Surface antigens increase susceptibility to paracoccidioides brasiliensis infection via interleukin-4 production (2008)
- Authors:
- USP affiliated authors: SILVA, JOÃO SANTANA DA - FMRP ; ROSSI, MARCOS ANTONIO - FMRP ; OLIVEIRA, EDUARDO BRANDT DE - FMRP ; MARTINEZ, ROBERTO - FMRP
- Unidade: FMRP
- Subjects: PARACOCCIDIOIDOMICOSE; IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Conference titles: International Congress on Paracoccidioidomycosis
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ABNT
OLIVEIRA, L. L. et al. Surface antigens increase susceptibility to paracoccidioides brasiliensis infection via interleukin-4 production. Biomédica. Bogotá: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 09 out. 2024. , 2008 -
APA
Oliveira, L. L., Cavassani, K. A., Rocha, F. A., Vancim, J. O., Moreira, A. P., Campanelli, A. P., et al. (2008). Surface antigens increase susceptibility to paracoccidioides brasiliensis infection via interleukin-4 production. Biomédica. Bogotá: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. -
NLM
Oliveira LL, Cavassani KA, Rocha FA, Vancim JO, Moreira AP, Campanelli AP, Milanezi CM, Martinez R, Rossi MA, Oliveira EB de, Silva JS da. Surface antigens increase susceptibility to paracoccidioides brasiliensis infection via interleukin-4 production. Biomédica. 2008 ; 28 193.[citado 2024 out. 09 ] -
Vancouver
Oliveira LL, Cavassani KA, Rocha FA, Vancim JO, Moreira AP, Campanelli AP, Milanezi CM, Martinez R, Rossi MA, Oliveira EB de, Silva JS da. Surface antigens increase susceptibility to paracoccidioides brasiliensis infection via interleukin-4 production. Biomédica. 2008 ; 28 193.[citado 2024 out. 09 ] - IL-12 and IL-4 modulates of the resistance to Paracoccidioides brasiliensis infection
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- Intercellular adhesion molecule-1 is required for the early formation of granulomas and participates in the resistance of mice to the infection with the fungus Paracoccidioides brasiliensis
- Anti- 'CD IND.4' abrogates rejection and restabliches long-term tolerance to sybgenetic newborn hearts grafted in mice chronically infected with trypanosoma cruzi
- Intercellular adhesion molecule 1 deficieney leads to impaired recruitment of T lymphocytes and enhanced host susceptibility to infection with Trypanosoma cruzi
- Role of interleukin (IL)-18 in experimental paracoccidioidomycosis
- B cell-deficient mice display enhanced susceptibility to Paracoccidioides brasiliensis infection
- Chemokine production and leukocyte recruitment to the lungs of Paracoccidioides brasiliensis-infected mice is modulated by interferon-'gama'
- Cell-free antigens from Paracoccidioides brasiliensis drive IL-4 production and increase the severity of paracoccidioidomycosis
- nitric oxide synthase-2 modulates chemokine production by Trypanosoma Cruzi-infected cardiac myocytes
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