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Artigo de periodico
AhR ligands modulate the differentiation of innate lymphoid cells and T helper cell subsets that control the severity of a pulmonary fungal infection (2021)
Araújo, Eliseu Frank de; Loures, Flávio Vieira ; Preite, Nycolas Willian ; Feriotti, Cláudia ; Galdino, Nayane Alves de Lima ; Costa, Tania Alves da; Calich, Vera Lúcia Garcia
Source: Frontiers in Immunology. Unidade: ICB
Subjects: IMUNOLOGIA, LINFÓCITOS T, PARACOCCIDIOIDES, PARACOCCIDIOIDOMICOSE, INFECÇÕES RESPIRATÓRIAS, DOENÇAS RESPIRATÓRIAS, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS, LEUCÓCITOS, CÉLULAS DENDRÍTICAS
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ABNT
ARAÚJO, Eliseu Frank de et al. AhR ligands modulate the differentiation of innate lymphoid cells and T helper cell subsets that control the severity of a pulmonary fungal infection. Frontiers in Immunology, v. 12, p. 1-17, 2021Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2021.630938. Acesso em: 03 nov. 2024.
APA
Araújo, E. F. de, Loures, F. V., Preite, N. W., Feriotti, C., Galdino, N. A. de L., Costa, T. A. da, & Calich, V. L. G. (2021). AhR ligands modulate the differentiation of innate lymphoid cells and T helper cell subsets that control the severity of a pulmonary fungal infection. Frontiers in Immunology, 12, 1-17. doi:10.3389/fimmu.2021.630938
NLM
Araújo EF de, Loures FV, Preite NW, Feriotti C, Galdino NA de L, Costa TA da, Calich VLG. AhR ligands modulate the differentiation of innate lymphoid cells and T helper cell subsets that control the severity of a pulmonary fungal infection [Internet]. Frontiers in Immunology. 2021 ; 12 1-17.[citado 2024 nov. 03 ] Available from: https://doi.org/10.3389/fimmu.2021.630938
Vancouver
Araújo EF de, Loures FV, Preite NW, Feriotti C, Galdino NA de L, Costa TA da, Calich VLG. AhR ligands modulate the differentiation of innate lymphoid cells and T helper cell subsets that control the severity of a pulmonary fungal infection [Internet]. Frontiers in Immunology. 2021 ; 12 1-17.[citado 2024 nov. 03 ] Available from: https://doi.org/10.3389/fimmu.2021.630938
Artigo de periodico
The IDO-AhR axis controls Th17/Treg immunity in a pulmonary model of fungal infection (2017)
Araújo, Eliseu Frank de; Feriotti, Claudia; Galdino, Nayane Alves de Lima; Preite, Nycolas Willian; Calich, Vera Lúcia Garcia; Loures, Flávio Vieira
Source: Frontiers in Immunology. Unidade: ICB
Subjects: IMUNOLOGIA, PARACOCCIDIOIDOMICOSE, LINFÓCITOS T, PARACOCCIDIOIDES BRASILIENSIS, ENZIMAS OXIRREDUTORAS, CÉLULAS DENDRÍTICAS
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ARAÚJO, Eliseu Frank de et al. The IDO-AhR axis controls Th17/Treg immunity in a pulmonary model of fungal infection. Frontiers in Immunology, v. 8, n. 880, p. 1-15, 2017Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2017.00880. Acesso em: 03 nov. 2024.
