10.1093/infdis/jiu136 - Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection
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