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Artigo de periodico
Invariant natural killer T cells as key players in host resistance against Paracoccidioides brasiliensis (2021)
Nogueira Neto, Joes ; Loures, Flavio Vieira ; Schanoski, Alessandra Soares ; Andrade, David Anibal Garrido ; Gonzatti, Michelangelo Bauwelz ; Costa, Tania Alves da; Vivanco, Bruno Camolese ; Xander, Patrícia ; Rosa, Daniela S. ; Calich, Vera Lúcia Garcia ; Keller, Alexandre C.
Source: Journal of Immunology Research. Unidades: ICB, BIOTECNOLOGIA
Subjects: IMUNOLOGIA, PARACOCCIDIOIDES BRASILIENSIS, PARACOCCIDIOIDES, IMUNOLOGIA CELULAR, CAMUNDONGOS, MACRÓFAGOS, HOSPEDEIRO IMUNOCOMPROMETIDO, VIRULÊNCIA, NEUTRÓFILOS
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NOGUEIRA NETO, Joes et al. Invariant natural killer T cells as key players in host resistance against Paracoccidioides brasiliensis. Journal of Immunology Research, v. 2021, p. 1-11, 2021Tradução . . Disponível em: https://doi.org/10.1155/2021/6673722. Acesso em: 15 ago. 2024.
APA
Nogueira Neto, J., Loures, F. V., Schanoski, A. S., Andrade, D. A. G., Gonzatti, M. B., Costa, T. A. da, et al. (2021). Invariant natural killer T cells as key players in host resistance against Paracoccidioides brasiliensis. Journal of Immunology Research, 2021, 1-11. doi:10.1155/2021/6673722
NLM
Nogueira Neto J, Loures FV, Schanoski AS, Andrade DAG, Gonzatti MB, Costa TA da, Vivanco BC, Xander P, Rosa DS, Calich VLG, Keller AC. Invariant natural killer T cells as key players in host resistance against Paracoccidioides brasiliensis [Internet]. Journal of Immunology Research. 2021 ; 2021 1-11.[citado 2024 ago. 15 ] Available from: https://doi.org/10.1155/2021/6673722
Vancouver
Nogueira Neto J, Loures FV, Schanoski AS, Andrade DAG, Gonzatti MB, Costa TA da, Vivanco BC, Xander P, Rosa DS, Calich VLG, Keller AC. Invariant natural killer T cells as key players in host resistance against Paracoccidioides brasiliensis [Internet]. Journal of Immunology Research. 2021 ; 2021 1-11.[citado 2024 ago. 15 ] Available from: https://doi.org/10.1155/2021/6673722
Artigo de periodico
AhR ligands modulate the differentiation of innate lymphoid cells and T helper cell subsets that control the severity of a pulmonary fungal infection (2021)
Araújo, Eliseu Frank de; Loures, Flávio Vieira ; Preite, Nycolas Willian ; Feriotti, Cláudia ; Galdino, Nayane Alves de Lima ; Costa, Tania Alves da; Calich, Vera Lúcia Garcia
Source: Frontiers in Immunology. Unidade: ICB
Subjects: IMUNOLOGIA, LINFÓCITOS T, PARACOCCIDIOIDES, PARACOCCIDIOIDOMICOSE, INFECÇÕES RESPIRATÓRIAS, DOENÇAS RESPIRATÓRIAS, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS, LEUCÓCITOS, CÉLULAS DENDRÍTICAS
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ARAÚJO, Eliseu Frank de et al. AhR ligands modulate the differentiation of innate lymphoid cells and T helper cell subsets that control the severity of a pulmonary fungal infection. Frontiers in Immunology, v. 12, p. 1-17, 2021Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2021.630938. Acesso em: 15 ago. 2024.
