Mitochondrial NAD(P)+ transhydrogenase is unevenly distributed in different brain regions, and its loss causes depressive-like behavior and motor dysfunction in mice (2020)
- Authors:
- Autor USP: FIGUEIRA, TIAGO REZENDE - EEFERP
- Unidade: EEFERP
- DOI: 10.1016/j.neuroscience.2020.05.042
- Subjects: NEUROCIÊNCIAS; COMPORTAMENTO; PROTEÍNAS DA MEMBRANA; CÉREBRO; MODELOS ANIMAIS
- Keywords: C57BL/6; Redox balance; Monoamine; Nicotinamide nucleotide transhydrogenase; Mitochondria; NADPH
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Neuroscience
- ISSN: 0306-4522
- Volume/Número/Paginação/Ano: v. 440, p. 210-229, 2020
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
FRANCISCO, Annelise et al. Mitochondrial NAD(P)+ transhydrogenase is unevenly distributed in different brain regions, and its loss causes depressive-like behavior and motor dysfunction in mice. Neuroscience, v. 440, p. 210-229, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2020.05.042. Acesso em: 23 maio 2025. -
APA
Francisco, A., Engel, D. F., Figueira, T. R., Rogério, F., Bem, A. F. de, & Castilho, R. F. (2020). Mitochondrial NAD(P)+ transhydrogenase is unevenly distributed in different brain regions, and its loss causes depressive-like behavior and motor dysfunction in mice. Neuroscience, 440, 210-229. doi:10.1016/j.neuroscience.2020.05.042 -
NLM
Francisco A, Engel DF, Figueira TR, Rogério F, Bem AF de, Castilho RF. Mitochondrial NAD(P)+ transhydrogenase is unevenly distributed in different brain regions, and its loss causes depressive-like behavior and motor dysfunction in mice [Internet]. Neuroscience. 2020 ; 440 210-229.[citado 2025 maio 23 ] Available from: https://doi.org/10.1016/j.neuroscience.2020.05.042 -
Vancouver
Francisco A, Engel DF, Figueira TR, Rogério F, Bem AF de, Castilho RF. Mitochondrial NAD(P)+ transhydrogenase is unevenly distributed in different brain regions, and its loss causes depressive-like behavior and motor dysfunction in mice [Internet]. Neuroscience. 2020 ; 440 210-229.[citado 2025 maio 23 ] Available from: https://doi.org/10.1016/j.neuroscience.2020.05.042 - Insights on how mitochondrial NAD(P)+ transhydrogenase plays a central metabolic role in proinflammatory macrophages. [Carta]
- Dichloroacetate reactivates pyruvate-supported peroxide removal by liver mitochondria and prevents NAFLD aggravation in NAD(P)+ transhydrogenase-null mice consuming a high-fat diet
- NADPH supply and the contribution of NAD(P)+ transhydrogenase (NNT) to H2O2 balance in skeletal muscle mitochondria
- Facilitation of Ca2+-induced opening of the mitochondrial permeability transition pore either by nicotinamide nucleotide transhydrogenase deficiency or statins treatment
- Nicotinamide nucleotide transhydrogenase is required for brain mitochondrial redox balance under hampered energy substrate metabolism and high‐fat diet
- Can NAD(P)+ transhydrogenase (NNT) mediate a physiologically meaningful increase in energy expenditure by mitochondria during H2O2 removal? [Carta]
- Mitochondrial calcium transport and the redox nature of the calcium-induced membrane permeability transition
Informações sobre o DOI: 10.1016/j.neuroscience.2020.05.042 (Fonte: oaDOI API)
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