Nicotinamide nucleotide transhydrogenase is required for brain mitochondrial redox balance under hampered energy substrate metabolism and high‐fat diet (2018)
- Authors:
- Autor USP: FIGUEIRA, TIAGO REZENDE - EEFERP
- Unidade: EEFERP
- DOI: 10.1111/jnc.14602
- Subjects: CÉREBRO; BIOQUÍMICA; NEUROQUÍMICA
- Keywords: Brain; C57BL/6; H+-thase; NAD(P)+transhydrogenase; Redox balance
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Publisher place: West Sussex
- Date published: 2018
- Source:
- Título: Journal of Neurochemistry
- ISSN: 0022-3042
- Volume/Número/Paginação/Ano: v. 147, n. 5, p. 663-677, 2018
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: bronze
-
ABNT
FRANCISCO, Annelise et al. Nicotinamide nucleotide transhydrogenase is required for brain mitochondrial redox balance under hampered energy substrate metabolism and high‐fat diet. Journal of Neurochemistry, v. 147, n. 5, p. 663-677, 2018Tradução . . Disponível em: https://doi.org/10.1111/jnc.14602. Acesso em: 20 mar. 2025. -
APA
Francisco, A., Ronchi, J. A., Navarro, C. D. C., Figueira, T. R., & Castilho, R. F. (2018). Nicotinamide nucleotide transhydrogenase is required for brain mitochondrial redox balance under hampered energy substrate metabolism and high‐fat diet. Journal of Neurochemistry, 147( 5), 663-677. doi:10.1111/jnc.14602 -
NLM
Francisco A, Ronchi JA, Navarro CDC, Figueira TR, Castilho RF. Nicotinamide nucleotide transhydrogenase is required for brain mitochondrial redox balance under hampered energy substrate metabolism and high‐fat diet [Internet]. Journal of Neurochemistry. 2018 ; 147( 5): 663-677.[citado 2025 mar. 20 ] Available from: https://doi.org/10.1111/jnc.14602 -
Vancouver
Francisco A, Ronchi JA, Navarro CDC, Figueira TR, Castilho RF. Nicotinamide nucleotide transhydrogenase is required for brain mitochondrial redox balance under hampered energy substrate metabolism and high‐fat diet [Internet]. Journal of Neurochemistry. 2018 ; 147( 5): 663-677.[citado 2025 mar. 20 ] Available from: https://doi.org/10.1111/jnc.14602 - Insights on how mitochondrial NAD(P)+ transhydrogenase plays a central metabolic role in proinflammatory macrophages. [Carta]
- Dichloroacetate reactivates pyruvate-supported peroxide removal by liver mitochondria and prevents NAFLD aggravation in NAD(P)+ transhydrogenase-null mice consuming a high-fat diet
- NADPH supply and the contribution of NAD(P)+ transhydrogenase (NNT) to H2O2 balance in skeletal muscle mitochondria
- Facilitation of Ca2+-induced opening of the mitochondrial permeability transition pore either by nicotinamide nucleotide transhydrogenase deficiency or statins treatment
- Mitochondrial NAD(P)+ transhydrogenase is unevenly distributed in different brain regions, and its loss causes depressive-like behavior and motor dysfunction in mice
- Can NAD(P)+ transhydrogenase (NNT) mediate a physiologically meaningful increase in energy expenditure by mitochondria during H2O2 removal? [Carta]
- Mitochondrial calcium transport and the redox nature of the calcium-induced membrane permeability transition
Informações sobre o DOI: 10.1111/jnc.14602 (Fonte: oaDOI API)
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