APA
Araújo, E. F. de, Feriotti, C., Galdino, N. A. de L., Preite, N. W., Calich, V. L. G., & Loures, F. V. (2017). The IDO-AhR axis controls Th17/Treg immunity in a pulmonary model of fungal infection. Frontiers in Immunology, 8( 880), 1-15. doi:10.3389/fimmu.2017.00880
NLM
Araújo EF de, Feriotti C, Galdino NA de L, Preite NW, Calich VLG, Loures FV. The IDO-AhR axis controls Th17/Treg immunity in a pulmonary model of fungal infection [Internet]. Frontiers in Immunology. 2017 ; 8( 880): 1-15.[citado 2024 nov. 03 ] Available from: https://doi.org/10.3389/fimmu.2017.00880
Vancouver
Araújo EF de, Feriotti C, Galdino NA de L, Preite NW, Calich VLG, Loures FV. The IDO-AhR axis controls Th17/Treg immunity in a pulmonary model of fungal infection [Internet]. Frontiers in Immunology. 2017 ; 8( 880): 1-15.[citado 2024 nov. 03 ] Available from: https://doi.org/10.3389/fimmu.2017.00880
Artigo de periodico
Disease tolerance mediated by phosphorylated indoleamine-2,3 dioxygenase confers resistance to a primary fungal pathogen (2017)
Araújo, Eliseu Frank de; Loures, Flavio Vieira ; Feriotti, Cláudia ; Costa, Tania; Vacca, Carmine; Puccetti, Paolo; Romani, Luigina; Calich, Vera Lúcia Garcia
Source: Frontiers in Immunology. Unidade: ICB
Subjects: IMUNOLOGIA, PARACOCCIDIOIDOMICOSE, CÉLULAS DENDRÍTICAS, ATIVAÇÃO ENZIMÁTICA, ENZIMAS OXIRREDUTORAS
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ARAÚJO, Eliseu Frank de et al. Disease tolerance mediated by phosphorylated indoleamine-2,3 dioxygenase confers resistance to a primary fungal pathogen. Frontiers in Immunology, v. 8, n. 1522, p. 1-14, 2017Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2017.01522. Acesso em: 03 nov. 2024.
APA
Araújo, E. F. de, Loures, F. V., Feriotti, C., Costa, T., Vacca, C., Puccetti, P., et al. (2017). Disease tolerance mediated by phosphorylated indoleamine-2,3 dioxygenase confers resistance to a primary fungal pathogen. Frontiers in Immunology, 8( 1522), 1-14. doi:10.3389/fimmu.2017.01522
NLM
Araújo EF de, Loures FV, Feriotti C, Costa T, Vacca C, Puccetti P, Romani L, Calich VLG. Disease tolerance mediated by phosphorylated indoleamine-2,3 dioxygenase confers resistance to a primary fungal pathogen [Internet]. Frontiers in Immunology. 2017 ; 8( 1522): 1-14.[citado 2024 nov. 03 ] Available from: https://doi.org/10.3389/fimmu.2017.01522
Vancouver
Araújo EF de, Loures FV, Feriotti C, Costa T, Vacca C, Puccetti P, Romani L, Calich VLG. Disease tolerance mediated by phosphorylated indoleamine-2,3 dioxygenase confers resistance to a primary fungal pathogen [Internet]. Frontiers in Immunology. 2017 ; 8( 1522): 1-14.[citado 2024 nov. 03 ] Available from: https://doi.org/10.3389/fimmu.2017.01522
Artigo de periodico
Tolerogenic plasmacytoid dendritic cells control Paracoccidioides brasiliensis infection by inducting regulatory T cells in an IDO-dependent manner (2016)
Araújo, Eliseu Frank de; Medeiros, Daniella Helena; Galdino, Nayane Alves de Lima; Condino-Neto, Antônio; Calich, Vera Lúcia Garcia; Loures, Flávio Vieira
Source: PLoS Pathogens. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
ARAÚJO, Eliseu Frank de et al. Tolerogenic plasmacytoid dendritic cells control Paracoccidioides brasiliensis infection by inducting regulatory T cells in an IDO-dependent manner. PLoS Pathogens, v. 12, n. 12, p. 1-29, 2016Tradução . . Disponível em: https://doi.org/10.1371/journal.ppat.1006115. Acesso em: 03 nov. 2024.
