Facilitation of Ca2+-induced opening of the mitochondrial permeability transition pore either by nicotinamide nucleotide transhydrogenase deficiency or statins treatment (2018)
- Authors:
- Autor USP: FIGUEIRA, TIAGO REZENDE - EEFERP
- Unidade: EEFERP
- DOI: 10.1002/cbin.10949
- Subjects: RATOS; MUTAÇÃO; MITOCÔNDRIAS; DIETA; FÍGADO GORDUROSO; PROTEÍNAS DE TRANSPORTE
- Keywords: C57BL/6J mice; Mitochondrial permeability transition; Nicotinamide nucleotide transhydrogenase; Nnt mutation; Pravastatin; Simvastatin
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Cell Biology International
- ISSN: 1065-6995
- Volume/Número/Paginação/Ano: v. 42, n. 6, p. 742-746, 2018
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
BUSANELLO, Estela N. B. et al. Facilitation of Ca2+-induced opening of the mitochondrial permeability transition pore either by nicotinamide nucleotide transhydrogenase deficiency or statins treatment. Cell Biology International, v. 42, n. 6, p. 742-746, 2018Tradução . . Disponível em: https://doi.org/10.1002/cbin.10949. Acesso em: 23 jul. 2024. -
APA
Busanello, E. N. B., Figueira, T. R., Marques, A. C., Navarro, C. D. C., Oliveira, H. C. F. de, & Vercesi, A. E. (2018). Facilitation of Ca2+-induced opening of the mitochondrial permeability transition pore either by nicotinamide nucleotide transhydrogenase deficiency or statins treatment. Cell Biology International, 42( 6), 742-746. doi:10.1002/cbin.10949 -
NLM
Busanello ENB, Figueira TR, Marques AC, Navarro CDC, Oliveira HCF de, Vercesi AE. Facilitation of Ca2+-induced opening of the mitochondrial permeability transition pore either by nicotinamide nucleotide transhydrogenase deficiency or statins treatment [Internet]. Cell Biology International. 2018 ; 42( 6): 742-746.[citado 2024 jul. 23 ] Available from: https://doi.org/10.1002/cbin.10949 -
Vancouver
Busanello ENB, Figueira TR, Marques AC, Navarro CDC, Oliveira HCF de, Vercesi AE. Facilitation of Ca2+-induced opening of the mitochondrial permeability transition pore either by nicotinamide nucleotide transhydrogenase deficiency or statins treatment [Internet]. Cell Biology International. 2018 ; 42( 6): 742-746.[citado 2024 jul. 23 ] Available from: https://doi.org/10.1002/cbin.10949 - Insights on how mitochondrial NAD(P)+ transhydrogenase plays a central metabolic role in proinflammatory macrophages. [Carta]
- Nicotinamide nucleotide transhydrogenase is required for brain mitochondrial redox balance under hampered energy substrate metabolism and high‐fat diet
- Mitochondrial NAD(P)+ transhydrogenase is unevenly distributed in different brain regions, and its loss causes depressive-like behavior and motor dysfunction in mice
- Dichloroacetate reactivates pyruvate-supported peroxide removal by liver mitochondria and prevents NAFLD aggravation in NAD(P)+ transhydrogenase-null mice consuming a high-fat diet
- Mitochondrial calcium transport and the redox nature of the calcium-induced membrane permeability transition
Informações sobre o DOI: 10.1002/cbin.10949 (Fonte: oaDOI API)
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