Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis (2015)
- Authors:
- USP affiliated authors: CALICH, VERA LUCIA GARCIA - ICB ; COSTA, TÂNIA ALVES DA - ICB
- Unidade: ICB
- DOI: 10.3389/fmicb.2015.00913
- Subjects: IMUNOLOGIA; PARACOCCIDIOIDOMICOSE (RESISTÊNCIA); INFLAMAÇÃO
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Frontiers in Microbiology
- ISSN: 1664-302X
- Volume/Número/Paginação/Ano: v. 6, n. 913, p. 1-13, 2015
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
FERIOTTI, Claudia et al. Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis. Frontiers in Microbiology, v. 6, n. 913, p. 1-13, 2015Tradução . . Disponível em: https://doi.org/10.3389/fmicb.2015.00913. Acesso em: 27 set. 2024. -
APA
Feriotti, C., Bazan, S. B., Loures, F. V., Araújo, E. F. de, Costa, T. A. da, & Calich, V. L. G. (2015). Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis. Frontiers in Microbiology, 6( 913), 1-13. doi:10.3389/fmicb.2015.00913 -
NLM
Feriotti C, Bazan SB, Loures FV, Araújo EF de, Costa TA da, Calich VLG. Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis [Internet]. Frontiers in Microbiology. 2015 ; 6( 913): 1-13.[citado 2024 set. 27 ] Available from: https://doi.org/10.3389/fmicb.2015.00913 -
Vancouver
Feriotti C, Bazan SB, Loures FV, Araújo EF de, Costa TA da, Calich VLG. Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis [Internet]. Frontiers in Microbiology. 2015 ; 6( 913): 1-13.[citado 2024 set. 27 ] Available from: https://doi.org/10.3389/fmicb.2015.00913 - Paracoccidoides brasiliensis 30 kDa adhesin: identification as a 14-3-3 protein, cloning and subcellular localization in infection models
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- In pulmonary Paracoccidioidomycosis IL-10 deficiency leads to increased immunity and regressive infection without enhancing tissue pathology
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- A função da IL-10 na paracoccidioidomicose pulmonar murina
- Loss- and gain-of-function approaches indicate a dual role exerted by regulatory T cells in pulmonary paracoccidioidomycosis
- Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis
- AhR ligands modulate the differentiation of innate lymphoid cells and T helper cell subsets that control the severity of a pulmonary fungal infection
- In vivo treatment with ril-12 protects susceptible mice from Paracoccidioides brasiliensis infection whereas in vivo neutralization of il-4 does not alter disease outcome
- Invariant natural killer T cells as key players in host resistance against Paracoccidioides brasiliensis
Informações sobre o DOI: 10.3389/fmicb.2015.00913 (Fonte: oaDOI API)
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