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Artigo de periodico
Myeloid-derived suppressor cells are associated with impaired Th1 and Th17 responses and severe pulmonary paracoccidioidomycosis which is reversed by anti-Gr1 therapy (2023)
Preite, Nycolas Willian ; Kaminski, Valéria de Lima ; Borges, Bruno Montanari ; Calich, Vera Lúcia Garcia ; Loures, Flávio Vieira
Source: Frontiers in Immunology. Unidade: ICB
Subjects: IMUNOLOGIA, CÉLULAS SANGUÍNEAS, MEDULA ÓSSEA DE ANIMAL, CÉLULAS MIELOIDES, PARACOCCIDIOIDOMICOSE, DERMATOPATIAS INFECCIOSAS, FÁRMACOS IMUNOSSUPRESSORES, PULMÃO, INFECÇÕES RESPIRATÓRIAS
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PREITE, Nycolas Willian et al. Myeloid-derived suppressor cells are associated with impaired Th1 and Th17 responses and severe pulmonary paracoccidioidomycosis which is reversed by anti-Gr1 therapy. Frontiers in Immunology, v. 14, p. 1-19, 2023Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2023.1039244. Acesso em: 17 nov. 2024.
APA
Preite, N. W., Kaminski, V. de L., Borges, B. M., Calich, V. L. G., & Loures, F. V. (2023). Myeloid-derived suppressor cells are associated with impaired Th1 and Th17 responses and severe pulmonary paracoccidioidomycosis which is reversed by anti-Gr1 therapy. Frontiers in Immunology, 14, 1-19. doi:10.3389/fimmu.2023.1039244
NLM
Preite NW, Kaminski V de L, Borges BM, Calich VLG, Loures FV. Myeloid-derived suppressor cells are associated with impaired Th1 and Th17 responses and severe pulmonary paracoccidioidomycosis which is reversed by anti-Gr1 therapy [Internet]. Frontiers in Immunology. 2023 ; 14 1-19.[citado 2024 nov. 17 ] Available from: https://doi.org/10.3389/fimmu.2023.1039244
Vancouver
Preite NW, Kaminski V de L, Borges BM, Calich VLG, Loures FV. Myeloid-derived suppressor cells are associated with impaired Th1 and Th17 responses and severe pulmonary paracoccidioidomycosis which is reversed by anti-Gr1 therapy [Internet]. Frontiers in Immunology. 2023 ; 14 1-19.[citado 2024 nov. 17 ] Available from: https://doi.org/10.3389/fimmu.2023.1039244
Artigo de periodico
AhR ligands modulate the differentiation of innate lymphoid cells and T helper cell subsets that control the severity of a pulmonary fungal infection (2021)
Araújo, Eliseu Frank de; Loures, Flávio Vieira ; Preite, Nycolas Willian ; Feriotti, Cláudia ; Galdino, Nayane Alves de Lima ; Costa, Tania Alves da; Calich, Vera Lúcia Garcia
Source: Frontiers in Immunology. Unidade: ICB
Subjects: IMUNOLOGIA, LINFÓCITOS T, PARACOCCIDIOIDES, PARACOCCIDIOIDOMICOSE, INFECÇÕES RESPIRATÓRIAS, DOENÇAS RESPIRATÓRIAS, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS, LEUCÓCITOS, CÉLULAS DENDRÍTICAS
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ARAÚJO, Eliseu Frank de et al. AhR ligands modulate the differentiation of innate lymphoid cells and T helper cell subsets that control the severity of a pulmonary fungal infection. Frontiers in Immunology, v. 12, p. 1-17, 2021Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2021.630938. Acesso em: 17 nov. 2024.
APA
Araújo, E. F. de, Loures, F. V., Preite, N. W., Feriotti, C., Galdino, N. A. de L., Costa, T. A. da, & Calich, V. L. G. (2021). AhR ligands modulate the differentiation of innate lymphoid cells and T helper cell subsets that control the severity of a pulmonary fungal infection. Frontiers in Immunology, 12, 1-17. doi:10.3389/fimmu.2021.630938
NLM
Araújo EF de, Loures FV, Preite NW, Feriotti C, Galdino NA de L, Costa TA da, Calich VLG. AhR ligands modulate the differentiation of innate lymphoid cells and T helper cell subsets that control the severity of a pulmonary fungal infection [Internet]. Frontiers in Immunology. 2021 ; 12 1-17.[citado 2024 nov. 17 ] Available from: https://doi.org/10.3389/fimmu.2021.630938
Vancouver
Araújo EF de, Loures FV, Preite NW, Feriotti C, Galdino NA de L, Costa TA da, Calich VLG. AhR ligands modulate the differentiation of innate lymphoid cells and T helper cell subsets that control the severity of a pulmonary fungal infection [Internet]. Frontiers in Immunology. 2021 ; 12 1-17.[citado 2024 nov. 17 ] Available from: https://doi.org/10.3389/fimmu.2021.630938
Artigo de periodico
Pulmonary paracoccidioidomycosis in AhR deficient hosts is severe and associated with defective Treg and Th22 responses (2020)
Araújo, Eliseu Frank de; Preite, Nycolas Willian; Veldhoen, Marc ; Loures, Flávio Vieira ; Calich, Vera Lúcia Garcia
Source: Scientific Reports. Unidade: ICB
Subjects: IMUNOLOGIA, CAMUNDONGOS, RATOS, PARACOCCIDIOIDES, CITOCINAS, LIGANTES, PARACOCCIDIOIDOMICOSE, ANTIFÚNGICOS
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ARAÚJO, Eliseu Frank de et al. Pulmonary paracoccidioidomycosis in AhR deficient hosts is severe and associated with defective Treg and Th22 responses. Scientific Reports, v. 10, p. 16 , 2020Tradução . . Disponível em: https://doi.org/10.1038/s41598-020-68322-6. Acesso em: 17 nov. 2024.
