Mitochondrial NAD(P) + transhydrogenase: from molecular features to physiology and disease (2022)
- Authors:
- Autor USP: FIGUEIRA, TIAGO REZENDE - EEFERP
- Unidade: EEFERP
- DOI: 10.1089/ars.2021.0111
- Subjects: MITOCÔNDRIAS; MOLÉCULA; FISIOLOGIA; DOENÇAS
- Keywords: Nicotinamide nucleotide transhydrogenase; Redox balance; Biosynthesis; Mitochondrion
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Publisher place: New Rochelle
- Date published: 2022
- Source:
- Título: Antioxidants & Redox Signaling
- ISSN: 1523-0864
- Volume/Número/Paginação/Ano: v. 36, n. 13-15, p. 864-884, 2022
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
FRANCISCO, Annelise e FIGUEIRA, Tiago Rezende e CASTILHO, Roger Frigério. Mitochondrial NAD(P) + transhydrogenase: from molecular features to physiology and disease. Antioxidants & Redox Signaling, v. 36, n. 13-15, p. 864-884, 2022Tradução . . Disponível em: https://doi.org/10.1089/ars.2021.0111. Acesso em: 01 jan. 2026. -
APA
Francisco, A., Figueira, T. R., & Castilho, R. F. (2022). Mitochondrial NAD(P) + transhydrogenase: from molecular features to physiology and disease. Antioxidants & Redox Signaling, 36( 13-15), 864-884. doi:10.1089/ars.2021.0111 -
NLM
Francisco A, Figueira TR, Castilho RF. Mitochondrial NAD(P) + transhydrogenase: from molecular features to physiology and disease [Internet]. Antioxidants & Redox Signaling. 2022 ; 36( 13-15): 864-884.[citado 2026 jan. 01 ] Available from: https://doi.org/10.1089/ars.2021.0111 -
Vancouver
Francisco A, Figueira TR, Castilho RF. Mitochondrial NAD(P) + transhydrogenase: from molecular features to physiology and disease [Internet]. Antioxidants & Redox Signaling. 2022 ; 36( 13-15): 864-884.[citado 2026 jan. 01 ] Available from: https://doi.org/10.1089/ars.2021.0111 - Insights on how mitochondrial NAD(P)+ transhydrogenase plays a central metabolic role in proinflammatory macrophages. [Carta]
- NADPH supply and the contribution of NAD(P)+ transhydrogenase (NNT) to H2O2 balance in skeletal muscle mitochondria
- Facilitation of Ca2+-induced opening of the mitochondrial permeability transition pore either by nicotinamide nucleotide transhydrogenase deficiency or statins treatment
- Dichloroacetate reactivates pyruvate-supported peroxide removal by liver mitochondria and prevents NAFLD aggravation in NAD(P)+ transhydrogenase-null mice consuming a high-fat diet
- Nicotinamide nucleotide transhydrogenase is required for brain mitochondrial redox balance under hampered energy substrate metabolism and high‐fat diet
- Can NAD(P)+ transhydrogenase (NNT) mediate a physiologically meaningful increase in energy expenditure by mitochondria during H2O2 removal? [Carta]
- Mitochondrial NAD(P)+ transhydrogenase is unevenly distributed in different brain regions, and its loss causes depressive-like behavior and motor dysfunction in mice
- Mitochondrial calcium transport and the redox nature of the calcium-induced membrane permeability transition
Informações sobre o DOI: 10.1089/ars.2021.0111 (Fonte: oaDOI API)
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