APA
Araújo, E. F. de, Loures, F. V., Preite, N. W., Feriotti, C., Galdino, N. A. de L., Costa, T. A. da, & Calich, V. L. G. (2021). AhR ligands modulate the differentiation of innate lymphoid cells and T helper cell subsets that control the severity of a pulmonary fungal infection. Frontiers in Immunology, 12, 1-17. doi:10.3389/fimmu.2021.630938
NLM
Araújo EF de, Loures FV, Preite NW, Feriotti C, Galdino NA de L, Costa TA da, Calich VLG. AhR ligands modulate the differentiation of innate lymphoid cells and T helper cell subsets that control the severity of a pulmonary fungal infection [Internet]. Frontiers in Immunology. 2021 ; 12 1-17.[citado 2024 ago. 15 ] Available from: https://doi.org/10.3389/fimmu.2021.630938
Vancouver
Araújo EF de, Loures FV, Preite NW, Feriotti C, Galdino NA de L, Costa TA da, Calich VLG. AhR ligands modulate the differentiation of innate lymphoid cells and T helper cell subsets that control the severity of a pulmonary fungal infection [Internet]. Frontiers in Immunology. 2021 ; 12 1-17.[citado 2024 ago. 15 ] Available from: https://doi.org/10.3389/fimmu.2021.630938
Artigo de periodico
Disease tolerance mediated by phosphorylated indoleamine-2,3 dioxygenase confers resistance to a primary fungal pathogen (2017)
Araújo, Eliseu Frank de; Loures, Flavio Vieira ; Feriotti, Cláudia ; Costa, Tania; Vacca, Carmine; Puccetti, Paolo; Romani, Luigina; Calich, Vera Lúcia Garcia
Source: Frontiers in Immunology. Unidade: ICB
Subjects: IMUNOLOGIA, PARACOCCIDIOIDOMICOSE, CÉLULAS DENDRÍTICAS, ATIVAÇÃO ENZIMÁTICA, ENZIMAS OXIRREDUTORAS
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ARAÚJO, Eliseu Frank de et al. Disease tolerance mediated by phosphorylated indoleamine-2,3 dioxygenase confers resistance to a primary fungal pathogen. Frontiers in Immunology, v. 8, n. 1522, p. 1-14, 2017Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2017.01522. Acesso em: 15 ago. 2024.
APA
Araújo, E. F. de, Loures, F. V., Feriotti, C., Costa, T., Vacca, C., Puccetti, P., et al. (2017). Disease tolerance mediated by phosphorylated indoleamine-2,3 dioxygenase confers resistance to a primary fungal pathogen. Frontiers in Immunology, 8( 1522), 1-14. doi:10.3389/fimmu.2017.01522
NLM
Araújo EF de, Loures FV, Feriotti C, Costa T, Vacca C, Puccetti P, Romani L, Calich VLG. Disease tolerance mediated by phosphorylated indoleamine-2,3 dioxygenase confers resistance to a primary fungal pathogen [Internet]. Frontiers in Immunology. 2017 ; 8( 1522): 1-14.[citado 2024 ago. 15 ] Available from: https://doi.org/10.3389/fimmu.2017.01522
Vancouver
Araújo EF de, Loures FV, Feriotti C, Costa T, Vacca C, Puccetti P, Romani L, Calich VLG. Disease tolerance mediated by phosphorylated indoleamine-2,3 dioxygenase confers resistance to a primary fungal pathogen [Internet]. Frontiers in Immunology. 2017 ; 8( 1522): 1-14.[citado 2024 ago. 15 ] Available from: https://doi.org/10.3389/fimmu.2017.01522
Artigo de periodico
Paracoccidioidomycosis Associated With a Heterozygous STAT4 Mutation and Impaired IFN-γ Immunity (2017)
Schimke, Lena F; Hibbard, James; Martinez-Barricarte, Ruben; Khan, Taj Ali; Cavalcante, Ricardo de Souza; Oliveira-Junior, Edgar Borges de; França, Tábata Takahashi; Iqbal, Asif; Yamamoto, Guilherme; Arslanian, Christina; Feriotti, Claudia; Costa, Tânia Alves; Bustamante, Jacinta; Boisson-Dupuis, Stéphanie; Casanova, Jean-Laurent; Barbuto, José Alexandre Marzagão ; Zatz, Mayana ; Mendes, Rinaldo Poncio; Calich, Vera Lúcia Garcia; Ochs, Hans D; Torgerson, Troy R; Cabral-Marques, Otavio; Condino Neto, Antonio
Source: Journal of Infectious Diseases. Unidades: ICB, IB
Subjects: PARACOCCIDIOIDOMICOSE, IMUNOLOGIA, MUTAÇÃO GENÉTICA, PREDISPOSIÇÃO GENÉTICA PARA DOENÇA, INFECÇÕES BACTERIANAS E MICOSES
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ABNT
SCHIMKE, Lena F et al. Paracoccidioidomycosis Associated With a Heterozygous STAT4 Mutation and Impaired IFN-γ Immunity. Journal of Infectious Diseases, v. 216, n. 12, p. 1623-1634, 2017Tradução . . Disponível em: https://doi.org/10.1093/infdis/jix522. Acesso em: 15 ago. 2024.