APA
Araújo, E. F. de, Preite, N. W., Veldhoen, M., Loures, F. V., & Calich, V. L. G. (2020). Pulmonary paracoccidioidomycosis in AhR deficient hosts is severe and associated with defective Treg and Th22 responses. Scientific Reports, 10, 16 . doi:10.1038/s41598-020-68322-6
NLM
Araújo EF de, Preite NW, Veldhoen M, Loures FV, Calich VLG. Pulmonary paracoccidioidomycosis in AhR deficient hosts is severe and associated with defective Treg and Th22 responses [Internet]. Scientific Reports. 2020 ; 10 16 .[citado 2024 nov. 17 ] Available from: https://doi.org/10.1038/s41598-020-68322-6
Vancouver
Araújo EF de, Preite NW, Veldhoen M, Loures FV, Calich VLG. Pulmonary paracoccidioidomycosis in AhR deficient hosts is severe and associated with defective Treg and Th22 responses [Internet]. Scientific Reports. 2020 ; 10 16 .[citado 2024 nov. 17 ] Available from: https://doi.org/10.1038/s41598-020-68322-6
Artigo de periodico
The IDO-AhR axis controls Th17/Treg immunity in a pulmonary model of fungal infection (2017)
Araújo, Eliseu Frank de; Feriotti, Claudia; Galdino, Nayane Alves de Lima; Preite, Nycolas Willian; Calich, Vera Lúcia Garcia; Loures, Flávio Vieira
Source: Frontiers in Immunology. Unidade: ICB
Subjects: IMUNOLOGIA, PARACOCCIDIOIDOMICOSE, LINFÓCITOS T, PARACOCCIDIOIDES BRASILIENSIS, ENZIMAS OXIRREDUTORAS, CÉLULAS DENDRÍTICAS
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ARAÚJO, Eliseu Frank de et al. The IDO-AhR axis controls Th17/Treg immunity in a pulmonary model of fungal infection. Frontiers in Immunology, v. 8, n. 880, p. 1-15, 2017Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2017.00880. Acesso em: 17 nov. 2024.
APA
Araújo, E. F. de, Feriotti, C., Galdino, N. A. de L., Preite, N. W., Calich, V. L. G., & Loures, F. V. (2017). The IDO-AhR axis controls Th17/Treg immunity in a pulmonary model of fungal infection. Frontiers in Immunology, 8( 880), 1-15. doi:10.3389/fimmu.2017.00880
NLM
Araújo EF de, Feriotti C, Galdino NA de L, Preite NW, Calich VLG, Loures FV. The IDO-AhR axis controls Th17/Treg immunity in a pulmonary model of fungal infection [Internet]. Frontiers in Immunology. 2017 ; 8( 880): 1-15.[citado 2024 nov. 17 ] Available from: https://doi.org/10.3389/fimmu.2017.00880
Vancouver
Araújo EF de, Feriotti C, Galdino NA de L, Preite NW, Calich VLG, Loures FV. The IDO-AhR axis controls Th17/Treg immunity in a pulmonary model of fungal infection [Internet]. Frontiers in Immunology. 2017 ; 8( 880): 1-15.[citado 2024 nov. 17 ] Available from: https://doi.org/10.3389/fimmu.2017.00880
Artigo de periodico
Tolerogenic plasmacytoid dendritic cells control Paracoccidioides brasiliensis infection by inducting regulatory T cells in an IDO-dependent manner (2016)
Araújo, Eliseu Frank de; Medeiros, Daniella Helena; Galdino, Nayane Alves de Lima; Condino-Neto, Antônio; Calich, Vera Lúcia Garcia; Loures, Flávio Vieira
Source: PLoS Pathogens. Unidade: ICB
Assunto: IMUNOLOGIA
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ARAÚJO, Eliseu Frank de et al. Tolerogenic plasmacytoid dendritic cells control Paracoccidioides brasiliensis infection by inducting regulatory T cells in an IDO-dependent manner. PLoS Pathogens, v. 12, n. 12, p. 1-29, 2016Tradução . . Disponível em: https://doi.org/10.1371/journal.ppat.1006115. Acesso em: 17 nov. 2024.
