ReP USP - Resultado da busca

Artigo de periodico-carta/editorial
Insights on how mitochondrial NAD(P)+ transhydrogenase plays a central metabolic role in proinflammatory macrophages. [Carta] (2022)
Figueira, Tiago Rezende ; Castilho, Roger Frigério
Fonte: American Journal of Physiology-Cell Physiology. Unidade: EEFERP
Assuntos: MACRÓFAGOS, MITOCÔNDRIAS, ESPÉCIES REATIVAS DE OXIGÊNIO, ANTIOXIDANTES
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ABNT
FIGUEIRA, Tiago Rezende e CASTILHO, Roger Frigério. Insights on how mitochondrial NAD(P)+ transhydrogenase plays a central metabolic role in proinflammatory macrophages. [Carta]. American Journal of Physiology-Cell Physiology. Rockville: Escola de Educação Física e Esporte de Ribeirão Preto, Universidade de São Paulo. Disponível em: https://doi.org/10.1152/ajpcell.00090.2022. Acesso em: 08 nov. 2024. , 2022
APA
Figueira, T. R., & Castilho, R. F. (2022). Insights on how mitochondrial NAD(P)+ transhydrogenase plays a central metabolic role in proinflammatory macrophages. [Carta]. American Journal of Physiology-Cell Physiology. Rockville: Escola de Educação Física e Esporte de Ribeirão Preto, Universidade de São Paulo. doi:10.1152/ajpcell.00090.2022
NLM
Figueira TR, Castilho RF. Insights on how mitochondrial NAD(P)+ transhydrogenase plays a central metabolic role in proinflammatory macrophages. [Carta] [Internet]. American Journal of Physiology-Cell Physiology. 2022 ; 323( 2): C648-C649.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1152/ajpcell.00090.2022
Vancouver
Figueira TR, Castilho RF. Insights on how mitochondrial NAD(P)+ transhydrogenase plays a central metabolic role in proinflammatory macrophages. [Carta] [Internet]. American Journal of Physiology-Cell Physiology. 2022 ; 323( 2): C648-C649.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1152/ajpcell.00090.2022
Artigo de periodico
Dichloroacetate reactivates pyruvate-supported peroxide removal by liver mitochondria and prevents NAFLD aggravation in NAD(P)+ transhydrogenase-null mice consuming a high-fat diet (2022)
Navarro, Claudia D. C.; Francisco, Annelise; Figueira, Tiago Rezende ; Ronchi, Juliana A.; Oliveira, Helena Coutinho Franco de; Vercesi, Anibal Eugenio; Castilho, Roger Frigério
Fonte: European Journal of Pharmacology. Unidade: EEFERP
Assuntos: FÍGADO GORDUROSO, MUTAÇÃO, GORDURAS NA DIETA
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ABNT
NAVARRO, Claudia D. C. et al. Dichloroacetate reactivates pyruvate-supported peroxide removal by liver mitochondria and prevents NAFLD aggravation in NAD(P)+ transhydrogenase-null mice consuming a high-fat diet. European Journal of Pharmacology, v. 917, p. 1-10, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2022.174750. Acesso em: 08 nov. 2024.
APA
Navarro, C. D. C., Francisco, A., Figueira, T. R., Ronchi, J. A., Oliveira, H. C. F. de, Vercesi, A. E., & Castilho, R. F. (2022). Dichloroacetate reactivates pyruvate-supported peroxide removal by liver mitochondria and prevents NAFLD aggravation in NAD(P)+ transhydrogenase-null mice consuming a high-fat diet. European Journal of Pharmacology, 917, 1-10. doi:10.1016/j.ejphar.2022.174750
NLM
Navarro CDC, Francisco A, Figueira TR, Ronchi JA, Oliveira HCF de, Vercesi AE, Castilho RF. Dichloroacetate reactivates pyruvate-supported peroxide removal by liver mitochondria and prevents NAFLD aggravation in NAD(P)+ transhydrogenase-null mice consuming a high-fat diet [Internet]. European Journal of Pharmacology. 2022 ; 917 1-10.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.ejphar.2022.174750
Vancouver
Navarro CDC, Francisco A, Figueira TR, Ronchi JA, Oliveira HCF de, Vercesi AE, Castilho RF. Dichloroacetate reactivates pyruvate-supported peroxide removal by liver mitochondria and prevents NAFLD aggravation in NAD(P)+ transhydrogenase-null mice consuming a high-fat diet [Internet]. European Journal of Pharmacology. 2022 ; 917 1-10.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.ejphar.2022.174750
Artigo de periodico
Mitochondrial NAD(P)+ transhydrogenase is unevenly distributed in different brain regions, and its loss causes depressive-like behavior and motor dysfunction in mice (2020)
Francisco, Annelise; Engel, Daiane F.; Figueira, Tiago Rezende ; Rogério, Fábio; Bem, Andreza F. de; Castilho, Roger F.