APA
Araújo, E. F. de, Medeiros, D. H., Galdino, N. A. de L., Condino-Neto, A., Calich, V. L. G., & Loures, F. V. (2016). Tolerogenic plasmacytoid dendritic cells control Paracoccidioides brasiliensis infection by inducting regulatory T cells in an IDO-dependent manner. PLoS Pathogens, 12( 12), 1-29. doi:10.1371/journal.ppat.1006115
NLM
Araújo EF de, Medeiros DH, Galdino NA de L, Condino-Neto A, Calich VLG, Loures FV. Tolerogenic plasmacytoid dendritic cells control Paracoccidioides brasiliensis infection by inducting regulatory T cells in an IDO-dependent manner [Internet]. PLoS Pathogens. 2016 ; 12( 12): 1-29.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1371/journal.ppat.1006115
Vancouver
Araújo EF de, Medeiros DH, Galdino NA de L, Condino-Neto A, Calich VLG, Loures FV. Tolerogenic plasmacytoid dendritic cells control Paracoccidioides brasiliensis infection by inducting regulatory T cells in an IDO-dependent manner [Internet]. PLoS Pathogens. 2016 ; 12( 12): 1-29.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1371/journal.ppat.1006115
Artigo de periodico
Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis (2015)
Feriotti, Claudia; Bazan, Silvia Boschi; Loures, Flávio Vieira; Araújo, Eliseu Frank de; Costa, Tânia Alves da; Calich, Vera Lucia Garcia
Source: Frontiers in Microbiology. Unidade: ICB
Subjects: IMUNOLOGIA, PARACOCCIDIOIDOMICOSE (RESISTÊNCIA), INFLAMAÇÃO
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ABNT
FERIOTTI, Claudia et al. Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis. Frontiers in Microbiology, v. 6, n. 913, p. 1-13, 2015Tradução . . Disponível em: https://doi.org/10.3389/fmicb.2015.00913. Acesso em: 03 nov. 2024.
APA
Feriotti, C., Bazan, S. B., Loures, F. V., Araújo, E. F. de, Costa, T. A. da, & Calich, V. L. G. (2015). Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis. Frontiers in Microbiology, 6( 913), 1-13. doi:10.3389/fmicb.2015.00913
NLM
Feriotti C, Bazan SB, Loures FV, Araújo EF de, Costa TA da, Calich VLG. Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis [Internet]. Frontiers in Microbiology. 2015 ; 6( 913): 1-13.[citado 2024 nov. 03 ] Available from: https://doi.org/10.3389/fmicb.2015.00913
Vancouver
Feriotti C, Bazan SB, Loures FV, Araújo EF de, Costa TA da, Calich VLG. Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis [Internet]. Frontiers in Microbiology. 2015 ; 6( 913): 1-13.[citado 2024 nov. 03 ] Available from: https://doi.org/10.3389/fmicb.2015.00913
Artigo de periodico
TLR-4 cooperates with Dectin-1 and mannose receptor to expand Th17 and Tc17 cells induced by Paracoccidioides brasiliensis stimulated dendritic cells (2015)
Loures, Flávio Vieira; Araújo, Eliseu Frank de; Feriotti, Claudia; Bazan, Silvia Boschi; Calich, Vera Lucia Garcia
Source: Frontiers in Microbiology. Unidade: ICB
Subjects: IMUNOLOGIA, PARACOCCIDIOIDES, PARACOCCIDIOIDES BRASILIENSIS, CÉLULAS DENDRITICAS
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ABNT
LOURES, Flávio Vieira et al. TLR-4 cooperates with Dectin-1 and mannose receptor to expand Th17 and Tc17 cells induced by Paracoccidioides brasiliensis stimulated dendritic cells. Frontiers in Microbiology, v. 6, n. 261, p. 1-11, 2015Tradução . . Disponível em: https://doi.org/10.3389/fmicb.2015.00261. Acesso em: 03 nov. 2024.