APA
Araújo, E. F. de, Medeiros, D. H., Galdino, N. A. de L., Condino-Neto, A., Calich, V. L. G., & Loures, F. V. (2016). Tolerogenic plasmacytoid dendritic cells control Paracoccidioides brasiliensis infection by inducting regulatory T cells in an IDO-dependent manner. PLoS Pathogens, 12( 12), 1-29. doi:10.1371/journal.ppat.1006115
NLM
Araújo EF de, Medeiros DH, Galdino NA de L, Condino-Neto A, Calich VLG, Loures FV. Tolerogenic plasmacytoid dendritic cells control Paracoccidioides brasiliensis infection by inducting regulatory T cells in an IDO-dependent manner [Internet]. PLoS Pathogens. 2016 ; 12( 12): 1-29.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1371/journal.ppat.1006115
Vancouver
Araújo EF de, Medeiros DH, Galdino NA de L, Condino-Neto A, Calich VLG, Loures FV. Tolerogenic plasmacytoid dendritic cells control Paracoccidioides brasiliensis infection by inducting regulatory T cells in an IDO-dependent manner [Internet]. PLoS Pathogens. 2016 ; 12( 12): 1-29.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1371/journal.ppat.1006115
Artigo de periodico
Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis (2015)
Feriotti, Claudia; Bazan, Silvia Boschi; Loures, Flávio Vieira; Araújo, Eliseu Frank de; Costa, Tânia Alves da; Calich, Vera Lucia Garcia
Source: Frontiers in Microbiology. Unidade: ICB
Subjects: IMUNOLOGIA, PARACOCCIDIOIDOMICOSE (RESISTÊNCIA), INFLAMAÇÃO
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FERIOTTI, Claudia et al. Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis. Frontiers in Microbiology, v. 6, n. 913, p. 1-13, 2015Tradução . . Disponível em: https://doi.org/10.3389/fmicb.2015.00913. Acesso em: 17 nov. 2024.
APA
Feriotti, C., Bazan, S. B., Loures, F. V., Araújo, E. F. de, Costa, T. A. da, & Calich, V. L. G. (2015). Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis. Frontiers in Microbiology, 6( 913), 1-13. doi:10.3389/fmicb.2015.00913
NLM
Feriotti C, Bazan SB, Loures FV, Araújo EF de, Costa TA da, Calich VLG. Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis [Internet]. Frontiers in Microbiology. 2015 ; 6( 913): 1-13.[citado 2024 nov. 17 ] Available from: https://doi.org/10.3389/fmicb.2015.00913
Vancouver
Feriotti C, Bazan SB, Loures FV, Araújo EF de, Costa TA da, Calich VLG. Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis [Internet]. Frontiers in Microbiology. 2015 ; 6( 913): 1-13.[citado 2024 nov. 17 ] Available from: https://doi.org/10.3389/fmicb.2015.00913
Artigo de periodico
TLR-4 cooperates with Dectin-1 and mannose receptor to expand Th17 and Tc17 cells induced by Paracoccidioides brasiliensis stimulated dendritic cells (2015)
Loures, Flávio Vieira; Araújo, Eliseu Frank de; Feriotti, Claudia; Bazan, Silvia Boschi; Calich, Vera Lucia Garcia
Source: Frontiers in Microbiology. Unidade: ICB
Subjects: IMUNOLOGIA, PARACOCCIDIOIDES, PARACOCCIDIOIDES BRASILIENSIS, CÉLULAS DENDRITICAS
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LOURES, Flávio Vieira et al. TLR-4 cooperates with Dectin-1 and mannose receptor to expand Th17 and Tc17 cells induced by Paracoccidioides brasiliensis stimulated dendritic cells. Frontiers in Microbiology, v. 6, n. 261, p. 1-11, 2015Tradução . . Disponível em: https://doi.org/10.3389/fmicb.2015.00261. Acesso em: 17 nov. 2024.