Fonte: Neuroscience. Unidade: EEFERP
Assuntos: NEUROCIÊNCIAS, COMPORTAMENTO, PROTEÍNAS DA MEMBRANA, CÉREBRO, MODELOS ANIMAIS
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ABNT
FRANCISCO, Annelise et al. Mitochondrial NAD(P)+ transhydrogenase is unevenly distributed in different brain regions, and its loss causes depressive-like behavior and motor dysfunction in mice. Neuroscience, v. 440, p. 210-229, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2020.05.042. Acesso em: 08 nov. 2024.
APA
Francisco, A., Engel, D. F., Figueira, T. R., Rogério, F., Bem, A. F. de, & Castilho, R. F. (2020). Mitochondrial NAD(P)+ transhydrogenase is unevenly distributed in different brain regions, and its loss causes depressive-like behavior and motor dysfunction in mice. Neuroscience, 440, 210-229. doi:10.1016/j.neuroscience.2020.05.042
NLM
Francisco A, Engel DF, Figueira TR, Rogério F, Bem AF de, Castilho RF. Mitochondrial NAD(P)+ transhydrogenase is unevenly distributed in different brain regions, and its loss causes depressive-like behavior and motor dysfunction in mice [Internet]. Neuroscience. 2020 ; 440 210-229.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.neuroscience.2020.05.042
Vancouver
Francisco A, Engel DF, Figueira TR, Rogério F, Bem AF de, Castilho RF. Mitochondrial NAD(P)+ transhydrogenase is unevenly distributed in different brain regions, and its loss causes depressive-like behavior and motor dysfunction in mice [Internet]. Neuroscience. 2020 ; 440 210-229.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.neuroscience.2020.05.042
Artigo de periodico
Mitochondrial calcium transport and the redox nature of the calcium-induced membrane permeability transition (2018)
Vercesi, Anibal E; Castilho, Roger F; Kowaltowski, Alicia Juliana ; Oliveira, Helena C. F; Souza-Pinto, Nadja Cristhina de ; Figueira, Tiago Rezende ; Busanello, Estela N. B
Fonte: Free Radical Biology and Medicine. Unidades: IQ, EEFERP
Assuntos: MITOCÔNDRIAS, PERMEABILIDADE DA MEMBRANA CELULAR, CÁLCIO
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ABNT
VERCESI, Anibal E et al. Mitochondrial calcium transport and the redox nature of the calcium-induced membrane permeability transition. Free Radical Biology and Medicine, v. 129, p. 1-24, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2018.08.034. Acesso em: 08 nov. 2024.
APA
Vercesi, A. E., Castilho, R. F., Kowaltowski, A. J., Oliveira, H. C. F., Souza-Pinto, N. C. de, Figueira, T. R., & Busanello, E. N. B. (2018). Mitochondrial calcium transport and the redox nature of the calcium-induced membrane permeability transition. Free Radical Biology and Medicine, 129, 1-24. doi:10.1016/j.freeradbiomed.2018.08.034
NLM
Vercesi AE, Castilho RF, Kowaltowski AJ, Oliveira HCF, Souza-Pinto NC de, Figueira TR, Busanello ENB. Mitochondrial calcium transport and the redox nature of the calcium-induced membrane permeability transition [Internet]. Free Radical Biology and Medicine. 2018 ; 129 1-24.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2018.08.034
Vancouver
Vercesi AE, Castilho RF, Kowaltowski AJ, Oliveira HCF, Souza-Pinto NC de, Figueira TR, Busanello ENB. Mitochondrial calcium transport and the redox nature of the calcium-induced membrane permeability transition [Internet]. Free Radical Biology and Medicine. 2018 ; 129 1-24.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2018.08.034
Artigo de periodico
Nicotinamide nucleotide transhydrogenase is required for brain mitochondrial redox balance under hampered energy substrate metabolism and high‐fat diet (2018)
Francisco, Annelise; Ronchi, Juliana A.; Navarro, Claudia D. C.; Figueira, Tiago Rezende ; Castilho, Roger F.
Fonte: Journal of Neurochemistry. Unidade: EEFERP
Assuntos: CÉREBRO, BIOQUÍMICA, NEUROQUÍMICA
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ABNT
FRANCISCO, Annelise et al. Nicotinamide nucleotide transhydrogenase is required for brain mitochondrial redox balance under hampered energy substrate metabolism and high‐fat diet. Journal of Neurochemistry, v. 147, n. 5, p. 663-677, 2018Tradução . . Disponível em: https://doi.org/10.1111/jnc.14602. Acesso em: 08 nov. 2024.