APA
Schimke, L. F., Hibbard, J., Martinez-Barricarte, R., Khan, T. A., Cavalcante, R. de S., Oliveira-Junior, E. B. de, et al. (2017). Paracoccidioidomycosis Associated With a Heterozygous STAT4 Mutation and Impaired IFN-γ Immunity. Journal of Infectious Diseases, 216( 12), 1623-1634. doi:10.1093/infdis/jix522
NLM
Schimke LF, Hibbard J, Martinez-Barricarte R, Khan TA, Cavalcante R de S, Oliveira-Junior EB de, França TT, Iqbal A, Yamamoto G, Arslanian C, Feriotti C, Costa TA, Bustamante J, Boisson-Dupuis S, Casanova J-L, Barbuto JAM, Zatz M, Mendes RP, Calich VLG, Ochs HD, Torgerson TR, Cabral-Marques O, Condino Neto A. Paracoccidioidomycosis Associated With a Heterozygous STAT4 Mutation and Impaired IFN-γ Immunity [Internet]. Journal of Infectious Diseases. 2017 ; 216( 12): 1623-1634.[citado 2024 ago. 15 ] Available from: https://doi.org/10.1093/infdis/jix522
Vancouver
Schimke LF, Hibbard J, Martinez-Barricarte R, Khan TA, Cavalcante R de S, Oliveira-Junior EB de, França TT, Iqbal A, Yamamoto G, Arslanian C, Feriotti C, Costa TA, Bustamante J, Boisson-Dupuis S, Casanova J-L, Barbuto JAM, Zatz M, Mendes RP, Calich VLG, Ochs HD, Torgerson TR, Cabral-Marques O, Condino Neto A. Paracoccidioidomycosis Associated With a Heterozygous STAT4 Mutation and Impaired IFN-γ Immunity [Internet]. Journal of Infectious Diseases. 2017 ; 216( 12): 1623-1634.[citado 2024 ago. 15 ] Available from: https://doi.org/10.1093/infdis/jix522
Artigo de periodico
NOD-like receptor P3 inflammasome controls protective TH1/Th17 immunity against pulmonary paracoccidioidomycosis (2017)
Feriotti, Claudia ; Araújo, Eliseu Frank de; Loures, Flavio Vieira ; Costa, Tania Alves da; Galdino, Nayane Alves de Lima ; Zamboni, Dario Simões ; Calich, Vera Lúcia Garcia
Source: Frontiers in Immunology. Unidades: ICB, FMRP
Subjects: PARACOCCIDIOIDOMICOSE, INFECÇÕES RESPIRATÓRIAS, RECEPTORES, LINFÓCITOS T, IMUNIDADE
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ABNT
FERIOTTI, Claudia et al. NOD-like receptor P3 inflammasome controls protective TH1/Th17 immunity against pulmonary paracoccidioidomycosis. Frontiers in Immunology, v. 8, 2017Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2017.00786. Acesso em: 15 ago. 2024.
APA
Feriotti, C., Araújo, E. F. de, Loures, F. V., Costa, T. A. da, Galdino, N. A. de L., Zamboni, D. S., & Calich, V. L. G. (2017). NOD-like receptor P3 inflammasome controls protective TH1/Th17 immunity against pulmonary paracoccidioidomycosis. Frontiers in Immunology, 8. doi:10.3389/fimmu.2017.00786
NLM
Feriotti C, Araújo EF de, Loures FV, Costa TA da, Galdino NA de L, Zamboni DS, Calich VLG. NOD-like receptor P3 inflammasome controls protective TH1/Th17 immunity against pulmonary paracoccidioidomycosis [Internet]. Frontiers in Immunology. 2017 ; 8[citado 2024 ago. 15 ] Available from: https://doi.org/10.3389/fimmu.2017.00786
Vancouver
Feriotti C, Araújo EF de, Loures FV, Costa TA da, Galdino NA de L, Zamboni DS, Calich VLG. NOD-like receptor P3 inflammasome controls protective TH1/Th17 immunity against pulmonary paracoccidioidomycosis [Internet]. Frontiers in Immunology. 2017 ; 8[citado 2024 ago. 15 ] Available from: https://doi.org/10.3389/fimmu.2017.00786
Artigo de periodico
Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis (2015)
Feriotti, Claudia; Bazan, Silvia Boschi; Loures, Flávio Vieira; Araújo, Eliseu Frank de; Costa, Tânia Alves da; Calich, Vera Lucia Garcia
Source: Frontiers in Microbiology. Unidade: ICB
Subjects: IMUNOLOGIA, PARACOCCIDIOIDOMICOSE (RESISTÊNCIA), INFLAMAÇÃO
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ABNT
FERIOTTI, Claudia et al. Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis. Frontiers in Microbiology, v. 6, n. 913, p. 1-13, 2015Tradução . . Disponível em: https://doi.org/10.3389/fmicb.2015.00913. Acesso em: 15 ago. 2024.