APA
Loures, F. V., Araújo, E. F. de, Feriotti, C., Bazan, S. B., & Calich, V. L. G. (2015). TLR-4 cooperates with Dectin-1 and mannose receptor to expand Th17 and Tc17 cells induced by Paracoccidioides brasiliensis stimulated dendritic cells. Frontiers in Microbiology, 6( 261), 1-11. doi:10.3389/fmicb.2015.00261
NLM
Loures FV, Araújo EF de, Feriotti C, Bazan SB, Calich VLG. TLR-4 cooperates with Dectin-1 and mannose receptor to expand Th17 and Tc17 cells induced by Paracoccidioides brasiliensis stimulated dendritic cells [Internet]. Frontiers in Microbiology. 2015 ; 6( 261): 1-11.[citado 2024 nov. 17 ] Available from: https://doi.org/10.3389/fmicb.2015.00261
Vancouver
Loures FV, Araújo EF de, Feriotti C, Bazan SB, Calich VLG. TLR-4 cooperates with Dectin-1 and mannose receptor to expand Th17 and Tc17 cells induced by Paracoccidioides brasiliensis stimulated dendritic cells [Internet]. Frontiers in Microbiology. 2015 ; 6( 261): 1-11.[citado 2024 nov. 17 ] Available from: https://doi.org/10.3389/fmicb.2015.00261
Artigo de periodico
Recognition of Aspergillus fumigatus hyphae by human plasmacytoid dendritic cells is mediated by dectin-2 and results in formation of extracellular traps (2015)
Loures, Flávio Vieira; Röhm, Marc; Lee, Chrono K.; Santos, Evelyn; Wang, Jennifer P.; Specht, Charles A.; Calich, Vera Lucia Garcia; Urban, Constantin F.; Levitz, Stuart M.
Source: PLoS Pathogens. Unidade: ICB
Subjects: IMUNOLOGIA, ASPERGILLUS, CÉLULAS DENDRÍTICAS, ANTIFÚNGICOS
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LOURES, Flávio Vieira et al. Recognition of Aspergillus fumigatus hyphae by human plasmacytoid dendritic cells is mediated by dectin-2 and results in formation of extracellular traps. PLoS Pathogens, v. 11, n. 2, p. 1-23, 2015Tradução . . Disponível em: https://doi.org/10.1371/journal.ppat.1004643. Acesso em: 17 nov. 2024.
APA
Loures, F. V., Röhm, M., Lee, C. K., Santos, E., Wang, J. P., Specht, C. A., et al. (2015). Recognition of Aspergillus fumigatus hyphae by human plasmacytoid dendritic cells is mediated by dectin-2 and results in formation of extracellular traps. PLoS Pathogens, 11( 2), 1-23. doi:10.1371/journal.ppat.1004643
NLM
Loures FV, Röhm M, Lee CK, Santos E, Wang JP, Specht CA, Calich VLG, Urban CF, Levitz SM. Recognition of Aspergillus fumigatus hyphae by human plasmacytoid dendritic cells is mediated by dectin-2 and results in formation of extracellular traps [Internet]. PLoS Pathogens. 2015 ; 11( 2): 1-23.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1371/journal.ppat.1004643
Vancouver
Loures FV, Röhm M, Lee CK, Santos E, Wang JP, Specht CA, Calich VLG, Urban CF, Levitz SM. Recognition of Aspergillus fumigatus hyphae by human plasmacytoid dendritic cells is mediated by dectin-2 and results in formation of extracellular traps [Internet]. PLoS Pathogens. 2015 ; 11( 2): 1-23.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1371/journal.ppat.1004643
Artigo de periodico
Loss- and gain-of-function approaches indicate a dual role exerted by regulatory T cells in pulmonary paracoccidioidomycosis (2015)
Bazan, Silvia Boschi; Costa, Tania Alves da; Araújo, Eliseu Frank de; Feriotti, Claudia; Loures, Flávio Vieira; Pretel, Fernando Delgado; Calich, Vera Lucia Garcia
Source: PLoS Neglected Tropical Diseases. Unidade: ICB
Subjects: IMUNOLOGIA, PARACOCCIDIOIDOMICOSE, IMUNIDADE CELULAR, CAMUNDONGOS
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ABNT
BAZAN, Silvia Boschi et al. Loss- and gain-of-function approaches indicate a dual role exerted by regulatory T cells in pulmonary paracoccidioidomycosis. PLoS Neglected Tropical Diseases, v. 9, n. 10, p. 1-24, 2015Tradução . . Disponível em: https://doi.org/10.1371/journal.pntd.0004189. Acesso em: 17 nov. 2024.
