Inhibition of cys lt, receptors leads to a less severe pulmonary paracoccidioidomycosis (PCM) (2004)
- Authors:
- USP affiliated authors: NEGRO, SONIA JANCAR - ICB ; CALICH, VERA LUCIA GARCIA - ICB
- Unidade: ICB
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Publisher place: Ouro Preto
- Date published: 2004
- Source:
- Título: Final Program and Abstracts
- Conference titles: Sociedade Brasileira de Imunologia
-
ABNT
RIBEIRO, R. R. L. e JANCAR, Sonia e CALICH, Vera Lúcia Garcia. Inhibition of cys lt, receptors leads to a less severe pulmonary paracoccidioidomycosis (PCM). 2004, Anais.. Ouro Preto: Instituto de Ciências Biomédicas, Universidade de São Paulo, 2004. . Acesso em: 10 fev. 2026. -
APA
Ribeiro, R. R. L., Jancar, S., & Calich, V. L. G. (2004). Inhibition of cys lt, receptors leads to a less severe pulmonary paracoccidioidomycosis (PCM). In Final Program and Abstracts. Ouro Preto: Instituto de Ciências Biomédicas, Universidade de São Paulo. -
NLM
Ribeiro RRL, Jancar S, Calich VLG. Inhibition of cys lt, receptors leads to a less severe pulmonary paracoccidioidomycosis (PCM). Final Program and Abstracts. 2004 ;[citado 2026 fev. 10 ] -
Vancouver
Ribeiro RRL, Jancar S, Calich VLG. Inhibition of cys lt, receptors leads to a less severe pulmonary paracoccidioidomycosis (PCM). Final Program and Abstracts. 2004 ;[citado 2026 fev. 10 ] - In vivo inhibition of leukotrienes receptor induces a less severe pulmonary paracoccidioidomycosis associated with a decreased inflammatory polymorphonuclear leukocytes influx to the lungs
- Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis
- cd28 costimulatory molecule deficiency in more severe paracoccidioides brasiliensis infection but protects mice from life-threatening immunopathology
- Paracoccidioides brasiliensis infection determines dentric cells to diferentiate to the plasmacytoid subpopulation which induces a more severe pulmonary infection when transfered to resistant mice
- The IDO-AhR axis controls Th17/Treg immunity in a pulmonary model of fungal infection
- TLR-2 is a negative regulator of TH17 cells and tissue pathology in a pulmonary model of fungal infection
- Absence of TLR2 results in less severe paracoccidioidomycosis but increased inflammatory response caused by PMN & TH17 cells
- Toll like receptors and the adaptor molecule MYD88 play an important role in pulmonary paracoccidioidomycosis
- TLR2 is a negative regulator of Th17 cells and tissue pathology in a pulmonary model of fungal infection
- Human alfa / beta and gama / delta t cells respond to proteins of the yeast form of paracoccidioides brasiliensis
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