APA
Loures, F. V., Araújo, E. F. de, Feriotti, C., Bazan, S. B., & Calich, V. L. G. (2015). TLR-4 cooperates with Dectin-1 and mannose receptor to expand Th17 and Tc17 cells induced by Paracoccidioides brasiliensis stimulated dendritic cells. Frontiers in Microbiology, 6( 261), 1-11. doi:10.3389/fmicb.2015.00261
NLM
Loures FV, Araújo EF de, Feriotti C, Bazan SB, Calich VLG. TLR-4 cooperates with Dectin-1 and mannose receptor to expand Th17 and Tc17 cells induced by Paracoccidioides brasiliensis stimulated dendritic cells [Internet]. Frontiers in Microbiology. 2015 ; 6( 261): 1-11.[citado 2024 nov. 03 ] Available from: https://doi.org/10.3389/fmicb.2015.00261
Vancouver
Loures FV, Araújo EF de, Feriotti C, Bazan SB, Calich VLG. TLR-4 cooperates with Dectin-1 and mannose receptor to expand Th17 and Tc17 cells induced by Paracoccidioides brasiliensis stimulated dendritic cells [Internet]. Frontiers in Microbiology. 2015 ; 6( 261): 1-11.[citado 2024 nov. 03 ] Available from: https://doi.org/10.3389/fmicb.2015.00261
Artigo de periodico
Loss- and gain-of-function approaches indicate a dual role exerted by regulatory T cells in pulmonary paracoccidioidomycosis (2015)
Bazan, Silvia Boschi; Costa, Tania Alves da; Araújo, Eliseu Frank de; Feriotti, Claudia; Loures, Flávio Vieira; Pretel, Fernando Delgado; Calich, Vera Lucia Garcia
Source: PLoS Neglected Tropical Diseases. Unidade: ICB
Subjects: IMUNOLOGIA, PARACOCCIDIOIDOMICOSE, IMUNIDADE CELULAR, CAMUNDONGOS
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ABNT
BAZAN, Silvia Boschi et al. Loss- and gain-of-function approaches indicate a dual role exerted by regulatory T cells in pulmonary paracoccidioidomycosis. PLoS Neglected Tropical Diseases, v. 9, n. 10, p. 1-24, 2015Tradução . . Disponível em: https://doi.org/10.1371/journal.pntd.0004189. Acesso em: 03 nov. 2024.
APA
Bazan, S. B., Costa, T. A. da, Araújo, E. F. de, Feriotti, C., Loures, F. V., Pretel, F. D., & Calich, V. L. G. (2015). Loss- and gain-of-function approaches indicate a dual role exerted by regulatory T cells in pulmonary paracoccidioidomycosis. PLoS Neglected Tropical Diseases, 9( 10), 1-24. doi:10.1371/journal.pntd.0004189
NLM
Bazan SB, Costa TA da, Araújo EF de, Feriotti C, Loures FV, Pretel FD, Calich VLG. Loss- and gain-of-function approaches indicate a dual role exerted by regulatory T cells in pulmonary paracoccidioidomycosis [Internet]. PLoS Neglected Tropical Diseases. 2015 ; 9( 10): 1-24.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1371/journal.pntd.0004189
Vancouver
Bazan SB, Costa TA da, Araújo EF de, Feriotti C, Loures FV, Pretel FD, Calich VLG. Loss- and gain-of-function approaches indicate a dual role exerted by regulatory T cells in pulmonary paracoccidioidomycosis [Internet]. PLoS Neglected Tropical Diseases. 2015 ; 9( 10): 1-24.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1371/journal.pntd.0004189
Artigo de periodico
Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis (2015)
Ribeiro, Laura Rossetti Rios; Loures, Flávio Vieira; Araújo, Eliseu Frank de; Feriotti, Cláudia; Costa, Tânia Alves da; Serezani, Carlos Henrique; Jancar, Sonia ; Calich, Vera Lucia Garcia
Source: Mediators of Inflammation. Unidade: ICB
Subjects: IMUNOLOGIA, PARACOCCIDIOIDES BRASILIENSIS, MACRÓFAGOS, INFECÇÕES RESPIRATÓRIAS
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ABNT
RIBEIRO, Laura Rossetti Rios et al. Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis. Mediators of Inflammation, v. 2015, p. 1-17, 2015Tradução . . Disponível em: https://doi.org/10.1155/2015/852574. Acesso em: 03 nov. 2024.