APA
Bazan, S. B., Costa, T. A. da, Araújo, E. F. de, Feriotti, C., Loures, F. V., Pretel, F. D., & Calich, V. L. G. (2015). Loss- and gain-of-function approaches indicate a dual role exerted by regulatory T cells in pulmonary paracoccidioidomycosis. PLoS Neglected Tropical Diseases, 9( 10), 1-24. doi:10.1371/journal.pntd.0004189
NLM
Bazan SB, Costa TA da, Araújo EF de, Feriotti C, Loures FV, Pretel FD, Calich VLG. Loss- and gain-of-function approaches indicate a dual role exerted by regulatory T cells in pulmonary paracoccidioidomycosis [Internet]. PLoS Neglected Tropical Diseases. 2015 ; 9( 10): 1-24.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1371/journal.pntd.0004189
Vancouver
Bazan SB, Costa TA da, Araújo EF de, Feriotti C, Loures FV, Pretel FD, Calich VLG. Loss- and gain-of-function approaches indicate a dual role exerted by regulatory T cells in pulmonary paracoccidioidomycosis [Internet]. PLoS Neglected Tropical Diseases. 2015 ; 9( 10): 1-24.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1371/journal.pntd.0004189
Artigo de periodico
Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis (2015)
Ribeiro, Laura Rossetti Rios; Loures, Flávio Vieira; Araújo, Eliseu Frank de; Feriotti, Cláudia; Costa, Tânia Alves da; Serezani, Carlos Henrique; Jancar, Sonia ; Calich, Vera Lucia Garcia
Source: Mediators of Inflammation. Unidade: ICB
Subjects: IMUNOLOGIA, PARACOCCIDIOIDES BRASILIENSIS, MACRÓFAGOS, INFECÇÕES RESPIRATÓRIAS
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RIBEIRO, Laura Rossetti Rios et al. Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis. Mediators of Inflammation, v. 2015, p. 1-17, 2015Tradução . . Disponível em: https://doi.org/10.1155/2015/852574. Acesso em: 17 nov. 2024.
APA
Ribeiro, L. R. R., Loures, F. V., Araújo, E. F. de, Feriotti, C., Costa, T. A. da, Serezani, C. H., et al. (2015). Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis. Mediators of Inflammation, 2015, 1-17. doi:10.1155/2015/852574
NLM
Ribeiro LRR, Loures FV, Araújo EF de, Feriotti C, Costa TA da, Serezani CH, Jancar S, Calich VLG. Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis [Internet]. Mediators of Inflammation. 2015 ; 2015 1-17.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1155/2015/852574
Vancouver
Ribeiro LRR, Loures FV, Araújo EF de, Feriotti C, Costa TA da, Serezani CH, Jancar S, Calich VLG. Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis [Internet]. Mediators of Inflammation. 2015 ; 2015 1-17.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1155/2015/852574
Artigo de periodico
Myeloid dendritic cells (DCs) of mice susceptible to Paracoccidioidomycosis suppress T cell responses whereas myeloid and plasmacytoid DCs from resistant mice induce effector and regulatory T cells (2013)
Pina, Adriana; Araujo, Eliseu Frank de; Felonato, Maíra; Loures, Flávio Vieira; Feriotti, Claudia; Bernardino, Simone; Barbuto, José Alexandre Marzagão ; Calich, Vera Lucia Garcia
Source: Infection and Immunity. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
PINA, Adriana et al. Myeloid dendritic cells (DCs) of mice susceptible to Paracoccidioidomycosis suppress T cell responses whereas myeloid and plasmacytoid DCs from resistant mice induce effector and regulatory T cells. Infection and Immunity, v. 81, n. 4, p. 1064-1077, 2013Tradução . . Disponível em: https://doi.org/10.1128/IAI.00736-12. Acesso em: 17 nov. 2024.
APA
Pina, A., Araujo, E. F. de, Felonato, M., Loures, F. V., Feriotti, C., Bernardino, S., et al. (2013). Myeloid dendritic cells (DCs) of mice susceptible to Paracoccidioidomycosis suppress T cell responses whereas myeloid and plasmacytoid DCs from resistant mice induce effector and regulatory T cells. Infection and Immunity, 81( 4), 1064-1077. doi:10.1128/IAI.00736-12
NLM
Pina A, Araujo EF de, Felonato M, Loures FV, Feriotti C, Bernardino S, Barbuto JAM, Calich VLG. Myeloid dendritic cells (DCs) of mice susceptible to Paracoccidioidomycosis suppress T cell responses whereas myeloid and plasmacytoid DCs from resistant mice induce effector and regulatory T cells [Internet]. Infection and Immunity. 2013 ; 81( 4): 1064-1077.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1128/IAI.00736-12
Vancouver
Pina A, Araujo EF de, Felonato M, Loures FV, Feriotti C, Bernardino S, Barbuto JAM, Calich VLG. Myeloid dendritic cells (DCs) of mice susceptible to Paracoccidioidomycosis suppress T cell responses whereas myeloid and plasmacytoid DCs from resistant mice induce effector and regulatory T cells [Internet]. Infection and Immunity. 2013 ; 81( 4): 1064-1077.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1128/IAI.00736-12
Artigo de periodico
Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection (2013)
Feriotti, Claudia; Loures, Flávio Vieira; Araújo, Eliseu Frank de; Costa, Tania Alves da; Calich, Vera Lucia Garcia
Source: PLOS ONE. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FERIOTTI, Claudia et al. Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection. PLOS ONE, v. 8, n. 1, p. 1-17, 2013Tradução . . Disponível em: https://doi.org/10.1590/S0004-27302013000100003. Acesso em: 17 nov. 2024.