APA
Feriotti, C., Bazan, S. B., Loures, F. V., Araújo, E. F. de, Costa, T. A. da, & Calich, V. L. G. (2015). Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis. Frontiers in Microbiology, 6( 913), 1-13. doi:10.3389/fmicb.2015.00913
NLM
Feriotti C, Bazan SB, Loures FV, Araújo EF de, Costa TA da, Calich VLG. Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis [Internet]. Frontiers in Microbiology. 2015 ; 6( 913): 1-13.[citado 2024 ago. 15 ] Available from: https://doi.org/10.3389/fmicb.2015.00913
Vancouver
Feriotti C, Bazan SB, Loures FV, Araújo EF de, Costa TA da, Calich VLG. Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis [Internet]. Frontiers in Microbiology. 2015 ; 6( 913): 1-13.[citado 2024 ago. 15 ] Available from: https://doi.org/10.3389/fmicb.2015.00913
Artigo de periodico
Loss- and gain-of-function approaches indicate a dual role exerted by regulatory T cells in pulmonary paracoccidioidomycosis (2015)
Bazan, Silvia Boschi; Costa, Tania Alves da; Araújo, Eliseu Frank de; Feriotti, Claudia; Loures, Flávio Vieira; Pretel, Fernando Delgado; Calich, Vera Lucia Garcia
Source: PLoS Neglected Tropical Diseases. Unidade: ICB
Subjects: IMUNOLOGIA, PARACOCCIDIOIDOMICOSE, IMUNIDADE CELULAR, CAMUNDONGOS
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ABNT
BAZAN, Silvia Boschi et al. Loss- and gain-of-function approaches indicate a dual role exerted by regulatory T cells in pulmonary paracoccidioidomycosis. PLoS Neglected Tropical Diseases, v. 9, n. 10, p. 1-24, 2015Tradução . . Disponível em: https://doi.org/10.1371/journal.pntd.0004189. Acesso em: 15 ago. 2024.
APA
Bazan, S. B., Costa, T. A. da, Araújo, E. F. de, Feriotti, C., Loures, F. V., Pretel, F. D., & Calich, V. L. G. (2015). Loss- and gain-of-function approaches indicate a dual role exerted by regulatory T cells in pulmonary paracoccidioidomycosis. PLoS Neglected Tropical Diseases, 9( 10), 1-24. doi:10.1371/journal.pntd.0004189
NLM
Bazan SB, Costa TA da, Araújo EF de, Feriotti C, Loures FV, Pretel FD, Calich VLG. Loss- and gain-of-function approaches indicate a dual role exerted by regulatory T cells in pulmonary paracoccidioidomycosis [Internet]. PLoS Neglected Tropical Diseases. 2015 ; 9( 10): 1-24.[citado 2024 ago. 15 ] Available from: https://doi.org/10.1371/journal.pntd.0004189
Vancouver
Bazan SB, Costa TA da, Araújo EF de, Feriotti C, Loures FV, Pretel FD, Calich VLG. Loss- and gain-of-function approaches indicate a dual role exerted by regulatory T cells in pulmonary paracoccidioidomycosis [Internet]. PLoS Neglected Tropical Diseases. 2015 ; 9( 10): 1-24.[citado 2024 ago. 15 ] Available from: https://doi.org/10.1371/journal.pntd.0004189
Artigo de periodico
Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis (2015)
Ribeiro, Laura Rossetti Rios; Loures, Flávio Vieira; Araújo, Eliseu Frank de; Feriotti, Cláudia; Costa, Tânia Alves da; Serezani, Carlos Henrique; Jancar, Sonia ; Calich, Vera Lucia Garcia
Source: Mediators of Inflammation. Unidade: ICB
Subjects: IMUNOLOGIA, PARACOCCIDIOIDES BRASILIENSIS, MACRÓFAGOS, INFECÇÕES RESPIRATÓRIAS
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ABNT
RIBEIRO, Laura Rossetti Rios et al. Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis. Mediators of Inflammation, v. 2015, p. 1-17, 2015Tradução . . Disponível em: https://doi.org/10.1155/2015/852574. Acesso em: 15 ago. 2024.