APA
Ribeiro, L. R. R., Loures, F. V., Araújo, E. F. de, Feriotti, C., Costa, T. A. da, Serezani, C. H., et al. (2015). Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis. Mediators of Inflammation, 2015, 1-17. doi:10.1155/2015/852574
NLM
Ribeiro LRR, Loures FV, Araújo EF de, Feriotti C, Costa TA da, Serezani CH, Jancar S, Calich VLG. Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis [Internet]. Mediators of Inflammation. 2015 ; 2015 1-17.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1155/2015/852574
Vancouver
Ribeiro LRR, Loures FV, Araújo EF de, Feriotti C, Costa TA da, Serezani CH, Jancar S, Calich VLG. Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis [Internet]. Mediators of Inflammation. 2015 ; 2015 1-17.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1155/2015/852574
Artigo de periodico
Dectin-1 induces M1 macrophages and prominent expansion of CD8+IL-17+ cells in pulmonary Paracoccidioidomycosis (2014)
Loures, Flávio V.; Araújo, Eliseu Frank de; Feriotti, Claudia; Bazan, Silvia B.; Costa, Tânia Alves da; Brown, Gordon D.; Calich, Vera Lucia Garcia
Source: Journal of Infectious Diseases. Unidade: ICB
Subjects: IMUNOLOGIA, MACRÓFAGOS, PARACOCCIDIOIDOMICOSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LOURES, Flávio V. et al. Dectin-1 induces M1 macrophages and prominent expansion of CD8+IL-17+ cells in pulmonary Paracoccidioidomycosis. Journal of Infectious Diseases, v. 210, n. 5, p. 762-773, 2014Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiu136. Acesso em: 03 nov. 2024.
APA
Loures, F. V., Araújo, E. F. de, Feriotti, C., Bazan, S. B., Costa, T. A. da, Brown, G. D., & Calich, V. L. G. (2014). Dectin-1 induces M1 macrophages and prominent expansion of CD8+IL-17+ cells in pulmonary Paracoccidioidomycosis. Journal of Infectious Diseases, 210( 5), 762-773. doi:10.1093/infdis/jiu136
NLM
Loures FV, Araújo EF de, Feriotti C, Bazan SB, Costa TA da, Brown GD, Calich VLG. Dectin-1 induces M1 macrophages and prominent expansion of CD8+IL-17+ cells in pulmonary Paracoccidioidomycosis [Internet]. Journal of Infectious Diseases. 2014 ; 210( 5): 762-773.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1093/infdis/jiu136
Vancouver
Loures FV, Araújo EF de, Feriotti C, Bazan SB, Costa TA da, Brown GD, Calich VLG. Dectin-1 induces M1 macrophages and prominent expansion of CD8+IL-17+ cells in pulmonary Paracoccidioidomycosis [Internet]. Journal of Infectious Diseases. 2014 ; 210( 5): 762-773.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1093/infdis/jiu136
Artigo de periodico
Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection (2013)
Feriotti, Claudia; Loures, Flávio Vieira; Araújo, Eliseu Frank de; Costa, Tania Alves da; Calich, Vera Lucia Garcia
Source: PLOS ONE. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
FERIOTTI, Claudia et al. Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection. PLOS ONE, v. 8, n. 1, p. 1-17, 2013Tradução . . Disponível em: https://doi.org/10.1590/S0004-27302013000100003. Acesso em: 03 nov. 2024.
APA
Feriotti, C., Loures, F. V., Araújo, E. F. de, Costa, T. A. da, & Calich, V. L. G. (2013). Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection. PLOS ONE, 8( 1), 1-17. doi:10.1590/S0004-27302013000100003
NLM
Feriotti C, Loures FV, Araújo EF de, Costa TA da, Calich VLG. Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection [Internet]. PLOS ONE. 2013 ; 8( 1): 1-17.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1590/S0004-27302013000100003
Vancouver
Feriotti C, Loures FV, Araújo EF de, Costa TA da, Calich VLG. Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection [Internet]. PLOS ONE. 2013 ; 8( 1): 1-17.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1590/S0004-27302013000100003
Artigo de periodico
MyD88 signaling is required for efficient innate and adaptive immune responses to Paracoccidioides brasiliensis infection (2011)
Loures, Flávio Vieira; Pina, Adriana; Felonato, Maíra; Feriotti, Claudia; Araújo, Eliseu Frank de; Calich, Vera Lucia Garcia
Source: Infection and Immunity. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LOURES, Flávio Vieira et al. MyD88 signaling is required for efficient innate and adaptive immune responses to Paracoccidioides brasiliensis infection. Infection and Immunity, v. 79, n. 6, p. 2470-2480, 2011Tradução . . Disponível em: https://doi.org/10.1128/IAI.00375-10. Acesso em: 03 nov. 2024.