APA
Feriotti, C., Loures, F. V., Araújo, E. F. de, Costa, T. A. da, & Calich, V. L. G. (2013). Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection. PLOS ONE, 8( 1), 1-17. doi:10.1590/S0004-27302013000100003
NLM
Feriotti C, Loures FV, Araújo EF de, Costa TA da, Calich VLG. Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection [Internet]. PLOS ONE. 2013 ; 8( 1): 1-17.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1590/S0004-27302013000100003
Vancouver
Feriotti C, Loures FV, Araújo EF de, Costa TA da, Calich VLG. Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection [Internet]. PLOS ONE. 2013 ; 8( 1): 1-17.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1590/S0004-27302013000100003
Artigo de periodico
Paracoccidioides brasiliensis lipids modulate macrophage activity via Toll-dependent or independent mechanisms (2012)
Loures, Flávio Vieira; Stroeder, Heleni; Almeida, Igor; Calich, Vera Lucia Garcia
Source: FEMS Immunology and Medical Microbiology. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
LOURES, Flávio Vieira et al. Paracoccidioides brasiliensis lipids modulate macrophage activity via Toll-dependent or independent mechanisms. FEMS Immunology and Medical Microbiology, v. 66, n. 1, p. 58-70, 2012Tradução . . Disponível em: https://doi.org/10.1111/j.1574-695X.2012.00986.x. Acesso em: 17 nov. 2024.
APA
Loures, F. V., Stroeder, H., Almeida, I., & Calich, V. L. G. (2012). Paracoccidioides brasiliensis lipids modulate macrophage activity via Toll-dependent or independent mechanisms. FEMS Immunology and Medical Microbiology, 66( 1), 58-70. doi:10.1111/j.1574-695X.2012.00986.x
NLM
Loures FV, Stroeder H, Almeida I, Calich VLG. Paracoccidioides brasiliensis lipids modulate macrophage activity via Toll-dependent or independent mechanisms [Internet]. FEMS Immunology and Medical Microbiology. 2012 ; 66( 1): 58-70.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1111/j.1574-695X.2012.00986.x
Vancouver
Loures FV, Stroeder H, Almeida I, Calich VLG. Paracoccidioides brasiliensis lipids modulate macrophage activity via Toll-dependent or independent mechanisms [Internet]. FEMS Immunology and Medical Microbiology. 2012 ; 66( 1): 58-70.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1111/j.1574-695X.2012.00986.x
Artigo de periodico
Anti-CD25 treatment depletes treg cells and decreases disease severity in susceptible and resistant mice infected with Paracoccidioides brasiliensis (2012)
Felonato, Maíra; Pina, Adriana; Araujo, Eliseu Frank de; Loures, Flávio Vieira; Bazan, Silvia B.; Feriotti, Cláudia; Calich, Vera Lucia Garcia
Source: PLOS ONE. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FELONATO, Maíra et al. Anti-CD25 treatment depletes treg cells and decreases disease severity in susceptible and resistant mice infected with Paracoccidioides brasiliensis. PLOS ONE, v. 7, n. 11, p. 1-20, 2012Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0051071. Acesso em: 17 nov. 2024.
APA
Felonato, M., Pina, A., Araujo, E. F. de, Loures, F. V., Bazan, S. B., Feriotti, C., & Calich, V. L. G. (2012). Anti-CD25 treatment depletes treg cells and decreases disease severity in susceptible and resistant mice infected with Paracoccidioides brasiliensis. PLOS ONE, 7( 11), 1-20. doi:10.1371/journal.pone.0051071
NLM
Felonato M, Pina A, Araujo EF de, Loures FV, Bazan SB, Feriotti C, Calich VLG. Anti-CD25 treatment depletes treg cells and decreases disease severity in susceptible and resistant mice infected with Paracoccidioides brasiliensis [Internet]. PLOS ONE. 2012 ; 7( 11): 1-20.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1371/journal.pone.0051071
Vancouver
Felonato M, Pina A, Araujo EF de, Loures FV, Bazan SB, Feriotti C, Calich VLG. Anti-CD25 treatment depletes treg cells and decreases disease severity in susceptible and resistant mice infected with Paracoccidioides brasiliensis [Internet]. PLOS ONE. 2012 ; 7( 11): 1-20.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1371/journal.pone.0051071
Artigo de periodico
MyD88 signaling is required for efficient innate and adaptive immune responses to Paracoccidioides brasiliensis infection (2011)
Loures, Flávio Vieira; Pina, Adriana; Felonato, Maíra; Feriotti, Claudia; Araújo, Eliseu Frank de; Calich, Vera Lucia Garcia
Source: Infection and Immunity. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LOURES, Flávio Vieira et al. MyD88 signaling is required for efficient innate and adaptive immune responses to Paracoccidioides brasiliensis infection. Infection and Immunity, v. 79, n. 6, p. 2470-2480, 2011Tradução . . Disponível em: https://doi.org/10.1128/IAI.00375-10. Acesso em: 17 nov. 2024.
