IL-12 and IL-4 modulates of the resistance to Paracoccidioides brasiliensis infection (2000)
- Authors:
- USP affiliated authors: MARTINEZ, ROBERTO - FMRP ; ROSSI, MARCOS ANTONIO - FMRP ; SILVA, JOAO SANTANA DA - FMRP
- Unidade: FMRP
- Assunto: DOENÇAS INFECCIOSAS
- Language: Inglês
- Imprenta:
- Publisher place: Florianópolis
- Date published: 2000
- Conference titles: Meeting of the Brazilian Society of Immunology
-
ABNT
LIVONESI, M. C. et al. IL-12 and IL-4 modulates of the resistance to Paracoccidioides brasiliensis infection. 2000, Anais.. Florianópolis: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2000. . Acesso em: 17 fev. 2026. -
APA
Livonesi, M. C., Souto, J. T., Campanelli, A. P., Martinez, R., Rossi, M. A., & Silva, J. S. da. (2000). IL-12 and IL-4 modulates of the resistance to Paracoccidioides brasiliensis infection. In . Florianópolis: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. -
NLM
Livonesi MC, Souto JT, Campanelli AP, Martinez R, Rossi MA, Silva JS da. IL-12 and IL-4 modulates of the resistance to Paracoccidioides brasiliensis infection. 2000 ;[citado 2026 fev. 17 ] -
Vancouver
Livonesi MC, Souto JT, Campanelli AP, Martinez R, Rossi MA, Silva JS da. IL-12 and IL-4 modulates of the resistance to Paracoccidioides brasiliensis infection. 2000 ;[citado 2026 fev. 17 ] - The role of 'CD4 POT.+'CD25 POT.+'T cells in the Paracoccidioides brasiliensis infection in mice
- Surface antigens increase susceptibility to paracoccidioides brasiliensis infection via interleukin-4 production
- Intercellular adhesion molecule-1 is required for the early formation of granulomas and participates in the resistance of mice to the infection with the fungus Paracoccidioides brasiliensis
- Chemokine production and leukocyte recruitment to the lungs of Paracoccidioides brasiliensis-infected mice is modulated by interferon-'gama'
- Cell-free antigens from Paracoccidioides brasiliensis drive IL-4 production and increase the severity of paracoccidioidomycosis
- nitric oxide synthase-2 modulates chemokine production by Trypanosoma Cruzi-infected cardiac myocytes
- B cell-deficient mice display enhanced susceptibility to Paracoccidioides brasiliensis infection
- Early dystrophin disruption in the pathogenesis of experimental chronic chagas cardiomyopathy
- Intercellular adhesion molecule 1 deficieney leads to impaired recruitment of T lymphocytes and enhanced host susceptibility to infection with Trypanosoma cruzi
- Early dystrophin disruption in the pathogenesis of experimental chronic Chagas cardiomyopathy
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