The role of Rip2 in the signaling pathwy downstream of NOD1 in response to Trypanosoma cruz/infection (2010)
- Authors:
- USP affiliated authors: ZAMBONI, DARIO SIMÕES - FMRP ; SILVA, JOÃO SANTANA DA - FMRP
- Unidade: FMRP
- Subjects: RECEPTORES; MACRÓFAGOS; TRYPANOSOMA CRUZI
- Language: Inglês
- Imprenta:
- Source:
- Título: Proceedings
- Conference titles: Annual Meeting of the Brazilian Society of Protozoology
-
ABNT
HORTA, Catarina V. et al. The role of Rip2 in the signaling pathwy downstream of NOD1 in response to Trypanosoma cruz/infection. 2010, Anais.. São Paulo: SBPz, 2010. . Acesso em: 13 mar. 2026. -
APA
Horta, C. V., Silva, G. K., Silva, J. S. da, & Zamboni, D. S. (2010). The role of Rip2 in the signaling pathwy downstream of NOD1 in response to Trypanosoma cruz/infection. In Proceedings. São Paulo: SBPz. -
NLM
Horta CV, Silva GK, Silva JS da, Zamboni DS. The role of Rip2 in the signaling pathwy downstream of NOD1 in response to Trypanosoma cruz/infection. Proceedings. 2010 ;[citado 2026 mar. 13 ] -
Vancouver
Horta CV, Silva GK, Silva JS da, Zamboni DS. The role of Rip2 in the signaling pathwy downstream of NOD1 in response to Trypanosoma cruz/infection. Proceedings. 2010 ;[citado 2026 mar. 13 ] - Critical role of ASC inflammasomes in host innate resistance to Trypanosoma cruzi infection
- Nucleotide-binding oligomerization domain-containing protein 2 prompts potent inflammatory stimuli during Neospora caninum infection
- Cutting edge: nucleotide binding oligomerization domain 1-dependent responses account for murine resistance against trypanosoma cruzi infection
- Ros-dependent NLRP3-inflammasome activation is critical for restriction of Leishmania amazonensis infection
- Relevance of the myeloid differentiation factor 88 (MyD88) on RANKL, OPG, and nod expressions induced by TLR and IL-1R signaling in bone marrow stromal cells
- Role of NLRC4 inflammasome in Paracoccidioides brasiliensis pulmonary inflammation
- The inflammatose contributes for restriction of leishmania (Leishmania) amazonensis infection in macrophages and in vivo
- Resistance to Leishmania spp. is mediated by the inflammasome induced duo: IL- 1β and nitric oxide
- Restriction of Leishmania amazonensis infection by the inflammasomes
- NOD1 and NOD2 receptors account to innate and adaptive immune response against Leishmania infection
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