APA
Loures, F. V., Pina, A., Felonato, M., Feriotti, C., Araújo, E. F. de, & Calich, V. L. G. (2011). MyD88 signaling is required for efficient innate and adaptive immune responses to Paracoccidioides brasiliensis infection. Infection and Immunity, 79( 6), 2470-2480. doi:10.1128/IAI.00375-10
NLM
Loures FV, Pina A, Felonato M, Feriotti C, Araújo EF de, Calich VLG. MyD88 signaling is required for efficient innate and adaptive immune responses to Paracoccidioides brasiliensis infection [Internet]. Infection and Immunity. 2011 ; 79( 6): 2470-2480.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1128/IAI.00375-10
Vancouver
Loures FV, Pina A, Felonato M, Feriotti C, Araújo EF de, Calich VLG. MyD88 signaling is required for efficient innate and adaptive immune responses to Paracoccidioides brasiliensis infection [Internet]. Infection and Immunity. 2011 ; 79( 6): 2470-2480.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1128/IAI.00375-10
Artigo de periodico
CD28 exerts protective and detrimental effects in a pulmonary model of paracoccidioidomycosis (2010)
Felonato, Maíra; Pina, Adriana; Bernardino, Simone; Loures, Flávio Vieira; Araújo, Eliseu Frank de; Calich, Vera Lúcia Garcia
Source: Infection and Immunity. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FELONATO, Maíra et al. CD28 exerts protective and detrimental effects in a pulmonary model of paracoccidioidomycosis. Infection and Immunity, v. 78, n. 11, p. 4922-4935, 2010Tradução . . Disponível em: https://doi.org/10.1128/IAI.00297-10. Acesso em: 17 nov. 2024.
APA
Felonato, M., Pina, A., Bernardino, S., Loures, F. V., Araújo, E. F. de, & Calich, V. L. G. (2010). CD28 exerts protective and detrimental effects in a pulmonary model of paracoccidioidomycosis. Infection and Immunity, 78( 11), 4922-4935. doi:10.1128/IAI.00297-10
NLM
Felonato M, Pina A, Bernardino S, Loures FV, Araújo EF de, Calich VLG. CD28 exerts protective and detrimental effects in a pulmonary model of paracoccidioidomycosis [Internet]. Infection and Immunity. 2010 ; 78( 11): 4922-4935.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1128/IAI.00297-10
Vancouver
Felonato M, Pina A, Bernardino S, Loures FV, Araújo EF de, Calich VLG. CD28 exerts protective and detrimental effects in a pulmonary model of paracoccidioidomycosis [Internet]. Infection and Immunity. 2010 ; 78( 11): 4922-4935.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1128/IAI.00297-10
Artigo de periodico
Toll-Like receptor 4 signaling leads to severe fungal infection associated with enhanced proinflammatory immunity and impaired expansion of regulatory T cells (2010)
Loures, Flávio Vieira; Pina, Adriana; Felonato, Maíra; Araújo, Eliseu Frank de; Leite, Katia R. M.; Calich, Vera Lúcia Garcia
Source: Infection and Immunity. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LOURES, Flávio Vieira et al. Toll-Like receptor 4 signaling leads to severe fungal infection associated with enhanced proinflammatory immunity and impaired expansion of regulatory T cells. Infection and Immunity, v. 78, n. 3, p. 1078-1088, 2010Tradução . . Acesso em: 17 nov. 2024.
APA
Loures, F. V., Pina, A., Felonato, M., Araújo, E. F. de, Leite, K. R. M., & Calich, V. L. G. (2010). Toll-Like receptor 4 signaling leads to severe fungal infection associated with enhanced proinflammatory immunity and impaired expansion of regulatory T cells. Infection and Immunity, 78( 3), 1078-1088.