APA
Loures, F. V., Pina, A., Felonato, M., Feriotti, C., Araújo, E. F. de, & Calich, V. L. G. (2011). MyD88 signaling is required for efficient innate and adaptive immune responses to Paracoccidioides brasiliensis infection. Infection and Immunity, 79( 6), 2470-2480. doi:10.1128/IAI.00375-10
NLM
Loures FV, Pina A, Felonato M, Feriotti C, Araújo EF de, Calich VLG. MyD88 signaling is required for efficient innate and adaptive immune responses to Paracoccidioides brasiliensis infection [Internet]. Infection and Immunity. 2011 ; 79( 6): 2470-2480.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1128/IAI.00375-10
Vancouver
Loures FV, Pina A, Felonato M, Feriotti C, Araújo EF de, Calich VLG. MyD88 signaling is required for efficient innate and adaptive immune responses to Paracoccidioides brasiliensis infection [Internet]. Infection and Immunity. 2011 ; 79( 6): 2470-2480.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1128/IAI.00375-10
Artigo de periodico
CD28 exerts protective and detrimental effects in a pulmonary model of paracoccidioidomycosis (2010)
Felonato, Maíra; Pina, Adriana; Bernardino, Simone; Loures, Flávio Vieira; Araújo, Eliseu Frank de; Calich, Vera Lúcia Garcia
Source: Infection and Immunity. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FELONATO, Maíra et al. CD28 exerts protective and detrimental effects in a pulmonary model of paracoccidioidomycosis. Infection and Immunity, v. 78, n. 11, p. 4922-4935, 2010Tradução . . Disponível em: https://doi.org/10.1128/IAI.00297-10. Acesso em: 03 nov. 2024.
APA
Felonato, M., Pina, A., Bernardino, S., Loures, F. V., Araújo, E. F. de, & Calich, V. L. G. (2010). CD28 exerts protective and detrimental effects in a pulmonary model of paracoccidioidomycosis. Infection and Immunity, 78( 11), 4922-4935. doi:10.1128/IAI.00297-10
NLM
Felonato M, Pina A, Bernardino S, Loures FV, Araújo EF de, Calich VLG. CD28 exerts protective and detrimental effects in a pulmonary model of paracoccidioidomycosis [Internet]. Infection and Immunity. 2010 ; 78( 11): 4922-4935.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1128/IAI.00297-10
Vancouver
Felonato M, Pina A, Bernardino S, Loures FV, Araújo EF de, Calich VLG. CD28 exerts protective and detrimental effects in a pulmonary model of paracoccidioidomycosis [Internet]. Infection and Immunity. 2010 ; 78( 11): 4922-4935.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1128/IAI.00297-10
Artigo de periodico
Toll-Like receptor 4 signaling leads to severe fungal infection associated with enhanced proinflammatory immunity and impaired expansion of regulatory T cells (2010)
Loures, Flávio Vieira; Pina, Adriana; Felonato, Maíra; Araújo, Eliseu Frank de; Leite, Katia R. M.; Calich, Vera Lúcia Garcia
Source: Infection and Immunity. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LOURES, Flávio Vieira et al. Toll-Like receptor 4 signaling leads to severe fungal infection associated with enhanced proinflammatory immunity and impaired expansion of regulatory T cells. Infection and Immunity, v. 78, n. 3, p. 1078-1088, 2010Tradução . . Acesso em: 03 nov. 2024.
APA
Loures, F. V., Pina, A., Felonato, M., Araújo, E. F. de, Leite, K. R. M., & Calich, V. L. G. (2010). Toll-Like receptor 4 signaling leads to severe fungal infection associated with enhanced proinflammatory immunity and impaired expansion of regulatory T cells. Infection and Immunity, 78( 3), 1078-1088.
NLM
Loures FV, Pina A, Felonato M, Araújo EF de, Leite KRM, Calich VLG. Toll-Like receptor 4 signaling leads to severe fungal infection associated with enhanced proinflammatory immunity and impaired expansion of regulatory T cells. Infection and Immunity. 2010 ; 78( 3): 1078-1088.[citado 2024 nov. 03 ]
Vancouver
Loures FV, Pina A, Felonato M, Araújo EF de, Leite KRM, Calich VLG. Toll-Like receptor 4 signaling leads to severe fungal infection associated with enhanced proinflammatory immunity and impaired expansion of regulatory T cells. Infection and Immunity. 2010 ; 78( 3): 1078-1088.[citado 2024 nov. 03 ]

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