APA
Francisco, A., Ronchi, J. A., Navarro, C. D. C., Figueira, T. R., & Castilho, R. F. (2018). Nicotinamide nucleotide transhydrogenase is required for brain mitochondrial redox balance under hampered energy substrate metabolism and high‐fat diet. Journal of Neurochemistry, 147( 5), 663-677. doi:10.1111/jnc.14602
NLM
Francisco A, Ronchi JA, Navarro CDC, Figueira TR, Castilho RF. Nicotinamide nucleotide transhydrogenase is required for brain mitochondrial redox balance under hampered energy substrate metabolism and high‐fat diet [Internet]. Journal of Neurochemistry. 2018 ; 147( 5): 663-677.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1111/jnc.14602
Vancouver
Francisco A, Ronchi JA, Navarro CDC, Figueira TR, Castilho RF. Nicotinamide nucleotide transhydrogenase is required for brain mitochondrial redox balance under hampered energy substrate metabolism and high‐fat diet [Internet]. Journal of Neurochemistry. 2018 ; 147( 5): 663-677.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1111/jnc.14602
Artigo de periodico
Facilitation of Ca2+-induced opening of the mitochondrial permeability transition pore either by nicotinamide nucleotide transhydrogenase deficiency or statins treatment (2018)
Busanello, Estela N. B.; Figueira, Tiago Rezende ; Marques, Ana C.; Navarro, Claudia D. C.; Oliveira, Helena Coutinho Franco de ; Vercesi, Anibal Eugenio
Fonte: Cell Biology International. Unidade: EEFERP
Assuntos: RATOS, MUTAÇÃO, MITOCÔNDRIAS, DIETA, FÍGADO GORDUROSO, PROTEÍNAS DE TRANSPORTE
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ABNT
BUSANELLO, Estela N. B. et al. Facilitation of Ca2+-induced opening of the mitochondrial permeability transition pore either by nicotinamide nucleotide transhydrogenase deficiency or statins treatment. Cell Biology International, v. 42, n. 6, p. 742-746, 2018Tradução . . Disponível em: https://doi.org/10.1002/cbin.10949. Acesso em: 08 nov. 2024.
APA
Busanello, E. N. B., Figueira, T. R., Marques, A. C., Navarro, C. D. C., Oliveira, H. C. F. de, & Vercesi, A. E. (2018). Facilitation of Ca2+-induced opening of the mitochondrial permeability transition pore either by nicotinamide nucleotide transhydrogenase deficiency or statins treatment. Cell Biology International, 42( 6), 742-746. doi:10.1002/cbin.10949
NLM
Busanello ENB, Figueira TR, Marques AC, Navarro CDC, Oliveira HCF de, Vercesi AE. Facilitation of Ca2+-induced opening of the mitochondrial permeability transition pore either by nicotinamide nucleotide transhydrogenase deficiency or statins treatment [Internet]. Cell Biology International. 2018 ; 42( 6): 742-746.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1002/cbin.10949
Vancouver
Busanello ENB, Figueira TR, Marques AC, Navarro CDC, Oliveira HCF de, Vercesi AE. Facilitation of Ca2+-induced opening of the mitochondrial permeability transition pore either by nicotinamide nucleotide transhydrogenase deficiency or statins treatment [Internet]. Cell Biology International. 2018 ; 42( 6): 742-746.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1002/cbin.10949
Artigo de periodico
Mitochondria as a source of reactive oxygen and nitrogen species: from molecular mechanisms to human health (2013)
Figueira, Tiago Rezende ; Barros, Mário Henrique de ; Camargo, Anamaria A.; Castilho, Roger F.; Ferreira, Julio Cesar Batista ; Kowaltowski, Alicia Juliana ; Sluse, Francis E.; Souza-Pinto, Nadja Cristhina de ; Vercesi, Anibal E.
Fonte: Antioxidants & Redox Signaling. Unidades: ICB, IQ
Assuntos: ANATOMIA, MICROBIOLOGIA
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ABNT
FIGUEIRA, Tiago Rezende et al. Mitochondria as a source of reactive oxygen and nitrogen species: from molecular mechanisms to human health. Antioxidants & Redox Signaling, v. 18, n. 16, p. 1-46, 2013Tradução . . Disponível em: https://doi.org/10.1089/ars.2012.4729. Acesso em: 08 nov. 2024.
APA
Figueira, T. R., Barros, M. H. de, Camargo, A. A., Castilho, R. F., Ferreira, J. C. B., Kowaltowski, A. J., et al. (2013). Mitochondria as a source of reactive oxygen and nitrogen species: from molecular mechanisms to human health. Antioxidants & Redox Signaling, 18( 16), 1-46. doi:10.1089/ars.2012.4729
NLM
Figueira TR, Barros MH de, Camargo AA, Castilho RF, Ferreira JCB, Kowaltowski AJ, Sluse FE, Souza-Pinto NC de, Vercesi AE. Mitochondria as a source of reactive oxygen and nitrogen species: from molecular mechanisms to human health [Internet]. Antioxidants & Redox Signaling. 2013 ; 18( 16): 1-46.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1089/ars.2012.4729
Vancouver
Figueira TR, Barros MH de, Camargo AA, Castilho RF, Ferreira JCB, Kowaltowski AJ, Sluse FE, Souza-Pinto NC de, Vercesi AE. Mitochondria as a source of reactive oxygen and nitrogen species: from molecular mechanisms to human health [Internet]. Antioxidants & Redox Signaling. 2013 ; 18( 16): 1-46.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1089/ars.2012.4729

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