NLM
Loures FV, Pina A, Felonato M, Araújo EF de, Leite KRM, Calich VLG. Toll-Like receptor 4 signaling leads to severe fungal infection associated with enhanced proinflammatory immunity and impaired expansion of regulatory T cells. Infection and Immunity. 2010 ; 78( 3): 1078-1088.[citado 2024 nov. 17 ]
Vancouver
Loures FV, Pina A, Felonato M, Araújo EF de, Leite KRM, Calich VLG. Toll-Like receptor 4 signaling leads to severe fungal infection associated with enhanced proinflammatory immunity and impaired expansion of regulatory T cells. Infection and Immunity. 2010 ; 78( 3): 1078-1088.[citado 2024 nov. 17 ]
Artigo de periodico
TLR2 is a negative regulator of Th17 cells and tissue pathology in a pulmonary model of fungal infection (2009)
Loures, Flávio Vieira; Pina, Adriana; Felonato, Maíra; Calich, Vera Lúcia Garcia
Source: Journal of Immunology. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LOURES, Flávio Vieira et al. TLR2 is a negative regulator of Th17 cells and tissue pathology in a pulmonary model of fungal infection. Journal of Immunology, v. 183, n. 2, p. 1279-90, 2009Tradução . . Acesso em: 17 nov. 2024.
APA
Loures, F. V., Pina, A., Felonato, M., & Calich, V. L. G. (2009). TLR2 is a negative regulator of Th17 cells and tissue pathology in a pulmonary model of fungal infection. Journal of Immunology, 183( 2), 1279-90.
NLM
Loures FV, Pina A, Felonato M, Calich VLG. TLR2 is a negative regulator of Th17 cells and tissue pathology in a pulmonary model of fungal infection. Journal of Immunology. 2009 ; 183( 2): 1279-90.[citado 2024 nov. 17 ]
Vancouver
Loures FV, Pina A, Felonato M, Calich VLG. TLR2 is a negative regulator of Th17 cells and tissue pathology in a pulmonary model of fungal infection. Journal of Immunology. 2009 ; 183( 2): 1279-90.[citado 2024 nov. 17 ]
Artigo de periodico
Innate immunity to Paracoccidioides brasiliensis infection (2008)
Calich, Vera Lúcia Garcia; Costa, Tânia Alves da; Felonato, Maíra; Arruda, Celina; Bernardino, Simone; Loures, Flávio Vieira; Ribeiro, Laura Raquel Rios; Valente-Ferreira, Rita de Cássia; Pina, Adriana
Source: Mycopathologia. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CALICH, Vera Lúcia Garcia et al. Innate immunity to Paracoccidioides brasiliensis infection. Mycopathologia, v. 165, n. 4-5, p. 223-236, 2008Tradução . . Disponível em: https://doi.org/10.1007/s11046-007-9048-1. Acesso em: 17 nov. 2024.
APA
Calich, V. L. G., Costa, T. A. da, Felonato, M., Arruda, C., Bernardino, S., Loures, F. V., et al. (2008). Innate immunity to Paracoccidioides brasiliensis infection. Mycopathologia, 165( 4-5), 223-236. doi:10.1007/s11046-007-9048-1
NLM
Calich VLG, Costa TA da, Felonato M, Arruda C, Bernardino S, Loures FV, Ribeiro LRR, Valente-Ferreira R de C, Pina A. Innate immunity to Paracoccidioides brasiliensis infection [Internet]. Mycopathologia. 2008 ; 165( 4-5): 223-236.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1007/s11046-007-9048-1
Vancouver
Calich VLG, Costa TA da, Felonato M, Arruda C, Bernardino S, Loures FV, Ribeiro LRR, Valente-Ferreira R de C, Pina A. Innate immunity to Paracoccidioides brasiliensis infection [Internet]. Mycopathologia. 2008 ; 165( 4-5): 223-236.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1007/s11046-007-9048-1
Trabalho de evento-resumo
The role of TLR-4 and TLR-2 in murine Paracoccidioidomycosis (PCM) (2005)
Loures, Flávio Vieira; Calich, Vera Lúcia Garcia
Source: Resumos. Conference titles: Congresso do Instituto de Ciências Biomédicas. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LOURES, Flávio Vieira e CALICH, Vera Lúcia Garcia. The role of TLR-4 and TLR-2 in murine Paracoccidioidomycosis (PCM). 2005, Anais.. São Paulo: Comissão de Cultura e Extensão Universitária do ICB/USP, 2005. . Acesso em: 17 nov. 2024.
APA
Loures, F. V., & Calich, V. L. G. (2005). The role of TLR-4 and TLR-2 in murine Paracoccidioidomycosis (PCM). In Resumos. São Paulo: Comissão de Cultura e Extensão Universitária do ICB/USP.
NLM
Loures FV, Calich VLG. The role of TLR-4 and TLR-2 in murine Paracoccidioidomycosis (PCM). Resumos. 2005 ;[citado 2024 nov. 17 ]
Vancouver
Loures FV, Calich VLG. The role of TLR-4 and TLR-2 in murine Paracoccidioidomycosis (PCM). Resumos. 2005 ;[citado 2024 nov. 17 ]

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