APA
Ribeiro, L. R. R., Loures, F. V., Araújo, E. F. de, Feriotti, C., Costa, T. A. da, Serezani, C. H., et al. (2015). Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis. Mediators of Inflammation, 2015, 1-17. doi:10.1155/2015/852574
NLM
Ribeiro LRR, Loures FV, Araújo EF de, Feriotti C, Costa TA da, Serezani CH, Jancar S, Calich VLG. Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis [Internet]. Mediators of Inflammation. 2015 ; 2015 1-17.[citado 2024 ago. 15 ] Available from: https://doi.org/10.1155/2015/852574
Vancouver
Ribeiro LRR, Loures FV, Araújo EF de, Feriotti C, Costa TA da, Serezani CH, Jancar S, Calich VLG. Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis [Internet]. Mediators of Inflammation. 2015 ; 2015 1-17.[citado 2024 ago. 15 ] Available from: https://doi.org/10.1155/2015/852574
Artigo de periodico
Dectin-1 induces M1 macrophages and prominent expansion of CD8+IL-17+ cells in pulmonary Paracoccidioidomycosis (2014)
Loures, Flávio V.; Araújo, Eliseu Frank de; Feriotti, Claudia; Bazan, Silvia B.; Costa, Tânia Alves da; Brown, Gordon D.; Calich, Vera Lucia Garcia
Source: Journal of Infectious Diseases. Unidade: ICB
Subjects: IMUNOLOGIA, MACRÓFAGOS, PARACOCCIDIOIDOMICOSE
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ABNT
LOURES, Flávio V. et al. Dectin-1 induces M1 macrophages and prominent expansion of CD8+IL-17+ cells in pulmonary Paracoccidioidomycosis. Journal of Infectious Diseases, v. 210, n. 5, p. 762-773, 2014Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiu136. Acesso em: 15 ago. 2024.
APA
Loures, F. V., Araújo, E. F. de, Feriotti, C., Bazan, S. B., Costa, T. A. da, Brown, G. D., & Calich, V. L. G. (2014). Dectin-1 induces M1 macrophages and prominent expansion of CD8+IL-17+ cells in pulmonary Paracoccidioidomycosis. Journal of Infectious Diseases, 210( 5), 762-773. doi:10.1093/infdis/jiu136
NLM
Loures FV, Araújo EF de, Feriotti C, Bazan SB, Costa TA da, Brown GD, Calich VLG. Dectin-1 induces M1 macrophages and prominent expansion of CD8+IL-17+ cells in pulmonary Paracoccidioidomycosis [Internet]. Journal of Infectious Diseases. 2014 ; 210( 5): 762-773.[citado 2024 ago. 15 ] Available from: https://doi.org/10.1093/infdis/jiu136
Vancouver
Loures FV, Araújo EF de, Feriotti C, Bazan SB, Costa TA da, Brown GD, Calich VLG. Dectin-1 induces M1 macrophages and prominent expansion of CD8+IL-17+ cells in pulmonary Paracoccidioidomycosis [Internet]. Journal of Infectious Diseases. 2014 ; 210( 5): 762-773.[citado 2024 ago. 15 ] Available from: https://doi.org/10.1093/infdis/jiu136
Artigo de periodico
Paracoccidoides brasiliensis 30 kDa adhesin: identification as a 14-3-3 protein, cloning and subcellular localization in infection models (2013)
Silva, Julhiany de Fatima da; Oliveira, Haroldo César de; Marcos, Caroline Maria; Silva, Rosângela Aparecida Moraes da; Costa, Tania Alves da; Calich, Vera Lucia García; Almeida, Ana Marisa Fusco; Mendes-Giannini, Maria José Soares
Source: PLOS ONE. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
SILVA, Julhiany de Fatima da et al. Paracoccidoides brasiliensis 30 kDa adhesin: identification as a 14-3-3 protein, cloning and subcellular localization in infection models. PLOS ONE, v. 8, n. 4, p. 1-10, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0062533. Acesso em: 15 ago. 2024.
APA
Silva, J. de F. da, Oliveira, H. C. de, Marcos, C. M., Silva, R. A. M. da, Costa, T. A. da, Calich, V. L. G., et al. (2013). Paracoccidoides brasiliensis 30 kDa adhesin: identification as a 14-3-3 protein, cloning and subcellular localization in infection models. PLOS ONE, 8( 4), 1-10. doi:10.1371/journal.pone.0062533
NLM
Silva J de F da, Oliveira HC de, Marcos CM, Silva RAM da, Costa TA da, Calich VLG, Almeida AMF, Mendes-Giannini MJS. Paracoccidoides brasiliensis 30 kDa adhesin: identification as a 14-3-3 protein, cloning and subcellular localization in infection models [Internet]. PLOS ONE. 2013 ;8( 4): 1-10.[citado 2024 ago. 15 ] Available from: https://doi.org/10.1371/journal.pone.0062533
Vancouver
Silva J de F da, Oliveira HC de, Marcos CM, Silva RAM da, Costa TA da, Calich VLG, Almeida AMF, Mendes-Giannini MJS. Paracoccidoides brasiliensis 30 kDa adhesin: identification as a 14-3-3 protein, cloning and subcellular localization in infection models [Internet]. PLOS ONE. 2013 ;8( 4): 1-10.[citado 2024 ago. 15 ] Available from: https://doi.org/10.1371/journal.pone.0062533
Artigo de periodico
Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection (2013)
Feriotti, Claudia; Loures, Flávio Vieira; Araújo, Eliseu Frank de; Costa, Tania Alves da; Calich, Vera Lucia Garcia
Source: PLOS ONE. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
FERIOTTI, Claudia et al. Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection. PLOS ONE, v. 8, n. 1, p. 1-17, 2013Tradução . . Disponível em: https://doi.org/10.1590/S0004-27302013000100003. Acesso em: 15 ago. 2024.
APA
Feriotti, C., Loures, F. V., Araújo, E. F. de, Costa, T. A. da, & Calich, V. L. G. (2013). Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection. PLOS ONE, 8( 1), 1-17. doi:10.1590/S0004-27302013000100003
NLM
Feriotti C, Loures FV, Araújo EF de, Costa TA da, Calich VLG. Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection [Internet]. PLOS ONE. 2013 ; 8( 1): 1-17.[citado 2024 ago. 15 ] Available from: https://doi.org/10.1590/S0004-27302013000100003
Vancouver
Feriotti C, Loures FV, Araújo EF de, Costa TA da, Calich VLG. Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection [Internet]. PLOS ONE. 2013 ; 8( 1): 1-17.[citado 2024 ago. 15 ] Available from: https://doi.org/10.1590/S0004-27302013000100003
Artigo de periodico
In pulmonary Paracoccidioidomycosis IL-10 deficiency leads to increased immunity and regressive infection without enhancing tissue pathology (2013)
Costa, Tânia Alves; Bazan, Silvia B.; Feriotti, Claudia; Araújo, Eliseu Frank; Bassi, Ênio José; Loures, Flávio V.; Calich, Vera Lucia Garcia
Source: Plos Neglected Tropical Diseases. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
COSTA, Tânia Alves et al. In pulmonary Paracoccidioidomycosis IL-10 deficiency leads to increased immunity and regressive infection without enhancing tissue pathology. Plos Neglected Tropical Diseases, v. 7, n. 10, p. 01-13, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pntd.0002512. Acesso em: 15 ago. 2024.
APA
Costa, T. A., Bazan, S. B., Feriotti, C., Araújo, E. F., Bassi, Ê. J., Loures, F. V., & Calich, V. L. G. (2013). In pulmonary Paracoccidioidomycosis IL-10 deficiency leads to increased immunity and regressive infection without enhancing tissue pathology. Plos Neglected Tropical Diseases, 7( 10), 01-13. doi:10.1371/journal.pntd.0002512
NLM
Costa TA, Bazan SB, Feriotti C, Araújo EF, Bassi ÊJ, Loures FV, Calich VLG. In pulmonary Paracoccidioidomycosis IL-10 deficiency leads to increased immunity and regressive infection without enhancing tissue pathology [Internet]. Plos Neglected Tropical Diseases. 2013 ; 7( 10): 01-13.[citado 2024 ago. 15 ] Available from: https://doi.org/10.1371/journal.pntd.0002512
Vancouver
Costa TA, Bazan SB, Feriotti C, Araújo EF, Bassi ÊJ, Loures FV, Calich VLG. In pulmonary Paracoccidioidomycosis IL-10 deficiency leads to increased immunity and regressive infection without enhancing tissue pathology [Internet]. Plos Neglected Tropical Diseases. 2013 ; 7( 10): 01-13.[citado 2024 ago. 15 ] Available from: https://doi.org/10.1371/journal.pntd.0002512

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